Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 127

1.

Rho-kinase directs Bazooka/Par-3 planar polarity during Drosophila axis elongation.

Simões Sde M, Blankenship JT, Weitz O, Farrell DL, Tamada M, Fernandez-Gonzalez R, Zallen JA.

Dev Cell. 2010 Sep 14;19(3):377-88. doi: 10.1016/j.devcel.2010.08.011.

2.

Bazooka is required for polarisation of the Drosophila anterior-posterior axis.

Doerflinger H, Vogt N, Torres IL, Mirouse V, Koch I, Nüsslein-Volhard C, St Johnston D.

Development. 2010 May;137(10):1765-73. doi: 10.1242/dev.045807.

3.

Rho GTPase and Shroom direct planar polarized actomyosin contractility during convergent extension.

Simões Sde M, Mainieri A, Zallen JA.

J Cell Biol. 2014 Feb 17;204(4):575-89. doi: 10.1083/jcb.201307070.

4.

A Par-1-Par-3-Centrosome Cell Polarity Pathway and Its Tuning for Isotropic Cell Adhesion.

Jiang T, McKinley RF, McGill MA, Angers S, Harris TJ.

Curr Biol. 2015 Oct 19;25(20):2701-8. doi: 10.1016/j.cub.2015.08.063. Epub 2015 Oct 8.

5.

aPKC phosphorylation of Bazooka defines the apical/lateral border in Drosophila epithelial cells.

Morais-de-Sá E, Mirouse V, St Johnston D.

Cell. 2010 Apr 30;141(3):509-23. doi: 10.1016/j.cell.2010.02.040.

7.

Membrane targeting of Bazooka/PAR-3 is mediated by direct binding to phosphoinositide lipids.

Krahn MP, Klopfenstein DR, Fischer N, Wodarz A.

Curr Biol. 2010 Apr 13;20(7):636-42. doi: 10.1016/j.cub.2010.01.065. Epub 2010 Mar 18.

8.

PP2A antagonizes phosphorylation of Bazooka by PAR-1 to control apical-basal polarity in dividing embryonic neuroblasts.

Krahn MP, Egger-Adam D, Wodarz A.

Dev Cell. 2009 Jun;16(6):901-8. doi: 10.1016/j.devcel.2009.04.011.

9.

Myosin-dependent junction remodelling controls planar cell intercalation and axis elongation.

Bertet C, Sulak L, Lecuit T.

Nature. 2004 Jun 10;429(6992):667-71.

PMID:
15190355
10.

Assembly of Bazooka polarity landmarks through a multifaceted membrane-association mechanism.

McKinley RF, Yu CG, Harris TJ.

J Cell Sci. 2012 Mar 1;125(Pt 5):1177-90. doi: 10.1242/jcs.091884. Epub 2012 Feb 2.

11.

Drosophila PAR-1 and 14-3-3 inhibit Bazooka/PAR-3 to establish complementary cortical domains in polarized cells.

Benton R, St Johnston D.

Cell. 2003 Dec 12;115(6):691-704. Erratum in: Cell. 2004 Jan 9;116(1):139.

12.

Patterned gene expression directs bipolar planar polarity in Drosophila.

Zallen JA, Wieschaus E.

Dev Cell. 2004 Mar;6(3):343-55.

13.

Formation of a Bazooka-Stardust complex is essential for plasma membrane polarity in epithelia.

Krahn MP, Bückers J, Kastrup L, Wodarz A.

J Cell Biol. 2010 Sep 6;190(5):751-60. doi: 10.1083/jcb.201006029.

14.
15.

Planar Cell Polarity Breaks the Symmetry of PAR Protein Distribution prior to Mitosis in Drosophila Sensory Organ Precursor Cells.

Besson C, Bernard F, Corson F, Rouault H, Reynaud E, Keder A, Mazouni K, Schweisguth F.

Curr Biol. 2015 Apr 20;25(8):1104-10. doi: 10.1016/j.cub.2015.02.073. Epub 2015 Apr 2.

16.

Bazooka and atypical protein kinase C are required to regulate oocyte differentiation in the Drosophila ovary.

Cox DN, Seyfried SA, Jan LY, Jan YN.

Proc Natl Acad Sci U S A. 2001 Dec 4;98(25):14475-80.

17.

Rap1 and Canoe/afadin are essential for establishment of apical-basal polarity in the Drosophila embryo.

Choi W, Harris NJ, Sumigray KD, Peifer M.

Mol Biol Cell. 2013 Apr;24(7):945-63. doi: 10.1091/mbc.E12-10-0736. Epub 2013 Jan 30.

18.

Par-1 and PP2A: Yin-Yang of Bazooka localization.

Choi KW, Nam SC, Mukhopadhyay B.

Fly (Austin). 2007 Jul-Aug;1(4):235-7. Epub 2007 Jul 3.

PMID:
18820442
19.

Src kinases mediate the interaction of the apical determinant Bazooka/PAR3 with STAT92E and increase signalling efficiency in Drosophila ectodermal cells.

Sotillos S, Krahn M, Espinosa-Vázquez JM, Hombría JC.

Development. 2013 Apr;140(7):1507-16. doi: 10.1242/dev.092320. Epub 2013 Mar 5.

20.

A modifier screen for Bazooka/PAR-3 interacting genes in the Drosophila embryo epithelium.

Shao W, Wu J, Chen J, Lee DM, Tishkina A, Harris TJ.

PLoS One. 2010 Apr 1;5(4):e9938. doi: 10.1371/journal.pone.0009938.

Supplemental Content

Support Center