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Items: 1 to 20 of 102

1.

Invasion of eukaryotic cells by Borrelia burgdorferi requires β(1) integrins and Src kinase activity.

Wu J, Weening EH, Faske JB, Höök M, Skare JT.

Infect Immun. 2011 Mar;79(3):1338-48. doi: 10.1128/IAI.01188-10. Epub 2010 Dec 20.

2.

Inactivation of the fibronectin-binding adhesin gene bbk32 significantly attenuates the infectivity potential of Borrelia burgdorferi.

Seshu J, Esteve-Gassent MD, Labandeira-Rey M, Kim JH, Trzeciakowski JP, Höök M, Skare JT.

Mol Microbiol. 2006 Mar;59(5):1591-601.

3.

Phagocytic Receptors Activate Syk and Src Signaling during Borrelia burgdorferi Phagocytosis.

Killpack TL, Ballesteros M, Bunnell SC, Bedugnis A, Kobzik L, Hu LT, Petnicki-Ocwieja T.

Infect Immun. 2017 Sep 20;85(10). pii: e00004-17. doi: 10.1128/IAI.00004-17. Print 2017 Oct.

4.

Invasion of human neuronal and glial cells by an infectious strain of Borrelia burgdorferi.

Livengood JA, Gilmore RD Jr.

Microbes Infect. 2006 Nov-Dec;8(14-15):2832-40. Epub 2006 Sep 22. Erratum in: Microbes Infect. 2015 Jun;17(6):e1.

PMID:
17045505
6.

Invasion of human skin fibroblasts by the Lyme disease spirochete, Borrelia burgdorferi.

Klempner MS, Noring R, Rogers RA.

J Infect Dis. 1993 May;167(5):1074-81.

PMID:
8486939
7.

BB0744 Affects Tissue Tropism and Spatial Distribution of Borrelia burgdorferi.

Wager B, Shaw DK, Groshong AM, Blevins JS, Skare JT.

Infect Immun. 2015 Sep;83(9):3693-703. doi: 10.1128/IAI.00828-15. Epub 2015 Jul 6.

8.

Immune evasion of Borrelia burgdorferi: insufficient killing of the pathogens by complement and antibody.

Kraiczy P, Skerka C, Kirschfink M, Zipfel PF, Brade V.

Int J Med Microbiol. 2002 Jun;291 Suppl 33:141-6. Review.

PMID:
12141738
9.

Borrelia burgdorferi lacking BBK32, a fibronectin-binding protein, retains full pathogenicity.

Li X, Liu X, Beck DS, Kantor FS, Fikrig E.

Infect Immun. 2006 Jun;74(6):3305-13.

10.

Arthropod- and host-specific Borrelia burgdorferi bbk32 expression and the inhibition of spirochete transmission.

Fikrig E, Feng W, Barthold SW, Telford SR 3rd, Flavell RA.

J Immunol. 2000 May 15;164(10):5344-51.

11.

OspC facilitates Borrelia burgdorferi invasion of Ixodes scapularis salivary glands.

Pal U, Yang X, Chen M, Bockenstedt LK, Anderson JF, Flavell RA, Norgard MV, Fikrig E.

J Clin Invest. 2004 Jan;113(2):220-30.

12.

Integrin alpha IIb beta 3 mediates binding of the Lyme disease agent Borrelia burgdorferi to human platelets.

Coburn J, Leong JM, Erban JK.

Proc Natl Acad Sci U S A. 1993 Aug 1;90(15):7059-63.

13.

The Borrelia burgdorferi integrin ligand P66 affects gene expression by human cells in culture.

LaFrance ME, Pierce JV, Antonara S, Coburn J.

Infect Immun. 2011 Aug;79(8):3249-61. doi: 10.1128/IAI.05122-11. Epub 2011 May 16.

14.

Borrelia burgdorferi lacking DbpBA exhibits an early survival defect during experimental infection.

Weening EH, Parveen N, Trzeciakowski JP, Leong JM, Höök M, Skare JT.

Infect Immun. 2008 Dec;76(12):5694-705. doi: 10.1128/IAI.00690-08. Epub 2008 Sep 22.

15.

Borrelia burgdorferi, an extracellular pathogen, circumvents osteopontin in inducing an inflammatory cytokine response.

Craig-Mylius K, Weber GF, Coburn J, Glickstein L.

J Leukoc Biol. 2005 May;77(5):710-8. Epub 2005 Feb 4.

PMID:
15695554
16.

Suppression of Long-Lived Humoral Immunity Following Borrelia burgdorferi Infection.

Elsner RA, Hastey CJ, Olsen KJ, Baumgarth N.

PLoS Pathog. 2015 Jul 2;11(7):e1004976. doi: 10.1371/journal.ppat.1004976. eCollection 2015 Jul.

17.

Temporal expression analysis of the Borrelia burgdorferi paralogous gene family 54 genes BBA64, BBA65, and BBA66 during persistent infection in mice.

Gilmore RD Jr, Howison RR, Schmit VL, Nowalk AJ, Clifton DR, Nolder C, Hughes JL, Carroll JA.

Infect Immun. 2007 Jun;75(6):2753-64. Epub 2007 Mar 19.

18.
19.

Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.

20.

Endothelial cells and fibroblasts amplify the arthritogenic type I IFN response in murine Lyme disease and are major sources of chemokines in Borrelia burgdorferi-infected joint tissue.

Lochhead RB, Sonderegger FL, Ma Y, Brewster JE, Cornwall D, Maylor-Hagen H, Miller JC, Zachary JF, Weis JH, Weis JJ.

J Immunol. 2012 Sep 1;189(5):2488-501. doi: 10.4049/jimmunol.1201095. Epub 2012 Jul 30.

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