Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 96

1.

Pain perception is altered by a nucleotide polymorphism in SCN9A.

Reimann F, Cox JJ, Belfer I, Diatchenko L, Zaykin DV, McHale DP, Drenth JP, Dai F, Wheeler J, Sanders F, Wood L, Wu TX, Karppinen J, Nikolajsen L, Männikkö M, Max MB, Kiselycznyk C, Poddar M, Te Morsche RH, Smith S, Gibson D, Kelempisioti A, Maixner W, Gribble FM, Woods CG.

Proc Natl Acad Sci U S A. 2010 Mar 16;107(11):5148-53. doi: 10.1073/pnas.0913181107. Epub 2010 Mar 8.

2.

The Effect of SCN9A Variation on Basal Pain Sensitivity in the General Population: An Experimental Study in Young Women.

Duan G, Guo S, Zhang Y, Ying Y, Huang P, Wang Q, Zhang L, Zhang X.

J Pain. 2015 Oct;16(10):971-80. doi: 10.1016/j.jpain.2015.06.011. Epub 2015 Jul 11.

PMID:
26168879
3.

The non-synonymous SNP, R1150W, in SCN9A is not associated with chronic widespread pain susceptibility.

Holliday KL, Thomson W, Neogi T, Felson DT, Wang K, Wu FC, Huhtaniemi IT, Bartfai G, Casanueva F, Forti G, Kula K, Punab M, Vanderschueren D, Macfarlane GJ, Horan MA, Ollier W, Payton A, Pendleton N, McBeth J.

Mol Pain. 2012 Sep 24;8:72. doi: 10.1186/1744-8069-8-72.

4.

A SCN9A gene-encoded dorsal root ganglia sodium channel polymorphism associated with severe fibromyalgia.

Vargas-Alarcon G, Alvarez-Leon E, Fragoso JM, Vargas A, Martinez A, Vallejo M, Martinez-Lavin M.

BMC Musculoskelet Disord. 2012 Feb 20;13:23. doi: 10.1186/1471-2474-13-23.

5.

Linkage between increased nociception and olfaction via a SCN9A haplotype.

Heimann D, Lötsch J, Hummel T, Doehring A, Oertel BG.

PLoS One. 2013 Jul 10;8(7):e68654. doi: 10.1371/journal.pone.0068654. Print 2013.

6.

Genotypic Analysis of SCN9A for Prediction of Postoperative Pain in Female Patients Undergoing Gynecological Laparoscopic Surgery.

Duan G, Xiang G, Guo S, Zhang Y, Ying Y, Huang P, Zheng H, Zhang M, Li N, Zhang X.

Pain Physician. 2016 Jan;19(1):E151-62.

7.

[Neuropathic pain associated with Nav1.7 mutations: clinical picture and treatment].

Doppler K, Sommer C.

Nervenarzt. 2013 Dec;84(12):1428-35. doi: 10.1007/s00115-012-3621-7. Review. German.

PMID:
24202110
8.

Congenital insensitivity to pain: novel SCN9A missense and in-frame deletion mutations.

Cox JJ, Sheynin J, Shorer Z, Reimann F, Nicholas AK, Zubovic L, Baralle M, Wraige E, Manor E, Levy J, Woods CG, Parvari R.

Hum Mutat. 2010 Sep;31(9):E1670-86. doi: 10.1002/humu.21325.

9.

Polymorphism in the SCN9A voltage-gated sodium channel gene associated with interstitial cystitis/bladder pain syndrome.

Reeder JE, Byler TK, Foster DC, Landas SK, Okafor H, Stearns G, Wood RW, Zhang Y, Mayer RD.

Urology. 2013 Jan;81(1):210.e1-4. doi: 10.1016/j.urology.2012.07.072. Epub 2012 Oct 24.

PMID:
23102778
10.

Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable.

Dib-Hajj SD, Estacion M, Jarecki BW, Tyrrell L, Fischer TZ, Lawden M, Cummins TR, Waxman SG.

Mol Pain. 2008 Sep 19;4:37. doi: 10.1186/1744-8069-4-37.

11.

Novel SCN9A mutations underlying extreme pain phenotypes: unexpected electrophysiological and clinical phenotype correlations.

Emery EC, Habib AM, Cox JJ, Nicholas AK, Gribble FM, Woods CG, Reimann F.

J Neurosci. 2015 May 20;35(20):7674-81. doi: 10.1523/JNEUROSCI.3935-14.2015.

12.

No mutations in the voltage-gated NaV1.7 sodium channel alpha1 subunit gene SCN9A in familial complex regional pain syndrome.

de Rooij AM, Gosso MF, Alsina-Sanchis E, Marinus J, van Hilten JJ, van den Maagdenberg AM.

Eur J Neurol. 2010 Jun 1;17(6):808-14. doi: 10.1111/j.1468-1331.2009.02931.x. Epub 2010 Jan 12.

PMID:
20074229
13.

Mutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders.

Drenth JP, Waxman SG.

J Clin Invest. 2007 Dec;117(12):3603-9. Review.

14.

An SCN9A channelopathy causes congenital inability to experience pain.

Cox JJ, Reimann F, Nicholas AK, Thornton G, Roberts E, Springell K, Karbani G, Jafri H, Mannan J, Raashid Y, Al-Gazali L, Hamamy H, Valente EM, Gorman S, Williams R, McHale DP, Wood JN, Gribble FM, Woods CG.

Nature. 2006 Dec 14;444(7121):894-8.

PMID:
17167479
15.

SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes.

Fertleman CR, Baker MD, Parker KA, Moffatt S, Elmslie FV, Abrahamsen B, Ostman J, Klugbauer N, Wood JN, Gardiner RM, Rees M.

Neuron. 2006 Dec 7;52(5):767-74.

16.

Two novel SCN9A mutations causing insensitivity to pain.

Nilsen KB, Nicholas AK, Woods CG, Mellgren SI, Nebuchennykh M, Aasly J.

Pain. 2009 May;143(1-2):155-8. doi: 10.1016/j.pain.2009.02.016. Epub 2009 Mar 21. Erratum in: Pain. 2009 Sep;145(1-2):264.

PMID:
19304393
17.

A sodium channel gene SCN9A polymorphism that increases nociceptor excitability.

Estacion M, Harty TP, Choi JS, Tyrrell L, Dib-Hajj SD, Waxman SG.

Ann Neurol. 2009 Dec;66(6):862-6. doi: 10.1002/ana.21895.

PMID:
20033988
18.

Painful and painless channelopathies.

Bennett DL, Woods CG.

Lancet Neurol. 2014 Jun;13(6):587-99. doi: 10.1016/S1474-4422(14)70024-9. Epub 2014 May 6. Review.

PMID:
24813307
19.

A single-nucleotide polymorphism in SCN9A may decrease postoperative pain sensitivity in the general population.

Duan G, Xiang G, Zhang X, Yuan R, Zhan H, Qi D.

Anesthesiology. 2013 Feb;118(2):436-42. doi: 10.1097/ALN.0b013e31827dde74.

PMID:
23364568
20.

Two novel mutations of SCN9A (Nav1.7) are associated with partial congenital insensitivity to pain.

Staud R, Price DD, Janicke D, Andrade E, Hadjipanayis AG, Eaton WT, Kaplan L, Wallace MR.

Eur J Pain. 2011 Mar;15(3):223-30. doi: 10.1016/j.ejpain.2010.07.003. Epub 2010 Aug 7.

Supplemental Content

Support Center