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Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.

Ellis-Connell AL, Iempridee T, Xu I, Mertz JE.

J Virol. 2010 Oct;84(19):10329-43. doi: 10.1128/JVI.00923-10. Epub 2010 Jul 28.


Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.

Ellis AL, Wang Z, Yu X, Mertz JE.

J Virol. 2010 Jun;84(12):6139-52. doi: 10.1128/JVI.02706-09. Epub 2010 Apr 7.


ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus.

Yu X, Wang Z, Mertz JE.

PLoS Pathog. 2007 Dec;3(12):e194.


Zinc finger E-box binding factor 1 plays a central role in regulating Epstein-Barr virus (EBV) latent-lytic switch and acts as a therapeutic target in EBV-associated gastric cancer.

Zhao J, Jin H, Cheung KF, Tong JH, Zhang S, Go MY, Tian L, Kang W, Leung PP, Zeng Z, Li X, To KF, Sung JJ, Yu J.

Cancer. 2012 Feb 15;118(4):924-36. doi: 10.1002/cncr.26184. Epub 2011 Jun 29.


ZEB negatively regulates the lytic-switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Perrigoue JG, Mertz JE.

J Virol. 2003 Jan;77(1):199-207.


MicroRNA miR-BART20-5p stabilizes Epstein-Barr virus latency by directly targeting BZLF1 and BRLF1.

Jung YJ, Choi H, Kim H, Lee SK.

J Virol. 2014 Aug;88(16):9027-37. doi: 10.1128/JVI.00721-14. Epub 2014 Jun 4.


Counteracting effects of cellular Notch and Epstein-Barr virus EBNA2: implications for stromal effects on virus-host interactions.

Rowe M, Raithatha S, Shannon-Lowe C.

J Virol. 2014 Oct;88(20):12065-76. doi: 10.1128/JVI.01431-14. Epub 2014 Aug 13.


Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.

Reusch JA, Nawandar DM, Wright KL, Kenney SC, Mertz JE.

J Virol. 2015 Feb;89(3):1731-43. doi: 10.1128/JVI.02781-14. Epub 2014 Nov 19.


Latent and lytic Epstein-Barr virus replication strategies.

Tsurumi T, Fujita M, Kudoh A.

Rev Med Virol. 2005 Jan-Feb;15(1):3-15. Review.


The B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1.

Robinson AR, Kwek SS, Kenney SC.

PLoS Pathog. 2012 Feb;8(2):e1002516. doi: 10.1371/journal.ppat.1002516. Epub 2012 Feb 9.


Shutoff of BZLF1 gene expression is necessary for immortalization of primary B cells by Epstein-Barr virus.

Yu X, McCarthy PJ, Wang Z, Gorlen DA, Mertz JE.

J Virol. 2012 Aug;86(15):8086-96. doi: 10.1128/JVI.00234-12. Epub 2012 May 23.


Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Mirocha SJ, Stephany HM, Puchalski JR, Mertz JE.

J Virol. 2001 Jan;75(2):867-77.


Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication.

Li Y, Long X, Huang L, Yang M, Yuan Y, Wang Y, Delecluse HJ, Kuang E.

J Virol. 2015 Nov 4;90(2):887-903. doi: 10.1128/JVI.01921-15.


Switching of EBV cycles between latent and lytic states.

Murata T, Tsurumi T.

Rev Med Virol. 2014 May;24(3):142-53. doi: 10.1002/rmv.1780. Epub 2013 Dec 11. Review.


BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.

Woellmer A, Arteaga-Salas JM, Hammerschmidt W.

PLoS Pathog. 2012 Sep;8(9):e1002902. doi: 10.1371/journal.ppat.1002902. Epub 2012 Sep 6.


miR-190 is upregulated in Epstein-Barr Virus type I latency and modulates cellular mRNAs involved in cell survival and viral reactivation.

Cramer EM, Shao Y, Wang Y, Yuan Y.

Virology. 2014 Sep;464-465:184-95. doi: 10.1016/j.virol.2014.06.029. Epub 2014 Aug 1.


5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.

Wille CK, Nawandar DM, Henning AN, Ma S, Oetting KM, Lee D, Lambert P, Johannsen EC, Kenney SC.

Proc Natl Acad Sci U S A. 2015 Dec 29;112(52):E7257-65. doi: 10.1073/pnas.1513432112. Epub 2015 Dec 9.


BZLF1 controlled by family repeat domain induces lytic cytotoxicity in Epstein-Barr virus-positive tumor cells.

Wang H, Zhao Y, Zeng L, Tang M, El-Deeb A, Li JJ, Cao Y.

Anticancer Res. 2004 Jan-Feb;24(1):67-74.


Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency.

Robinson AR, Kwek SS, Hagemeier SR, Wille CK, Kenney SC.

J Virol. 2011 Sep;85(17):8940-53. doi: 10.1128/JVI.00569-11. Epub 2011 Jun 22.


The lytic phase of epstein-barr virus requires a viral genome with 5-methylcytosine residues in CpG sites.

Kalla M, Göbel C, Hammerschmidt W.

J Virol. 2012 Jan;86(1):447-58. doi: 10.1128/JVI.06314-11. Epub 2011 Oct 26.

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