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Items: 1 to 20 of 143

1.

Altered distribution of RhoA in Alzheimer's disease and AbetaPP overexpressing mice.

Huesa G, Baltrons MA, Gómez-Ramos P, Morán A, García A, Hidalgo J, Francés S, Santpere G, Ferrer I, Galea E.

J Alzheimers Dis. 2010;19(1):37-56. doi: 10.3233/JAD-2010-1203.

PMID:
20061625
3.

Enhanced accumulation of tau in doubly transgenic mice expressing mutant betaAPP and presenilin-1.

Samura E, Shoji M, Kawarabayashi T, Sasaki A, Matsubara E, Murakami T, Wuhua X, Tamura S, Ikeda M, Ishiguro K, Saido TC, Westaway D, St George Hyslop P, Harigaya Y, Abe K.

Brain Res. 2006 Jun 13;1094(1):192-9. Epub 2006 May 19.

PMID:
16713590
5.

Amyloid precursor protein cytoplasmic domain with phospho-Thr668 accumulates in Alzheimer's disease and its transgenic models: a role to mediate interaction of Abeta and tau.

Shin RW, Ogino K, Shimabuku A, Taki T, Nakashima H, Ishihara T, Kitamoto T.

Acta Neuropathol. 2007 Jun;113(6):627-36. Epub 2007 Mar 13.

PMID:
17431643
6.

Characterisation of cytoskeletal abnormalities in mice transgenic for wild-type human tau and familial Alzheimer's disease mutants of APP and presenilin-1.

Boutajangout A, Authelet M, Blanchard V, Touchet N, Tremp G, Pradier L, Brion JP.

Neurobiol Dis. 2004 Feb;15(1):47-60.

PMID:
14751770
7.

Reduced Reelin expression accelerates amyloid-beta plaque formation and tau pathology in transgenic Alzheimer's disease mice.

Kocherhans S, Madhusudan A, Doehner J, Breu KS, Nitsch RM, Fritschy JM, Knuesel I.

J Neurosci. 2010 Jul 7;30(27):9228-40. doi: 10.1523/JNEUROSCI.0418-10.2010.

8.

Neurofibrillary pathology in transgenic mice overexpressing V717F beta-amyloid precursor protein.

Masliah E, Sisk A, Mallory M, Games D.

J Neuropathol Exp Neurol. 2001 Apr;60(4):357-68.

PMID:
11305871
10.

Amyloid cored plaques in Tg2576 transgenic mice are characterized by giant plaques, slightly activated microglia, and the lack of paired helical filament-typed, dystrophic neurites.

Sasaki A, Shoji M, Harigaya Y, Kawarabayashi T, Ikeda M, Naito M, Matsubara E, Abe K, Nakazato Y.

Virchows Arch. 2002 Oct;441(4):358-67. Epub 2002 May 1.

PMID:
12404061
11.

The beta-amyloid protein of Alzheimer's disease increases neuronal CRMP-2 phosphorylation by a Rho-GTP mechanism.

Petratos S, Li QX, George AJ, Hou X, Kerr ML, Unabia SE, Hatzinisiriou I, Maksel D, Aguilar MI, Small DH.

Brain. 2008 Jan;131(Pt 1):90-108. Epub 2007 Nov 13.

PMID:
18000012
12.

Neurofibrillary tangles and tau phosphorylation.

Brion JP, Anderton BH, Authelet M, Dayanandan R, Leroy K, Lovestone S, Octave JN, Pradier L, Touchet N, Tremp G.

Biochem Soc Symp. 2001;(67):81-8.

PMID:
11447842
13.

Notch-1 immunoexpression is increased in Alzheimer's and Pick's disease.

Nagarsheth MH, Viehman A, Lippa SM, Lippa CF.

J Neurol Sci. 2006 May 15;244(1-2):111-6. Epub 2006 Feb 13.

PMID:
16473372
14.

Constitutive Dyrk1A is abnormally expressed in Alzheimer disease, Down syndrome, Pick disease, and related transgenic models.

Ferrer I, Barrachina M, Puig B, Martínez de Lagrán M, Martí E, Avila J, Dierssen M.

Neurobiol Dis. 2005 Nov;20(2):392-400.

PMID:
16242644
15.

Mechanisms of neurofibrillary degeneration and the formation of neurofibrillary tangles.

Iqbal K, Alonso AC, Gong CX, Khatoon S, Pei JJ, Wang JZ, Grundke-Iqbal I.

J Neural Transm Suppl. 1998;53:169-80. Review.

PMID:
9700655
16.

Truncated tau at D421 is associated with neurodegeneration and tangle formation in the brain of Alzheimer transgenic models.

Zhang Q, Zhang X, Sun A.

Acta Neuropathol. 2009 Jun;117(6):687-97. doi: 10.1007/s00401-009-0491-6. Epub 2009 Feb 4.

PMID:
19190923
18.

Activation of c-Jun N-terminal kinase and p38 in an Alzheimer's disease model is associated with amyloid deposition.

Savage MJ, Lin YG, Ciallella JR, Flood DG, Scott RW.

J Neurosci. 2002 May 1;22(9):3376-85.

19.

Neuronal pentraxin 1 contributes to the neuronal damage evoked by amyloid-beta and is overexpressed in dystrophic neurites in Alzheimer's brain.

Abad MA, Enguita M, DeGregorio-Rocasolano N, Ferrer I, Trullas R.

J Neurosci. 2006 Dec 6;26(49):12735-47.

20.

Pick's disease: hyperphosphorylated tau protein segregates to the somatoaxonal compartment.

Probst A, Tolnay M, Langui D, Goedert M, Spillantini MG.

Acta Neuropathol. 1996 Dec;92(6):588-96.

PMID:
8960316

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