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Items: 1 to 20 of 95

1.

Integrins as triggers of Epstein-Barr virus fusion and epithelial cell infection.

Hutt-Fletcher LM, Chesnokova LS.

Virulence. 2010 Sep-Oct;1(5):395-8. doi: 10.4161/viru.1.5.12546. Review.

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Epstein-Barr virus glycoprotein gB and gHgL can mediate fusion and entry in trans, and heat can act as a partial surrogate for gHgL and trigger a conformational change in gB.

Chesnokova LS, Ahuja MK, Hutt-Fletcher LM.

J Virol. 2014 Nov;88(21):12193-201. doi: 10.1128/JVI.01597-14. Epub 2014 Aug 20.

4.

Fusion of epithelial cells by Epstein-Barr virus proteins is triggered by binding of viral glycoproteins gHgL to integrins alphavbeta6 or alphavbeta8.

Chesnokova LS, Nishimura SL, Hutt-Fletcher LM.

Proc Natl Acad Sci U S A. 2009 Dec 1;106(48):20464-9. doi: 10.1073/pnas.0907508106. Epub 2009 Nov 17. Erratum in: Proc Natl Acad Sci U S A. 2010 Feb 16;107(7):3275.

5.

Inhibition of EBV-mediated membrane fusion by anti-gHgL antibodies.

Sathiyamoorthy K, Jiang J, Möhl BS, Chen J, Zhou ZH, Longnecker R, Jardetzky TS.

Proc Natl Acad Sci U S A. 2017 Oct 10;114(41):E8703-E8710. doi: 10.1073/pnas.1704661114. Epub 2017 Sep 22.

6.

Membrane anchoring of Epstein-Barr virus gp42 inhibits fusion with B cells even with increased flexibility allowed by engineered spacers.

Rowe CL, Chen J, Jardetzky TS, Longnecker R.

MBio. 2015 Jan 6;6(1). pii: e02285-14. doi: 10.1128/mBio.02285-14.

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Use of gHgL for attachment of Epstein-Barr virus to epithelial cells compromises infection.

Borza CM, Morgan AJ, Turk SM, Hutt-Fletcher LM.

J Virol. 2004 May;78(10):5007-14.

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Assembly and architecture of the EBV B cell entry triggering complex.

Sathiyamoorthy K, Jiang J, Hu YX, Rowe CL, Möhl BS, Chen J, Jiang W, Mellins ED, Longnecker R, Zhou ZH, Jardetzky TS.

PLoS Pathog. 2014 Aug 21;10(8):e1004309. doi: 10.1371/journal.ppat.1004309. eCollection 2014 Aug.

10.

The KGD motif of Epstein-Barr virus gH/gL is bifunctional, orchestrating infection of B cells and epithelial cells.

Chen J, Rowe CL, Jardetzky TS, Longnecker R.

MBio. 2012 Jan 3;3(1). pii: e00290-11. doi: 10.1128/mBio.00290-11. Print 2012.

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Epstein-Barr Virus Fusion with Epithelial Cells Triggered by gB Is Restricted by a gL Glycosylation Site.

Möhl BS, Chen J, Park SJ, Jardetzky TS, Longnecker R.

J Virol. 2017 Nov 14;91(23). pii: e01255-17. doi: 10.1128/JVI.01255-17. Print 2017 Dec 1.

14.

Epstein-Barr virus gH is essential for penetration of B cells but also plays a role in attachment of virus to epithelial cells.

Molesworth SJ, Lake CM, Borza CM, Turk SM, Hutt-Fletcher LM.

J Virol. 2000 Jul;74(14):6324-32.

15.

αvβ6- and αvβ8-integrins serve as interchangeable receptors for HSV gH/gL to promote endocytosis and activation of membrane fusion.

Gianni T, Salvioli S, Chesnokova LS, Hutt-Fletcher LM, Campadelli-Fiume G.

PLoS Pathog. 2013;9(12):e1003806. doi: 10.1371/journal.ppat.1003806. Epub 2013 Dec 19.

16.

Structural basis for Epstein-Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins.

Sathiyamoorthy K, Hu YX, Möhl BS, Chen J, Longnecker R, Jardetzky TS.

Nat Commun. 2016 Dec 8;7:13557. doi: 10.1038/ncomms13557.

17.

The COMPLEXity in herpesvirus entry.

Sathiyamoorthy K, Chen J, Longnecker R, Jardetzky TS.

Curr Opin Virol. 2017 Jun;24:97-104. doi: 10.1016/j.coviro.2017.04.006. Epub 2017 May 21. Review.

PMID:
28538165
18.

Structural and Mechanistic Insights into the Tropism of Epstein-Barr Virus.

Möhl BS, Chen J, Sathiyamoorthy K, Jardetzky TS, Longnecker R.

Mol Cells. 2016 Apr 30;39(4):286-91. doi: 10.14348/molcells.2016.0066. Epub 2016 Apr 6. Review.

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The conserved disulfide bond within domain II of Epstein-Barr virus gH has divergent roles in membrane fusion with epithelial cells and B cells.

Möhl BS, Sathiyamoorthy K, Jardetzky TS, Longnecker R.

J Virol. 2014 Dec;88(23):13570-9. doi: 10.1128/JVI.02272-14. Epub 2014 Sep 17.

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