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Alkylation base damage is converted into repairable double-strand breaks and complex intermediates in G2 cells lacking AP endonuclease.

Ma W, Westmoreland JW, Gordenin DA, Resnick MA.

PLoS Genet. 2011 Apr;7(4):e1002059. doi: 10.1371/journal.pgen.1002059. Epub 2011 Apr 28.


RAD50 is required for efficient initiation of resection and recombinational repair at random, gamma-induced double-strand break ends.

Westmoreland J, Ma W, Yan Y, Van Hulle K, Malkova A, Resnick MA.

PLoS Genet. 2009 Sep;5(9):e1000656. doi: 10.1371/journal.pgen.1000656. Epub 2009 Sep 18.


Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease.

Westmoreland JW, Resnick MA.

PLoS Genet. 2013 Mar;9(3):e1003420. doi: 10.1371/journal.pgen.1003420. Epub 2013 Mar 28.


Apn1 and Apn2 endonucleases prevent accumulation of repair-associated DNA breaks in budding yeast as revealed by direct chromosomal analysis.

Ma W, Resnick MA, Gordenin DA.

Nucleic Acids Res. 2008 Apr;36(6):1836-46. doi: 10.1093/nar/gkm1148. Epub 2008 Feb 11.


Involvement of two endonuclease III homologs in the base excision repair pathway for the processing of DNA alkylation damage in Saccharomyces cerevisiae.

Hanna M, Chow BL, Morey NJ, Jinks-Robertson S, Doetsch PW, Xiao W.

DNA Repair (Amst). 2004 Jan 5;3(1):51-9.


Rad51 replication fork recruitment is required for DNA damage tolerance.

González-Prieto R, Muñoz-Cabello AM, Cabello-Lobato MJ, Prado F.

EMBO J. 2013 May 2;32(9):1307-21. doi: 10.1038/emboj.2013.73. Epub 2013 Apr 5.


Homologous recombination protects mammalian cells from replication-associated DNA double-strand breaks arising in response to methyl methanesulfonate.

Nikolova T, Ensminger M, Löbrich M, Kaina B.

DNA Repair (Amst). 2010 Oct 5;9(10):1050-63. doi: 10.1016/j.dnarep.2010.07.005. Epub 2010 Aug 13.


Ctf4 Prevents Genome Rearrangements by Suppressing DNA Double-Strand Break Formation and Its End Resection at Arrested Replication Forks.

Sasaki M, Kobayashi T.

Mol Cell. 2017 May 18;66(4):533-545.e5. doi: 10.1016/j.molcel.2017.04.020.


The repair of DNA methylation damage in Saccharomyces cerevisiae.

Xiao W, Chow BL, Rathgeber L.

Curr Genet. 1996 Dec;30(6):461-8.


The role of DNA double-strand breaks in spontaneous homologous recombination in S. cerevisiae.

Lettier G, Feng Q, de Mayolo AA, Erdeniz N, Reid RJ, Lisby M, Mortensen UH, Rothstein R.

PLoS Genet. 2006 Nov 10;2(11):e194. Epub 2006 Oct 5.


A postincision-deficient TFIIH causes replication fork breakage and uncovers alternative Rad51- or Pol32-mediated restart mechanisms.

Moriel-Carretero M, Aguilera A.

Mol Cell. 2010 Mar 12;37(5):690-701. doi: 10.1016/j.molcel.2010.02.008.


Histone H3K56 acetylation, Rad52, and non-DNA repair factors control double-strand break repair choice with the sister chromatid.

Muñoz-Galván S, Jimeno S, Rothstein R, Aguilera A.

PLoS Genet. 2013;9(1):e1003237. doi: 10.1371/journal.pgen.1003237. Epub 2013 Jan 24.


Ctf18 is required for homologous recombination-mediated double-strand break repair.

Ogiwara H, Ohuchi T, Ui A, Tada S, Enomoto T, Seki M.

Nucleic Acids Res. 2007;35(15):4989-5000. Epub 2007 Jul 18.


Functional interplay between the 53BP1-ortholog Rad9 and the Mre11 complex regulates resection, end-tethering and repair of a double-strand break.

Ferrari M, Dibitetto D, De Gregorio G, Eapen VV, Rawal CC, Lazzaro F, Tsabar M, Marini F, Haber JE, Pellicioli A.

PLoS Genet. 2015 Jan 8;11(1):e1004928. doi: 10.1371/journal.pgen.1004928. eCollection 2015 Jan.


Mre11-Rad50-Xrs2 and Sae2 promote 5' strand resection of DNA double-strand breaks.

Nicolette ML, Lee K, Guo Z, Rani M, Chow JM, Lee SE, Paull TT.

Nat Struct Mol Biol. 2010 Dec;17(12):1478-85. doi: 10.1038/nsmb.1957. Epub 2010 Nov 21.

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