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Items: 1 to 20 of 85

1.

FR-like EBNA1 binding repeats in the human genome.

d'Hérouël AF, Birgersdotter A, Werner M.

Virology. 2010 Sep 30;405(2):524-9. doi: 10.1016/j.virol.2010.06.040. Epub 2010 Jul 22.

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In vivo dynamics of EBNA1-oriP interaction during latent and lytic replication of Epstein-Barr virus.

Daikoku T, Kudoh A, Fujita M, Sugaya Y, Isomura H, Tsurumi T.

J Biol Chem. 2004 Dec 24;279(52):54817-25. Epub 2004 Oct 21.

7.

Reconstitution of Epstein-Barr virus-based plasmid partitioning in budding yeast.

Kapoor P, Shire K, Frappier L.

EMBO J. 2001 Jan 15;20(1-2):222-30.

8.

Multiple EBNA1-binding sites within oriPI are required for EBNA1-dependent transactivation of the Epstein-Barr virus C promoter.

Zetterberg H, Boreström C, Nilsson T, Rymo L.

Int J Oncol. 2004 Sep;25(3):693-6.

PMID:
15289871
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12.

The 2.2 A structure of a permanganate-sensitive DNA site bound by the Epstein-Barr virus origin binding protein, EBNA1.

Bochkarev A, Bochkareva E, Frappier L, Edwards AM.

J Mol Biol. 1998 Dec 18;284(5):1273-8. Review.

PMID:
9878348
13.

EBNA1 distorts oriP, the Epstein-Barr virus latent replication origin.

Frappier L, O'Donnell M.

J Virol. 1992 Mar;66(3):1786-90.

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The affinity of EBNA1 for its origin of DNA synthesis is a determinant of the origin's replicative efficiency.

Lindner SE, Zeller K, Schepers A, Sugden B.

J Virol. 2008 Jun;82(12):5693-702. doi: 10.1128/JVI.00332-08. Epub 2008 Apr 2.

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BZLF1 controlled by family repeat domain induces lytic cytotoxicity in Epstein-Barr virus-positive tumor cells.

Wang H, Zhao Y, Zeng L, Tang M, El-Deeb A, Li JJ, Cao Y.

Anticancer Res. 2004 Jan-Feb;24(1):67-74.

20.

Latency pattern of Epstein-Barr virus and methylation status in Epstein-Barr virus-associated hemophagocytic syndrome.

Yoshioka M, Kikuta H, Ishiguro N, Endo R, Kobayashi K.

J Med Virol. 2003 Jul;70(3):410-9.

PMID:
12767005

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