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Items: 1 to 20 of 154

1.

Lymphoadenopathy during lyme borreliosis is caused by spirochete migration-induced specific B cell activation.

Tunev SS, Hastey CJ, Hodzic E, Feng S, Barthold SW, Baumgarth N.

PLoS Pathog. 2011 May;7(5):e1002066. doi: 10.1371/journal.ppat.1002066. Epub 2011 May 26.

2.

MyD88- and TRIF-independent induction of type I interferon drives naive B cell accumulation but not loss of lymph node architecture in Lyme disease.

Hastey CJ, Ochoa J, Olsen KJ, Barthold SW, Baumgarth N.

Infect Immun. 2014 Apr;82(4):1548-58. doi: 10.1128/IAI.00969-13. Epub 2014 Jan 22.

3.

Dynamics of connective-tissue localization during chronic Borrelia burgdorferi infection.

Imai DM, Feng S, Hodzic E, Barthold SW.

Lab Invest. 2013 Aug;93(8):900-10. doi: 10.1038/labinvest.2013.81. Epub 2013 Jun 24.

4.

Delays and diversions mark the development of B cell responses to Borrelia burgdorferi infection.

Hastey CJ, Elsner RA, Barthold SW, Baumgarth N.

J Immunol. 2012 Jun 1;188(11):5612-22. doi: 10.4049/jimmunol.1103735. Epub 2012 Apr 30.

5.

Antibody Response to Lyme Disease Spirochetes in the Context of VlsE-Mediated Immune Evasion.

Rogovskyy AS, Gillis DC, Ionov Y, Gerasimov E, Zelikovsky A.

Infect Immun. 2016 Dec 29;85(1). pii: e00890-16. doi: 10.1128/IAI.00890-16. Print 2017 Jan.

6.

Disordered lymphoid purine metabolism contributes to the pathogenesis of persistent Borrelia garinii infection in mice.

Yegutkin GG, Hytönen J, Samburski SS, Yrjänäinen H, Jalkanen S, Viljanen MK.

J Immunol. 2010 May 1;184(9):5112-20. doi: 10.4049/jimmunol.0902760. Epub 2010 Mar 31.

7.

MyD88 plays a unique role in host defense but not arthritis development in Lyme disease.

Bolz DD, Sundsbak RS, Ma Y, Akira S, Kirschning CJ, Zachary JF, Weis JH, Weis JJ.

J Immunol. 2004 Aug 1;173(3):2003-10.

8.

Outer surface protein OspC is an antiphagocytic factor that protects Borrelia burgdorferi from phagocytosis by macrophages.

Carrasco SE, Troxell B, Yang Y, Brandt SL, Li H, Sandusky GE, Condon KW, Serezani CH, Yang XF.

Infect Immun. 2015 Dec;83(12):4848-60. doi: 10.1128/IAI.01215-15. Epub 2015 Oct 5.

9.
10.

Spirochete antigens persist near cartilage after murine Lyme borreliosis therapy.

Bockenstedt LK, Gonzalez DG, Haberman AM, Belperron AA.

J Clin Invest. 2012 Jul;122(7):2652-60. doi: 10.1172/JCI58813. Epub 2012 Jun 25.

11.

Genetic variation at the vlsE locus of Borrelia burgdorferi within ticks and mice over the course of a single transmission cycle.

Ohnishi J, Schneider B, Messer WB, Piesman J, de Silva AM.

J Bacteriol. 2003 Aug;185(15):4432-41.

12.

OspC facilitates Borrelia burgdorferi invasion of Ixodes scapularis salivary glands.

Pal U, Yang X, Chen M, Bockenstedt LK, Anderson JF, Flavell RA, Norgard MV, Fikrig E.

J Clin Invest. 2004 Jan;113(2):220-30.

13.

A surface enolase participates in Borrelia burgdorferi-plasminogen interaction and contributes to pathogen survival within feeding ticks.

Nogueira SV, Smith AA, Qin JH, Pal U.

Infect Immun. 2012 Jan;80(1):82-90. doi: 10.1128/IAI.05671-11. Epub 2011 Oct 24.

14.

Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.

15.

Infection of mice with lyme disease spirochetes constitutively producing outer surface proteins a and B.

Strother KO, Hodzic E, Barthold SW, de Silva AM.

Infect Immun. 2007 Jun;75(6):2786-94. Epub 2007 Mar 19.

16.
17.

Molecular adaptation of Borrelia burgdorferi in the murine host.

Liang FT, Nelson FK, Fikrig E.

J Exp Med. 2002 Jul 15;196(2):275-80.

18.

Borrelia burgdorferi adhere to blood vessels in the dura mater and are associated with increased meningeal T cells during murine disseminated borreliosis.

Divan A, Casselli T, Narayanan SA, Mukherjee S, Zawieja DC, Watt JA, Brissette CA, Newell-Rogers MK.

PLoS One. 2018 May 3;13(5):e0196893. doi: 10.1371/journal.pone.0196893. eCollection 2018.

19.

Functional analysis of the Borrelia burgdorferi bba64 gene product in murine infection via tick infestation.

Patton TG, Dietrich G, Dolan MC, Piesman J, Carroll JA, Gilmore RD Jr.

PLoS One. 2011 May 3;6(5):e19536. doi: 10.1371/journal.pone.0019536.

20.

The caspase 1 inflammasome is not required for control of murine Lyme borreliosis.

Liu N, Belperron AA, Booth CJ, Bockenstedt LK.

Infect Immun. 2009 Aug;77(8):3320-7. doi: 10.1128/IAI.00100-09. Epub 2009 Jun 1.

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