Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 99

1.

Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines.

Zhao B, Mar JC, Maruo S, Lee S, Gewurz BE, Johannsen E, Holton K, Rubio R, Takada K, Quackenbush J, Kieff E.

Proc Natl Acad Sci U S A. 2011 Jan 4;108(1):337-42. doi: 10.1073/pnas.1017419108. Epub 2010 Dec 20.

2.

Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites.

Wang A, Welch R, Zhao B, Ta T, KeleĊŸ S, Johannsen E.

J Virol. 2015 Dec 30;90(6):2906-19. doi: 10.1128/JVI.02737-15.

3.

Epstein-Barr virus nuclear protein EBNA3C residues critical for maintaining lymphoblastoid cell growth.

Maruo S, Wu Y, Ito T, Kanda T, Kieff ED, Takada K.

Proc Natl Acad Sci U S A. 2009 Mar 17;106(11):4419-24. doi: 10.1073/pnas.0813134106. Epub 2009 Feb 23.

4.

Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression.

Maruo S, Zhao B, Johannsen E, Kieff E, Zou J, Takada K.

Proc Natl Acad Sci U S A. 2011 Feb 1;108(5):1919-24. doi: 10.1073/pnas.1019599108. Epub 2011 Jan 18.

5.

Epstein-Barr virus nuclear protein 3C domains necessary for lymphoblastoid cell growth: interaction with RBP-Jkappa regulates TCL1.

Lee S, Sakakibara S, Maruo S, Zhao B, Calderwood MA, Holthaus AM, Lai CY, Takada K, Kieff E, Johannsen E.

J Virol. 2009 Dec;83(23):12368-77. doi: 10.1128/JVI.01403-09. Epub 2009 Sep 23.

6.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.

Jiang S, Willox B, Zhou H, Holthaus AM, Wang A, Shi TT, Maruo S, Kharchenko PV, Johannsen EC, Kieff E, Zhao B.

Proc Natl Acad Sci U S A. 2014 Jan 7;111(1):421-6. doi: 10.1073/pnas.1321704111. Epub 2013 Dec 16.

7.

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Skalska L, White RE, Parker GA, Turro E, Sinclair AJ, Paschos K, Allday MJ.

PLoS Pathog. 2013 Feb;9(2):e1003187. doi: 10.1371/journal.ppat.1003187. Epub 2013 Feb 21. Erratum in: PLoS Pathog. 2013 Mar; 9(3): doi:10.1371/annotation/2c4b89c1-6d4a-4bf3-9467-9367227d7e74. Turro, Ernest [added].

8.
9.

Epstein-Barr virus nuclear antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis.

Cai Q, Guo Y, Xiao B, Banerjee S, Saha A, Lu J, Glisovic T, Robertson ES.

PLoS Pathog. 2011 Dec;7(12):e1002418. doi: 10.1371/journal.ppat.1002418. Epub 2011 Dec 8.

10.

Epstein-Barr virus nuclear protein 3C binds to the N-terminal (NTD) and beta trefoil domains (BTD) of RBP/CSL; only the NTD interaction is essential for lymphoblastoid cell growth.

Calderwood MA, Lee S, Holthaus AM, Blacklow SC, Kieff E, Johannsen E.

Virology. 2011 May 25;414(1):19-25. doi: 10.1016/j.virol.2011.02.018. Epub 2011 Mar 26.

11.

Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.

Schmidt SC, Jiang S, Zhou H, Willox B, Holthaus AM, Kharchenko PV, Johannsen EC, Kieff E, Zhao B.

Proc Natl Acad Sci U S A. 2015 Jan 13;112(2):554-9. doi: 10.1073/pnas.1422580112. Epub 2014 Dec 24.

12.

EBNA3A association with RBP-Jkappa down-regulates c-myc and Epstein-Barr virus-transformed lymphoblast growth.

Cooper A, Johannsen E, Maruo S, Cahir-McFarland E, Illanes D, Davidson D, Kieff E.

J Virol. 2003 Jan;77(2):999-1010.

13.

The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth.

Ohashi M, Holthaus AM, Calderwood MA, Lai CY, Krastins B, Sarracino D, Johannsen E.

PLoS Pathog. 2015 Apr 9;11(4):e1004822. doi: 10.1371/journal.ppat.1004822. eCollection 2015 Apr.

14.

Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

Skalska L, White RE, Franz M, Ruhmann M, Allday MJ.

PLoS Pathog. 2010 Jun 10;6(6):e1000951. doi: 10.1371/journal.ppat.1000951.

15.

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

Kalchschmidt JS, Gillman AC, Paschos K, Bazot Q, Kempkes B, Allday MJ.

PLoS Pathog. 2016 Jan 11;12(1):e1005383. doi: 10.1371/journal.ppat.1005383. eCollection 2016 Jan.

16.

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth.

Zhao B, Zou J, Wang H, Johannsen E, Peng CW, Quackenbush J, Mar JC, Morton CC, Freedman ML, Blacklow SC, Aster JC, Bernstein BE, Kieff E.

Proc Natl Acad Sci U S A. 2011 Sep 6;108(36):14902-7. doi: 10.1073/pnas.1108892108. Epub 2011 Jul 11.

17.
18.
20.

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Bazot Q, Paschos K, Skalska L, Kalchschmidt JS, Parker GA, Allday MJ.

PLoS Pathog. 2015 Jul 8;11(7):e1005031. doi: 10.1371/journal.ppat.1005031. eCollection 2015 Jul.

Supplemental Content

Support Center