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Items: 1 to 20 of 142

1.

Site-1 protease is essential to growth plate maintenance and is a critical regulator of chondrocyte hypertrophic differentiation in postnatal mice.

Patra D, DeLassus E, Hayashi S, Sandell LJ.

J Biol Chem. 2011 Aug 19;286(33):29227-40. doi: 10.1074/jbc.M110.208686. Epub 2011 Jun 7.

2.

Site-1 protease is essential for endochondral bone formation in mice.

Patra D, Xing X, Davies S, Bryan J, Franz C, Hunziker EB, Sandell LJ.

J Cell Biol. 2007 Nov 19;179(4):687-700.

3.

Cartilage-specific ablation of site-1 protease in mice results in the endoplasmic reticulum entrapment of type IIb procollagen and down-regulation of cholesterol and lipid homeostasis.

Patra D, DeLassus E, Liang G, Sandell LJ.

PLoS One. 2014 Aug 22;9(8):e105674. doi: 10.1371/journal.pone.0105674. eCollection 2014.

4.

Cartilage-specific ablation of XBP1 signaling in mouse results in a chondrodysplasia characterized by reduced chondrocyte proliferation and delayed cartilage maturation and mineralization.

Cameron TL, Gresshoff IL, Bell KM, Piróg KA, Sampurno L, Hartley CL, Sanford EM, Wilson R, Ermann J, Boot-Handford RP, Glimcher LH, Briggs MD, Bateman JF.

Osteoarthritis Cartilage. 2015 Apr;23(4):661-70. doi: 10.1016/j.joca.2015.01.001. Epub 2015 Jan 17.

5.

Reduced chondrocyte proliferation and chondrodysplasia in mice lacking the integrin-linked kinase in chondrocytes.

Terpstra L, Prud'homme J, Arabian A, Takeda S, Karsenty G, Dedhar S, St-Arnaud R.

J Cell Biol. 2003 Jul 7;162(1):139-48. Epub 2003 Jun 30.

6.

Chondrocyte-specific knockout of Cbfβ reveals the indispensable function of Cbfβ in chondrocyte maturation, growth plate development and trabecular bone formation in mice.

Wu M, Li YP, Zhu G, Lu Y, Wang Y, Jules J, McConnell M, Serra R, Shao JZ, Chen W.

Int J Biol Sci. 2014 Jul 29;10(8):861-72. doi: 10.7150/ijbs.8521. eCollection 2014.

7.

Partial rescue of postnatal growth plate abnormalities in Ihh mutants by expression of a constitutively active PTH/PTHrP receptor.

Maeda Y, Schipani E, Densmore MJ, Lanske B.

Bone. 2010 Feb;46(2):472-8. doi: 10.1016/j.bone.2009.09.009. Epub 2009 Sep 15.

8.

Defective postnatal endochondral bone development by chondrocyte-specific targeted expression of parathyroid hormone type 2 receptor.

Panda DK, Goltzman D, Karaplis AC.

Am J Physiol Endocrinol Metab. 2012 Dec 15;303(12):E1489-501. doi: 10.1152/ajpendo.00254.2012. Epub 2012 Oct 23.

9.

Complementary interplay between matrix metalloproteinase-9, vascular endothelial growth factor and osteoclast function drives endochondral bone formation.

Ortega N, Wang K, Ferrara N, Werb Z, Vu TH.

Dis Model Mech. 2010 Mar-Apr;3(3-4):224-35. doi: 10.1242/dmm.004226. Epub 2010 Feb 8.

10.

Atf4 regulates chondrocyte proliferation and differentiation during endochondral ossification by activating Ihh transcription.

Wang W, Lian N, Li L, Moss HE, Wang W, Perrien DS, Elefteriou F, Yang X.

Development. 2009 Dec;136(24):4143-53. doi: 10.1242/dev.043281. Epub 2009 Nov 11.

11.

The critical role of the epidermal growth factor receptor in endochondral ossification.

Zhang X, Siclari VA, Lan S, Zhu J, Koyama E, Dupuis HL, Enomoto-Iwamoto M, Beier F, Qin L.

J Bone Miner Res. 2011 Nov;26(11):2622-33. doi: 10.1002/jbmr.502.

12.

Perlecan modulates VEGF signaling and is essential for vascularization in endochondral bone formation.

Ishijima M, Suzuki N, Hozumi K, Matsunobu T, Kosaki K, Kaneko H, Hassell JR, Arikawa-Hirasawa E, Yamada Y.

Matrix Biol. 2012 May;31(4):234-45. doi: 10.1016/j.matbio.2012.02.006. Epub 2012 Mar 7.

13.

Secreted frizzled related protein 1 regulates Wnt signaling for BMP2 induced chondrocyte differentiation.

Gaur T, Rich L, Lengner CJ, Hussain S, Trevant B, Ayers D, Stein JL, Bodine PV, Komm BS, Stein GS, Lian JB.

J Cell Physiol. 2006 Jul;208(1):87-96.

PMID:
16575902
14.

Targeted overexpression of parathyroid hormone-related peptide in chondrocytes causes chondrodysplasia and delayed endochondral bone formation.

Weir EC, Philbrick WM, Amling M, Neff LA, Baron R, Broadus AE.

Proc Natl Acad Sci U S A. 1996 Sep 17;93(19):10240-5.

15.

Conditional deletion of Indian hedgehog from collagen type 2alpha1-expressing cells results in abnormal endochondral bone formation.

Razzaque MS, Soegiarto DW, Chang D, Long F, Lanske B.

J Pathol. 2005 Dec;207(4):453-61.

PMID:
16278811
16.

Inhibition of cyclooxygenase-2 impacts chondrocyte hypertrophic differentiation during endochondral ossification.

Welting TJ, Caron MM, Emans PJ, Janssen MP, Sanen K, Coolsen MM, Voss L, Surtel DA, Cremers A, Voncken JW, van Rhijn LW.

Eur Cell Mater. 2011 Dec 19;22:420-36; discussion 436-7.

17.

Targeted expression of SHH affects chondrocyte differentiation, growth plate organization, and Sox9 expression.

Tavella S, Biticchi R, Schito A, Minina E, Di Martino D, Pagano A, Vortkamp A, Horton WA, Cancedda R, Garofalo S.

J Bone Miner Res. 2004 Oct;19(10):1678-88. Epub 2004 Jul 12.

18.

Persistent expression of Twist1 in chondrocytes causes growth plate abnormalities and dwarfism in mice.

Guzzo RM, Andreeva V, Spicer DB, Drissi MH.

Int J Dev Biol. 2011;55(6):641-7. doi: 10.1387/ijdb.103274rg. Erratum in: Int J Dev Biol. 2012;56(4):311.

19.

ER Stress During the Pubertal Growth Spurt Results in Impaired Long-Bone Growth in Chondrocyte-Specific ERp57 Knockout Mice.

Linz A, Knieper Y, Gronau T, Hansen U, Aszodi A, Garbi N, Hämmerling GJ, Pap T, Bruckner P, Dreier R.

J Bone Miner Res. 2015 Aug;30(8):1481-93. doi: 10.1002/jbmr.2484. Epub 2015 Jun 16.

20.

A new histomorphometric method to assess growth plate chondrodysplasia and its application to the toothless (tl, Csf1(null)) osteopetrotic rat.

Devraj K, Bonassar LJ, MacKay CA, Mason-Savas A, Gartland A, Odgren PR.

Connect Tissue Res. 2004;45(1):1-10.

PMID:
15203935

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