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Items: 1 to 20 of 129

1.

Caenorhabditis elegans generates biologically relevant levels of genotoxic metabolites from aflatoxin B1 but not benzo[a]pyrene in vivo.

Leung MC, Goldstone JV, Boyd WA, Freedman JH, Meyer JN.

Toxicol Sci. 2010 Dec;118(2):444-53. doi: 10.1093/toxsci/kfq295. Epub 2010 Sep 23.

2.

Aflatoxin B₁-Induced Developmental and DNA Damage in Caenorhabditis elegans.

Feng WH, Xue KS, Tang L, Williams PL, Wang JS.

Toxins (Basel). 2016 Dec 26;9(1). pii: E9. doi: 10.3390/toxins9010009.

3.

The application of the comet assay to assess the genotoxicity of environmental pollutants in the nematode Caenorhabditis elegans.

Imanikia S, Galea F, Nagy E, Phillips DH, Stürzenbaum SR, Arlt VM.

Environ Toxicol Pharmacol. 2016 Jul;45:356-61. doi: 10.1016/j.etap.2016.06.020. Epub 2016 Jun 20.

4.

Dietary restriction modulated carcinogen-DNA adduct formation and the carcinogen-induced DNA strand breaks.

Chou MW, Chen W, Mikhailova MV, Nichols J, Weis C, Jackson CD, Hart RW, Chung KT.

Toxicol Lett. 1997 Jun 16;92(1):21-30.

PMID:
9242354
5.

High susceptibility to aflatoxin B1 and benzo[a]pyrene of BALB3T3 A31-1-1 cells expressing monkey CYP1A1.

Itoh S, Tagawa S, Sawada M, Kamataki T.

J Toxicol Sci. 1993 Aug;18(3):207-10.

PMID:
8246314
6.

Metabolism and cytotoxicity of aflatoxin b1 in cytochrome p-450-expressing human lung cells.

Van Vleet TR, Klein PJ, Coulombe RA Jr.

J Toxicol Environ Health A. 2002 Jun 28;65(12):853-67.

PMID:
12079611
7.
9.

Nucleotide excision repair genes are expressed at low levels and are not detectably inducible in Caenorhabditis elegans somatic tissues, but their function is required for normal adult life after UVC exposure.

Boyd WA, Crocker TL, Rodriguez AM, Leung MC, Lehmann DW, Freedman JH, Van Houten B, Meyer JN.

Mutat Res. 2010 Jan 5;683(1-2):57-67. doi: 10.1016/j.mrfmmm.2009.10.008.

10.

Metabolism of benzo(a)pyrene, dimethylbenzanthracene and aflatoxin B1 by camel liver microsomes.

Raza H, Montague W.

Comp Biochem Physiol Pharmacol Toxicol Endocrinol. 1994 Mar;107(3):379-86.

PMID:
8061945
11.

Metabolic activation of benzo[a]pyrene in vitro by hepatic cytochrome P450 contrasts with detoxification in vivo: experiments with hepatic cytochrome P450 reductase null mice.

Arlt VM, Stiborová M, Henderson CJ, Thiemann M, Frei E, Aimová D, Singh R, Gamboa da Costa G, Schmitz OJ, Farmer PB, Wolf CR, Phillips DH.

Carcinogenesis. 2008 Mar;29(3):656-65. doi: 10.1093/carcin/bgn002. Epub 2008 Jan 19.

PMID:
18204078
12.

Aflatoxin B1 formamidopyrimidine adducts are preferentially repaired by the nucleotide excision repair pathway in vivo.

Alekseyev YO, Hamm ML, Essigmann JM.

Carcinogenesis. 2004 Jun;25(6):1045-51. Epub 2004 Jan 23.

PMID:
14742311
13.
15.
16.

Dietary modulation of the biotransformation and genotoxicity of aflatoxin B(1).

Gross-Steinmeyer K, Eaton DL.

Toxicology. 2012 Sep 28;299(2-3):69-79. doi: 10.1016/j.tox.2012.05.016. Epub 2012 May 26. Review.

PMID:
22640941
17.
18.

Up-regulation of nucleotide excision repair in mouse lung and liver following chronic exposure to aflatoxin B₁ and its dependence on p53 genotype.

Mulder JE, Bondy GS, Mehta R, Massey TE.

Toxicol Appl Pharmacol. 2014 Mar 1;275(2):96-103. doi: 10.1016/j.taap.2013.12.016. Epub 2013 Dec 29.

PMID:
24380836
19.
20.

Biochemical basis for the extreme sensitivity of turkeys to aflatoxin B(1).

Klein PJ, Buckner R, Kelly J, Coulombe RA Jr.

Toxicol Appl Pharmacol. 2000 May 15;165(1):45-52.

PMID:
10814552

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