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Homeostatic adaptation to endoplasmic reticulum stress depends on Ire1 kinase activity.

Rubio C, Pincus D, Korennykh A, Schuck S, El-Samad H, Walter P.

J Cell Biol. 2011 Apr 4;193(1):171-84. doi: 10.1083/jcb.201007077.


Attenuation of yeast UPR is essential for survival and is mediated by IRE1 kinase.

Chawla A, Chakrabarti S, Ghosh G, Niwa M.

J Cell Biol. 2011 Apr 4;193(1):41-50. doi: 10.1083/jcb.201008071.


Specificity in endoplasmic reticulum-stress signaling in yeast entails a step-wise engagement of HAC1 mRNA to clusters of the stress sensor Ire1.

van Anken E, Pincus D, Coyle S, Aragón T, Osman C, Lari F, Gómez Puerta S, Korennykh AV, Walter P.

Elife. 2014 Dec 30;3:e05031. doi: 10.7554/eLife.05031.


ER stress signaling by regulated splicing: IRE1/HAC1/XBP1.

Back SH, Schröder M, Lee K, Zhang K, Kaufman RJ.

Methods. 2005 Apr;35(4):395-416.


Membrane aberrancy and unfolded proteins activate the endoplasmic reticulum stress sensor Ire1 in different ways.

Promlek T, Ishiwata-Kimata Y, Shido M, Sakuramoto M, Kohno K, Kimata Y.

Mol Biol Cell. 2011 Sep;22(18):3520-32. doi: 10.1091/mbc.E11-04-0295.


Messenger RNA targeting to endoplasmic reticulum stress signalling sites.

Aragón T, van Anken E, Pincus D, Serafimova IM, Korennykh AV, Rubio CA, Walter P.

Nature. 2009 Feb 5;457(7230):736-40. doi: 10.1038/nature07641.


A novel role for protein kinase Kin2 in regulating HAC1 mRNA translocation, splicing, and translation.

Anshu A, Mannan MA, Chakraborty A, Chakrabarti S, Dey M.

Mol Cell Biol. 2015 Jan;35(1):199-210. doi: 10.1128/MCB.00981-14.


F-actin and a type-II myosin are required for efficient clustering of the ER stress sensor Ire1.

Ishiwata-Kimata Y, Yamamoto YH, Takizawa K, Kohno K, Kimata Y.

Cell Struct Funct. 2013 Jul 6;38(2):135-43.


Loss of subcellular lipid transport due to ARV1 deficiency disrupts organelle homeostasis and activates the unfolded protein response.

Shechtman CF, Henneberry AL, Seimon TA, Tinkelenberg AH, Wilcox LJ, Lee E, Fazlollahi M, Munkacsi AB, Bussemaker HJ, Tabas I, Sturley SL.

J Biol Chem. 2011 Apr 8;286(14):11951-9. doi: 10.1074/jbc.M110.215038.


IRE1- and HAC1-independent transcriptional regulation in the unfolded protein response of yeast.

Schröder M, Clark R, Kaufman RJ.

Mol Microbiol. 2003 Aug;49(3):591-606.


Bypassing a kinase activity with an ATP-competitive drug.

Papa FR, Zhang C, Shokat K, Walter P.

Science. 2003 Nov 28;302(5650):1533-7.


PMT1 deficiency enhances basal UPR activity and extends replicative lifespan of Saccharomyces cerevisiae.

Cui HJ, Liu XG, McCormick M, Wasko BM, Zhao W, He X, Yuan Y, Fang BX, Sun XR, Kennedy BK, Suh Y, Zhou ZJ, Kaeberlein M, Feng WL.

Age (Dordr). 2015 Jun;37(3):9788. doi: 10.1007/s11357-015-9788-7.


Identification of mitogen-activated protein kinase signaling pathways that confer resistance to endoplasmic reticulum stress in Saccharomyces cerevisiae.

Chen Y, Feldman DE, Deng C, Brown JA, De Giacomo AF, Gaw AF, Shi G, Le QT, Brown JM, Koong AC.

Mol Cancer Res. 2005 Dec;3(12):669-77.


Measuring signaling by the unfolded protein response.

Cox DJ, Strudwick N, Ali AA, Paton AW, Paton JC, Schröder M.

Methods Enzymol. 2011;491:261-92. doi: 10.1016/B978-0-12-385928-0.00015-8.


The yeast Rab GTPase Ypt1 modulates unfolded protein response dynamics by regulating the stability of HAC1 RNA.

Tsvetanova NG, Riordan DP, Brown PO.

PLoS Genet. 2012;8(7):e1002862. doi: 10.1371/journal.pgen.1002862.


Ricin inhibits activation of the unfolded protein response by preventing splicing of the HAC1 mRNA.

Parikh BA, Tortora A, Li XP, Tumer NE.

J Biol Chem. 2008 Mar 7;283(10):6145-53. doi: 10.1074/jbc.M707981200.

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