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Items: 1 to 20 of 141

1.

Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

Skalska L, White RE, Franz M, Ruhmann M, Allday MJ.

PLoS Pathog. 2010 Jun 10;6(6):e1000951. doi: 10.1371/journal.ppat.1000951.

2.

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Skalska L, White RE, Parker GA, Turro E, Sinclair AJ, Paschos K, Allday MJ.

PLoS Pathog. 2013 Feb;9(2):e1003187. doi: 10.1371/journal.ppat.1003187. Epub 2013 Feb 21. Erratum in: PLoS Pathog. 2013 Mar; 9(3): doi:10.1371/annotation/2c4b89c1-6d4a-4bf3-9467-9367227d7e74. Turro, Ernest [added].

3.

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

Bazot Q, Paschos K, Allday MJ.

J Virol. 2018 Mar 14;92(7). pii: e01918-17. doi: 10.1128/JVI.01918-17. Print 2018 Apr 1.

4.

Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression.

Maruo S, Zhao B, Johannsen E, Kieff E, Zou J, Takada K.

Proc Natl Acad Sci U S A. 2011 Feb 1;108(5):1919-24. doi: 10.1073/pnas.1019599108. Epub 2011 Jan 18.

5.

Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.

Schmidt SC, Jiang S, Zhou H, Willox B, Holthaus AM, Kharchenko PV, Johannsen EC, Kieff E, Zhao B.

Proc Natl Acad Sci U S A. 2015 Jan 13;112(2):554-9. doi: 10.1073/pnas.1422580112. Epub 2014 Dec 24.

6.

The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth.

Ohashi M, Holthaus AM, Calderwood MA, Lai CY, Krastins B, Sarracino D, Johannsen E.

PLoS Pathog. 2015 Apr 9;11(4):e1004822. doi: 10.1371/journal.ppat.1004822. eCollection 2015 Apr.

7.

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

Kalchschmidt JS, Gillman AC, Paschos K, Bazot Q, Kempkes B, Allday MJ.

PLoS Pathog. 2016 Jan 11;12(1):e1005383. doi: 10.1371/journal.ppat.1005383. eCollection 2016 Jan.

8.

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Bazot Q, Paschos K, Skalska L, Kalchschmidt JS, Parker GA, Allday MJ.

PLoS Pathog. 2015 Jul 8;11(7):e1005031. doi: 10.1371/journal.ppat.1005031. eCollection 2015 Jul.

9.

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

Styles CT, Bazot Q, Parker GA, White RE, Paschos K, Allday MJ.

PLoS Biol. 2017 Aug 3;15(8):e2001992. doi: 10.1371/journal.pbio.2001992. eCollection 2017 Aug.

10.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.

Jiang S, Willox B, Zhou H, Holthaus AM, Wang A, Shi TT, Maruo S, Kharchenko PV, Johannsen EC, Kieff E, Zhao B.

Proc Natl Acad Sci U S A. 2014 Jan 7;111(1):421-6. doi: 10.1073/pnas.1321704111. Epub 2013 Dec 16.

11.

Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9.

Harth-Hertle ML, Scholz BA, Erhard F, Glaser LV, Dölken L, Zimmer R, Kempkes B.

PLoS Pathog. 2013 Sep;9(9):e1003638. doi: 10.1371/journal.ppat.1003638. Epub 2013 Sep 19.

12.

Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites.

Wang A, Welch R, Zhao B, Ta T, Keleş S, Johannsen E.

J Virol. 2015 Dec 30;90(6):2906-19. doi: 10.1128/JVI.02737-15.

13.

Epstein-Barr virus nuclear antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis.

Cai Q, Guo Y, Xiao B, Banerjee S, Saha A, Lu J, Glisovic T, Robertson ES.

PLoS Pathog. 2011 Dec;7(12):e1002418. doi: 10.1371/journal.ppat.1002418. Epub 2011 Dec 8.

14.

EBNA3C-mediated regulation of aurora kinase B contributes to Epstein-Barr virus-induced B-cell proliferation through modulation of the activities of the retinoblastoma protein and apoptotic caspases.

Jha HC, Lu J, Saha A, Cai Q, Banerjee S, Prasad MA, Robertson ES.

J Virol. 2013 Nov;87(22):12121-38. doi: 10.1128/JVI.02379-13. Epub 2013 Aug 28.

15.

Epstein-Barr virus nuclear protein EBNA3C is required for cell cycle progression and growth maintenance of lymphoblastoid cells.

Maruo S, Wu Y, Ishikawa S, Kanda T, Iwakiri D, Takada K.

Proc Natl Acad Sci U S A. 2006 Dec 19;103(51):19500-5. Epub 2006 Dec 11.

16.

BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV.

Paschos K, Parker GA, Watanatanasup E, White RE, Allday MJ.

Nucleic Acids Res. 2012 Aug;40(15):7233-46. doi: 10.1093/nar/gks391. Epub 2012 May 14.

17.

EBNA3C attenuates the function of p53 through interaction with inhibitor of growth family proteins 4 and 5.

Saha A, Bamidele A, Murakami M, Robertson ES.

J Virol. 2011 Mar;85(5):2079-88. doi: 10.1128/JVI.02279-10. Epub 2010 Dec 22.

19.

Physical and functional interactions between the corepressor CtBP and the Epstein-Barr virus nuclear antigen EBNA3C.

Touitou R, Hickabottom M, Parker G, Crook T, Allday MJ.

J Virol. 2001 Aug;75(16):7749-55.

20.

Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim.

Paschos K, Smith P, Anderton E, Middeldorp JM, White RE, Allday MJ.

PLoS Pathog. 2009 Jun;5(6):e1000492. doi: 10.1371/journal.ppat.1000492. Epub 2009 Jun 26.

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