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Items: 1 to 20 of 105

1.

Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis.

Milanese M, Zappettini S, Onofri F, Musazzi L, Tardito D, Bonifacino T, Messa M, Racagni G, Usai C, Benfenati F, Popoli M, Bonanno G.

J Neurochem. 2011 Mar;116(6):1028-42. doi: 10.1111/j.1471-4159.2010.07155.x. Epub 2011 Jan 20.

2.

Glutamate release induced by activation of glycine and GABA transporters in spinal cord is enhanced in a mouse model of amyotrophic lateral sclerosis.

Raiteri L, Zappettini S, Stigliani S, Paluzzi S, Raiteri M, Bonanno G.

Neurotoxicology. 2005 Oct;26(5):883-92.

PMID:
15885796
3.

Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.

Neurobiol Dis. 2014 Apr;64:48-59. doi: 10.1016/j.nbd.2013.11.006. Epub 2013 Dec 19.

4.

Group I metabotropic glutamate autoreceptors induce abnormal glutamate exocytosis in a mouse model of amyotrophic lateral sclerosis.

Giribaldi F, Milanese M, Bonifacino T, Anna Rossi PI, Di Prisco S, Pittaluga A, Tacchetti C, Puliti A, Usai C, Bonanno G.

Neuropharmacology. 2013 Mar;66:253-63. doi: 10.1016/j.neuropharm.2012.05.018. Epub 2012 May 23.

PMID:
22634363
5.

Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: a mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis.

Milanese M, Bonifacino T, Fedele E, Rebosio C, Cattaneo L, Benfenati F, Usai C, Bonanno G.

Neurobiol Dis. 2015 Feb;74:314-24. doi: 10.1016/j.nbd.2014.12.004. Epub 2014 Dec 10.

PMID:
25497732
6.

Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease.

Bonifacino T, Musazzi L, Milanese M, Seguini M, Marte A, Gallia E, Cattaneo L, Onofri F, Popoli M, Bonanno G.

Neurobiol Dis. 2016 Nov;95:122-33. doi: 10.1016/j.nbd.2016.07.011. Epub 2016 Jul 16.

PMID:
27425885
7.

Excessive and precocious glutamate release in a mouse model of amyotrophic lateral sclerosis.

Raiteri L, Stigliani S, Zappettini S, Mercuri NB, Raiteri M, Bonanno G.

Neuropharmacology. 2004 May;46(6):782-92.

PMID:
15033338
9.

In vitro activation of GAT1 transporters expressed in spinal cord gliosomes stimulates glutamate release that is abnormally elevated in the SOD1/G93A(+) mouse model of amyotrophic lateral sclerosis.

Milanese M, Zappettini S, Jacchetti E, Bonifacino T, Cervetto C, Usai C, Bonanno G.

J Neurochem. 2010 Apr;113(2):489-501. doi: 10.1111/j.1471-4159.2010.06628.x. Epub 2010 Feb 1.

10.

Transcriptional profiling in the lumbar spinal cord of a mouse model of amyotrophic lateral sclerosis: a role for wild-type superoxide dismutase 1 in sporadic disease?

D'Arrigo A, Colavito D, Peña-Altamira E, Fabris M, Dam M, Contestabile A, Leon A.

J Mol Neurosci. 2010 Jul;41(3):404-15. doi: 10.1007/s12031-010-9332-2. Epub 2010 Feb 23. Erratum in: J Mol Neurosci. 2010 Jul;41(3):416.

PMID:
20177826
11.
12.

GAB(A) receptors present higher affinity and modified subunit composition in spinal motor neurons from a genetic model of amyotrophic lateral sclerosis.

Carunchio I, Mollinari C, Pieri M, Merlo D, Zona C.

Eur J Neurosci. 2008 Oct;28(7):1275-85. doi: 10.1111/j.1460-9568.2008.06436.x.

PMID:
18973555
13.

Nordihydroguaiaretic acid increases glutamate uptake in vitro and in vivo: therapeutic implications for amyotrophic lateral sclerosis.

Boston-Howes W, Williams EO, Bogush A, Scolere M, Pasinelli P, Trotti D.

Exp Neurol. 2008 Sep;213(1):229-37. doi: 10.1016/j.expneurol.2008.06.010. Epub 2008 Jun 24.

14.

Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1(G93A) mouse, a model of amyotrophic lateral sclerosis.

Nutini M, Frazzini V, Marini C, Spalloni A, Sensi SL, Longone P.

Neuropharmacology. 2011 Jun;60(7-8):1200-8. doi: 10.1016/j.neuropharm.2010.11.001. Epub 2010 Nov 5.

PMID:
21056589
15.

Regulation of system x(c)- in the SOD1-G93A mouse model of ALS.

Albano R, Liu X, Lobner D.

Exp Neurol. 2013 Dec;250:69-73. doi: 10.1016/j.expneurol.2013.09.008. Epub 2013 Sep 13.

PMID:
24041987
16.

Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.

Nardo G, Iennaco R, Fusi N, Heath PR, Marino M, Trolese MC, Ferraiuolo L, Lawrence N, Shaw PJ, Bendotti C.

Brain. 2013 Nov;136(Pt 11):3305-32. doi: 10.1093/brain/awt250. Epub 2013 Sep 24.

PMID:
24065725
17.

Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.

Li QX, Mok SS, Laughton KM, McLean CA, Volitakis I, Cherny RA, Cheung NS, White AR, Masters CL.

Aging Cell. 2006 Apr;5(2):153-65.

18.

Transgenic SOD1 G93A mice develop reduced GLT-1 in spinal cord without alterations in cerebrospinal fluid glutamate levels.

Bendotti C, Tortarolo M, Suchak SK, Calvaresi N, Carvelli L, Bastone A, Rizzi M, Rattray M, Mennini T.

J Neurochem. 2001 Nov;79(4):737-46.

19.

Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis.

Chang Q, Martin LJ.

J Neurosci. 2011 Feb 23;31(8):2815-27. doi: 10.1523/JNEUROSCI.2475-10.2011.

20.

GLT-1 glutamate transporter levels are unchanged in mice expressing G93A human mutant SOD1.

Deitch JS, Alexander GM, Del Valle L, Heiman-Patterson TD.

J Neurol Sci. 2002 Jan 15;193(2):117-26.

PMID:
11790392

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