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Relationship between neuropathology and disease progression in the SOD1(G93A) ALS mouse.

Yang WW, Sidman RL, Taksir TV, Treleaven CM, Fidler JA, Cheng SH, Dodge JC, Shihabuddin LS.

Exp Neurol. 2011 Feb;227(2):287-95. doi: 10.1016/j.expneurol.2010.11.019.


Influence of methylene blue on microglia-induced inflammation and motor neuron degeneration in the SOD1(G93A) model for ALS.

Dibaj P, Zschüntzsch J, Steffens H, Scheffel J, Göricke B, Weishaupt JH, Le Meur K, Kirchhoff F, Hanisch UK, Schomburg ED, Neusch C.

PLoS One. 2012;7(8):e43963. doi: 10.1371/journal.pone.0043963.


Ablation of P2X7 receptor exacerbates gliosis and motoneuron death in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.

Apolloni S, Amadio S, Montilli C, Volonté C, D'Ambrosi N.

Hum Mol Genet. 2013 Oct 15;22(20):4102-16. doi: 10.1093/hmg/ddt259.


Effect of thymic stimulation of CD4+ T cell expansion on disease onset and progression in mutant SOD1 mice.

Sheean RK, Weston RH, Perera ND, D'Amico A, Nutt SL, Turner BJ.

J Neuroinflammation. 2015 Feb 27;12:40. doi: 10.1186/s12974-015-0254-3.


In vivo quantification of spinal and bulbar motor neuron degeneration in the G93A-SOD1 transgenic mouse model of ALS by T2 relaxation time and apparent diffusion coefficient.

Niessen HG, Angenstein F, Sander K, Kunz WS, Teuchert M, Ludolph AC, Heinze HJ, Scheich H, Vielhaber S.

Exp Neurol. 2006 Oct;201(2):293-300.


Transcriptional profiling in the lumbar spinal cord of a mouse model of amyotrophic lateral sclerosis: a role for wild-type superoxide dismutase 1 in sporadic disease?

D'Arrigo A, Colavito D, Peña-Altamira E, Fabris M, Dam M, Contestabile A, Leon A.

J Mol Neurosci. 2010 Jul;41(3):404-15. doi: 10.1007/s12031-010-9332-2. Erratum in: J Mol Neurosci. 2010 Jul;41(3):416.


Hind limb muscle atrophy precedes cerebral neuronal degeneration in G93A-SOD1 mouse model of amyotrophic lateral sclerosis: a longitudinal MRI study.

Marcuzzo S, Zucca I, Mastropietro A, de Rosbo NK, Cavalcante P, Tartari S, Bonanno S, Preite L, Mantegazza R, Bernasconi P.

Exp Neurol. 2011 Sep;231(1):30-7. doi: 10.1016/j.expneurol.2011.05.007.


The proinflammatory action of microglial P2 receptors is enhanced in SOD1 models for amyotrophic lateral sclerosis.

D'Ambrosi N, Finocchi P, Apolloni S, Cozzolino M, Ferri A, Padovano V, Pietrini G, Carrì MT, Volonté C.

J Immunol. 2009 Oct 1;183(7):4648-56. doi: 10.4049/jimmunol.0901212.


Dismutase-competent SOD1 mutant accumulation in myelinating Schwann cells is not detrimental to normal or transgenic ALS model mice.

Turner BJ, Ackerley S, Davies KE, Talbot K.

Hum Mol Genet. 2010 Mar 1;19(5):815-24. doi: 10.1093/hmg/ddp550.


Inducible nitric oxide synthase up-regulation in a transgenic mouse model of familial amyotrophic lateral sclerosis.

Almer G, Vukosavic S, Romero N, Przedborski S.

J Neurochem. 1999 Jun;72(6):2415-25.


The human G93A-SOD1 mutation in a pre-symptomatic rat model of amyotrophic lateral sclerosis increases the vulnerability to a mild spinal cord compression.

Jokic N, Yip PK, Michael-Titus A, Priestley JV, Malaspina A.

BMC Genomics. 2010 Nov 15;11:633. doi: 10.1186/1471-2164-11-633.


Ablation of proliferating microglia does not affect motor neuron degeneration in amyotrophic lateral sclerosis caused by mutant superoxide dismutase.

Gowing G, Philips T, Van Wijmeersch B, Audet JN, Dewil M, Van Den Bosch L, Billiau AD, Robberecht W, Julien JP.

J Neurosci. 2008 Oct 8;28(41):10234-44. doi: 10.1523/JNEUROSCI.3494-08.2008.


Spinal cord pathology is ameliorated by P2X7 antagonism in a SOD1-mutant mouse model of amyotrophic lateral sclerosis.

Apolloni S, Amadio S, Parisi C, Matteucci A, Potenza RL, Armida M, Popoli P, D'Ambrosi N, Volonté C.

Dis Model Mech. 2014 Sep;7(9):1101-9. doi: 10.1242/dmm.017038.


A potential role for the p75 low-affinity neurotrophin receptor in spinal motor neuron degeneration in murine and human amyotrophic lateral sclerosis.

Lowry KS, Murray SS, McLean CA, Talman P, Mathers S, Lopes EC, Cheema SS.

Amyotroph Lateral Scler Other Motor Neuron Disord. 2001 Sep;2(3):127-34.


Up-regulation of neural and cell cycle-related microRNAs in brain of amyotrophic lateral sclerosis mice at late disease stage.

Marcuzzo S, Bonanno S, Kapetis D, Barzago C, Cavalcante P, D'Alessandro S, Mantegazza R, Bernasconi P.

Mol Brain. 2015 Jan 28;8:5. doi: 10.1186/s13041-015-0095-0.


Activation of the stress-activated MAP kinase, p38, but not JNK in cortical motor neurons during early presymptomatic stages of amyotrophic lateral sclerosis in transgenic mice.

Holasek SS, Wengenack TM, Kandimalla KK, Montano C, Gregor DM, Curran GL, Poduslo JF.

Brain Res. 2005 May 31;1045(1-2):185-98.


Immune reactivity in a mouse model of familial ALS correlates with disease progression.

Alexianu ME, Kozovska M, Appel SH.

Neurology. 2001 Oct 9;57(7):1282-9.


Molecular chaperone mediated late-stage neuroprotection in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Novoselov SS, Mustill WJ, Gray AL, Dick JR, Kanuga N, Kalmar B, Greensmith L, Cheetham ME.

PLoS One. 2013 Aug 30;8(8):e73944. doi: 10.1371/journal.pone.0073944.

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