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Items: 1 to 20 of 340

1.

Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.

Ellis-Connell AL, Iempridee T, Xu I, Mertz JE.

J Virol. 2010 Oct;84(19):10329-43. doi: 10.1128/JVI.00923-10. Epub 2010 Jul 28.

2.

Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.

Ellis AL, Wang Z, Yu X, Mertz JE.

J Virol. 2010 Jun;84(12):6139-52. doi: 10.1128/JVI.02706-09. Epub 2010 Apr 7.

3.

ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus.

Yu X, Wang Z, Mertz JE.

PLoS Pathog. 2007 Dec;3(12):e194.

4.

MicroRNA miR-BART20-5p stabilizes Epstein-Barr virus latency by directly targeting BZLF1 and BRLF1.

Jung YJ, Choi H, Kim H, Lee SK.

J Virol. 2014 Aug;88(16):9027-37. doi: 10.1128/JVI.00721-14. Epub 2014 Jun 4.

5.

Zinc finger E-box binding factor 1 plays a central role in regulating Epstein-Barr virus (EBV) latent-lytic switch and acts as a therapeutic target in EBV-associated gastric cancer.

Zhao J, Jin H, Cheung KF, Tong JH, Zhang S, Go MY, Tian L, Kang W, Leung PP, Zeng Z, Li X, To KF, Sung JJ, Yu J.

Cancer. 2012 Feb 15;118(4):924-36. doi: 10.1002/cncr.26184. Epub 2011 Jun 29.

6.

Counteracting effects of cellular Notch and Epstein-Barr virus EBNA2: implications for stromal effects on virus-host interactions.

Rowe M, Raithatha S, Shannon-Lowe C.

J Virol. 2014 Oct;88(20):12065-76. doi: 10.1128/JVI.01431-14. Epub 2014 Aug 13.

7.

ZEB negatively regulates the lytic-switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Perrigoue JG, Mertz JE.

J Virol. 2003 Jan;77(1):199-207.

8.

Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.

Reusch JA, Nawandar DM, Wright KL, Kenney SC, Mertz JE.

J Virol. 2015 Feb;89(3):1731-43. doi: 10.1128/JVI.02781-14. Epub 2014 Nov 19.

9.

Latent and lytic Epstein-Barr virus replication strategies.

Tsurumi T, Fujita M, Kudoh A.

Rev Med Virol. 2005 Jan-Feb;15(1):3-15. Review.

PMID:
15386591
10.

The B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1.

Robinson AR, Kwek SS, Kenney SC.

PLoS Pathog. 2012 Feb;8(2):e1002516. doi: 10.1371/journal.ppat.1002516. Epub 2012 Feb 9.

11.

Shutoff of BZLF1 gene expression is necessary for immortalization of primary B cells by Epstein-Barr virus.

Yu X, McCarthy PJ, Wang Z, Gorlen DA, Mertz JE.

J Virol. 2012 Aug;86(15):8086-96. doi: 10.1128/JVI.00234-12. Epub 2012 May 23.

12.

Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.

Kraus RJ, Yu X, Cordes BA, Sathiamoorthi S, Iempridee T, Nawandar DM, Ma S, Romero-Masters JC, McChesney KG, Lin Z, Makielski KR, Lee DL, Lambert PF, Johannsen EC, Kenney SC, Mertz JE.

PLoS Pathog. 2017 Jun 15;13(6):e1006404. doi: 10.1371/journal.ppat.1006404. eCollection 2017 Jun.

13.

Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Mirocha SJ, Stephany HM, Puchalski JR, Mertz JE.

J Virol. 2001 Jan;75(2):867-77.

14.

Switching of EBV cycles between latent and lytic states.

Murata T, Tsurumi T.

Rev Med Virol. 2014 May;24(3):142-53. doi: 10.1002/rmv.1780. Epub 2013 Dec 11. Review.

PMID:
24339346
15.

PARP1 restricts Epstein Barr Virus lytic reactivation by binding the BZLF1 promoter.

Lupey-Green LN, Moquin SA, Martin KA, McDevitt SM, Hulse M, Caruso LB, Pomerantz RT, Miranda JL, Tempera I.

Virology. 2017 Jul;507:220-230. doi: 10.1016/j.virol.2017.04.006. Epub 2017 Apr 26.

16.

BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.

Woellmer A, Arteaga-Salas JM, Hammerschmidt W.

PLoS Pathog. 2012 Sep;8(9):e1002902. doi: 10.1371/journal.ppat.1002902. Epub 2012 Sep 6.

17.

Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral Replication.

Li Y, Long X, Huang L, Yang M, Yuan Y, Wang Y, Delecluse HJ, Kuang E.

J Virol. 2015 Nov 4;90(2):887-903. doi: 10.1128/JVI.01921-15. Print 2016 Jan 15.

18.

Interferon-γ-inducible protein 16 (IFI16) is required for the maintenance of Epstein-Barr virus latency.

Pisano G, Roy A, Ahmed Ansari M, Kumar B, Chikoti L, Chandran B.

Virol J. 2017 Nov 13;14(1):221. doi: 10.1186/s12985-017-0891-5.

19.

The lytic phase of epstein-barr virus requires a viral genome with 5-methylcytosine residues in CpG sites.

Kalla M, Göbel C, Hammerschmidt W.

J Virol. 2012 Jan;86(1):447-58. doi: 10.1128/JVI.06314-11. Epub 2011 Oct 26.

20.

Mutant Cellular AP-1 Proteins Promote Expression of a Subset of Epstein-Barr Virus Late Genes in the Absence of Lytic Viral DNA Replication.

Lyons DE, Yu KP, Vander Heiden JA, Heston L, Dittmer DP, El-Guindy A, Miller G.

J Virol. 2018 Sep 12;92(19). pii: e01062-18. doi: 10.1128/JVI.01062-18. Print 2018 Oct 1.

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