Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 145


Aberrant CpG island hypermethylation in dysplastic nodules and early HCC of hepatitis B virus-related human multistep hepatocarcinogenesis.

Um TH, Kim H, Oh BK, Kim MS, Kim KS, Jung G, Park YN.

J Hepatol. 2011 May;54(5):939-47. doi: 10.1016/j.jhep.2010.08.021. Epub 2010 Oct 29.


Aberrant CpG island hypermethylation along multistep hepatocarcinogenesis.

Lee S, Lee HJ, Kim JH, Lee HS, Jang JJ, Kang GH.

Am J Pathol. 2003 Oct;163(4):1371-8.


High frequency of promoter hypermethylation of RASSF1A and p16 and its relationship to aflatoxin B1-DNA adduct levels in human hepatocellular carcinoma.

Zhang YJ, Ahsan H, Chen Y, Lunn RM, Wang LY, Chen SY, Lee PH, Chen CJ, Santella RM.

Mol Carcinog. 2002 Oct;35(2):85-92.


Chromosomal instability, telomere shortening, and inactivation of p21(WAF1/CIP1) in dysplastic nodules of hepatitis B virus-associated multistep hepatocarcinogenesis.

Lee YH, Oh BK, Yoo JE, Yoon SM, Choi J, Kim KS, Park YN.

Mod Pathol. 2009 Aug;22(8):1121-31. doi: 10.1038/modpathol.2009.76. Epub 2009 May 22.


Comprehensive DNA methylation analysis of hepatitis B virus genome in infected liver tissues.

Jain S, Chang TT, Chen S, Boldbaatar B, Clemens A, Lin SY, Yan R, Hu CT, Guo H, Block TM, Song W, Su YH.

Sci Rep. 2015 May 22;5:10478. doi: 10.1038/srep10478.


[Aberrant methylation of multiple genes and its clinical implication in hepatocellular carcinoma].

Lou C, Yang B, Gao YT, Wang YJ, Nie FH, Yuan Q, Zhang CL, Du Z.

Zhonghua Zhong Liu Za Zhi. 2008 Nov;30(11):831-6. Chinese.


Methylation profiling of twenty promoter-CpG islands of genes which may contribute to hepatocellular carcinogenesis.

Yu J, Ni M, Xu J, Zhang H, Gao B, Gu J, Chen J, Zhang L, Wu M, Zhen S, Zhu J.

BMC Cancer. 2002 Nov 15;2:29. Epub 2002 Nov 15.


[Chronic hepatitis B virus infection and the methylation status of p16INK4A promoter].

Zhu R, Li BZ, Ling YQ, Zhang HP, Li H, Liu Y, Hu XQ, Zhu HG.

Zhonghua Zhong Liu Za Zhi. 2007 Mar;29(3):166-70. Chinese.


Epigenetic alteration of the SOCS1 gene in hepatocellular carcinoma.

Chu PY, Yeh CM, Hsu NC, Chang YS, Chang JG, Yeh KT.

Swiss Med Wkly. 2010 Jul 15;140:w13065. doi: 10.4414/smw.2010.13065.


RASSF1A and DOK1 Promoter Methylation Levels in Hepatocellular Carcinoma, Cirrhotic and Non-Cirrhotic Liver, and Correlation with Liver Cancer in Brazilian Patients.

Ara├║jo OC, Rosa AS, Fernandes A, Niel C, Villela-Nogueira CA, Pannain V, Araujo NM.

PLoS One. 2016 Apr 14;11(4):e0153796. doi: 10.1371/journal.pone.0153796. eCollection 2016.


Aberrant promoter methylation profiles of tumor suppressor genes in hepatocellular carcinoma.

Yang B, Guo M, Herman JG, Clark DP.

Am J Pathol. 2003 Sep;163(3):1101-7.


Differential DNA methylation associated with hepatitis B virus infection in hepatocellular carcinoma.

Su PF, Lee TC, Lin PJ, Lee PH, Jeng YM, Chen CH, Liang JD, Chiou LL, Huang GT, Lee HS.

Int J Cancer. 2007 Sep 15;121(6):1257-64.


Aberrant methylation of multiple tumor suppressor genes in aging liver, chronic hepatitis, and hepatocellular carcinoma.

Nishida N, Nagasaka T, Nishimura T, Ikai I, Boland CR, Goel A.

Hepatology. 2008 Mar;47(3):908-18.


Integrative analysis of aberrant Wnt signaling in hepatitis B virus-related hepatocellular carcinoma.

Ding SL, Yang ZW, Wang J, Zhang XL, Chen XM, Lu FM.

World J Gastroenterol. 2015 May 28;21(20):6317-28. doi: 10.3748/wjg.v21.i20.6317. Review.


RASSF1A-mediated regulation of AREG via the Hippo pathway in hepatocellular carcinoma.

Ahn EY, Kim JS, Kim GJ, Park YN.

Mol Cancer Res. 2013 Jul;11(7):748-58. doi: 10.1158/1541-7786.MCR-12-0665. Epub 2013 Apr 17.


Quantitative evaluation of RASSF1A methylation in the non-lesional, regenerative and neoplastic liver.

Di Gioia S, Bianchi P, Destro A, Grizzi F, Malesci A, Laghi L, Levrero M, Morabito A, Roncalli M.

BMC Cancer. 2006 Apr 10;6:89.


Hepatitis B virus-associated multistep hepatocarcinogenesis: a stepwise increase in allelic alterations.

Lee JM, Wong CM, Ng IO.

Cancer Res. 2008 Jul 15;68(14):5988-96. doi: 10.1158/0008-5472.CAN-08-0905.

Supplemental Content

Support Center