Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 124

1.

Burn-induced heart failure: lipopolysaccharide binding protein improves burn and endotoxin-induced cardiac contractility deficits.

Niederbichler AD, Hoesel LM, Ipaktchi K, Olivarez L, Erdmann M, Vogt PM, Su GL, Arbabi S, Westfall MV, Wang SC, Hemmila MR.

J Surg Res. 2011 Jan;165(1):128-35. doi: 10.1016/j.jss.2009.06.012. Epub 2009 Dec 6.

2.

Cardiomyocyte function after burn injury and lipopolysaccharide exposure: single-cell contraction analysis and cytokine secretion profile.

Niederbichler AD, Westfall MV, Su GL, Donnerberg J, Usman A, Vogt PM, Ipaktchi KR, Arbabi S, Wang SC, Hemmila MR.

Shock. 2006 Feb;25(2):176-83.

PMID:
16525357
3.

C5a-blockade improves burn-induced cardiac dysfunction.

Hoesel LM, Niederbichler AD, Schaefer J, Ipaktchi KR, Gao H, Rittirsch D, Pianko MJ, Vogt PM, Sarma JV, Su GL, Arbabi S, Westfall MV, Wang SC, Hemmila MR, Ward PA.

J Immunol. 2007 Jun 15;178(12):7902-10.

4.

Competition between rBPI23, a recombinant fragment of bactericidal/permeability-increasing protein, and lipopolysaccharide (LPS)-binding protein for binding to LPS and gram-negative bacteria.

Gazzano-Santoro H, Mészáros K, Birr C, Carroll SF, Theofan G, Horwitz AH, Lim E, Aberle S, Kasler H, Parent JB.

Infect Immun. 1994 Apr;62(4):1185-91.

5.

Local wound p38 MAPK inhibition attenuates burn-induced cardiac dysfunction.

Hoesel LM, Mattar AF, Arbabi S, Niederbichler AD, Ipaktchi K, Su GL, Westfall MV, Wang SC, Hemmila MR.

Surgery. 2009 Oct;146(4):775-85; discussion 785-6. doi: 10.1016/j.surg.2009.06.019.

6.

Normal rat hepatic stellate cells respond to endotoxin in LBP-independent manner to produce inhibitor(s) of DNA synthesis in hepatocytes.

Thirunavukkarasu C, Uemura T, Wang LF, Watkins SC, Gandhi CR.

J Cell Physiol. 2005 Aug;204(2):654-65.

PMID:
15828022
8.

Bovine recombinant lipopolysaccharide binding protein (BRLBP) regulated apoptosis and inflammation response in lipopolysaccharide-challenged bovine mammary epithelial cells (BMEC).

Sun Y, Li L, Wu J, Yu P, Li C, Tang J, Li X, Huang S, Wang G.

Mol Immunol. 2015 Jun;65(2):205-14. doi: 10.1016/j.molimm.2015.01.026. Epub 2015 Feb 18.

PMID:
25700343
9.
10.

Lipopolysaccharide (LPS)-binding protein accelerates the binding of LPS to CD14.

Hailman E, Lichenstein HS, Wurfel MM, Miller DS, Johnson DA, Kelley M, Busse LA, Zukowski MM, Wright SD.

J Exp Med. 1994 Jan 1;179(1):269-77.

11.

TLR4 inactivation and rBPI(21) block burn-induced myocardial contractile dysfunction.

Thomas JA, Tsen MF, White DJ, Horton JW.

Am J Physiol Heart Circ Physiol. 2002 Oct;283(4):H1645-55. Epub 2002 Jun 13.

12.

Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function.

Maass DL, White J, Sanders B, Horton JW.

Am J Physiol Heart Circ Physiol. 2005 Feb;288(2):H744-51. Epub 2004 Sep 23.

13.

Altered Kupffer cell function in biliary obstruction.

Minter RM, Fan MH, Sun J, Niederbichler A, Ipaktchi K, Arbabi S, Hemmila MR, Remick DG, Wang SC, Su GL.

Surgery. 2005 Aug;138(2):236-45.

PMID:
16153432
14.

Skin lipopolysaccharide-binding protein and IL-1beta production after thermal injury.

Klein RD, Su GL, Aminlari A, Zhang H, Steinstraesser L, Alarcon WH, Wang SC.

J Burn Care Rehabil. 2000 Jul-Aug;21(4):345-52.

PMID:
10935817
15.

Lipopolysaccharide-binding protein down-regulates the expression of interleukin-6 by human gingival fibroblast.

Ren L, Leung WK, Loo TW, Jin L.

J Periodontal Res. 2005 Oct;40(5):407-16.

PMID:
16105094
16.

Kupffer cell activation by lipopolysaccharide in rats: role for lipopolysaccharide binding protein and toll-like receptor 4.

Su GL, Klein RD, Aminlari A, Zhang HY, Steinstraesser L, Alarcon WH, Remick DG, Wang SC.

Hepatology. 2000 Apr;31(4):932-6.

17.
18.

Influence of CD14, LBP and BPI in the monocyte response to LPS of different polysaccharide chain length.

Jahr TG, Sundan A, Lichenstein HS, Espevik T.

Scand J Immunol. 1995 Jul;42(1):119-27.

PMID:
7543211
19.
20.

An amino-terminal fragment of human lipopolysaccharide-binding protein retains lipid A binding but not CD14-stimulatory activity.

Theofan G, Horwitz AH, Williams RE, Liu PS, Chan I, Birr C, Carroll SF, Mészáros K, Parent JB, Kasler H, et al.

J Immunol. 1994 Apr 1;152(7):3623-9.

PMID:
7511654

Supplemental Content

Support Center