Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 96


Epigenetic regulation of latent HSV-1 gene expression.

Bloom DC, Giordani NV, Kwiatkowski DL.

Biochim Biophys Acta. 2010 Mar-Apr;1799(3-4):246-56. doi: 10.1016/j.bbagrm.2009.12.001. Epub 2010 Jan 4. Review.


The polycomb group protein Bmi1 binds to the herpes simplex virus 1 latent genome and maintains repressive histone marks during latency.

Kwiatkowski DL, Thompson HW, Bloom DC.

J Virol. 2009 Aug;83(16):8173-81. doi: 10.1128/JVI.00686-09. Epub 2009 Jun 10.


Transcription of the herpes simplex virus latency-associated transcript promotes the formation of facultative heterochromatin on lytic promoters.

Cliffe AR, Garber DA, Knipe DM.

J Virol. 2009 Aug;83(16):8182-90. doi: 10.1128/JVI.00712-09. Epub 2009 Jun 10.


MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAs.

Umbach JL, Kramer MF, Jurak I, Karnowski HW, Coen DM, Cullen BR.

Nature. 2008 Aug 7;454(7205):780-3. doi: 10.1038/nature07103. Epub 2008 Jul 2.


Herpesviral latency-associated transcript gene promotes assembly of heterochromatin on viral lytic-gene promoters in latent infection.

Wang QY, Zhou C, Johnson KE, Colgrove RC, Coen DM, Knipe DM.

Proc Natl Acad Sci U S A. 2005 Nov 1;102(44):16055-9. Epub 2005 Oct 24.


The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected Neurons.

Nicoll MP, Hann W, Shivkumar M, Harman LE, Connor V, Coleman HM, Proença JT, Efstathiou S.

PLoS Pathog. 2016 Apr 7;12(4):e1005539. doi: 10.1371/journal.ppat.1005539. eCollection 2016 Apr.


CTCF Binding Sites in the Herpes Simplex Virus 1 Genome Display Site-Specific CTCF Occupation, Protein Recruitment, and Insulator Function.

Washington SD, Musarrat F, Ertel MK, Backes GL, Neumann DM.

J Virol. 2018 Mar 28;92(8). pii: e00156-18. doi: 10.1128/JVI.00156-18. Print 2018 Apr 15.


CTCF-dependent chromatin boundary element between the latency-associated transcript and ICP0 promoters in the herpes simplex virus type 1 genome.

Chen Q, Lin L, Smith S, Huang J, Berger SL, Zhou J.

J Virol. 2007 May;81(10):5192-201. Epub 2007 Jan 31.


CTCF occupation of the herpes simplex virus 1 genome is disrupted at early times postreactivation in a transcription-dependent manner.

Ertel MK, Cammarata AL, Hron RJ, Neumann DM.

J Virol. 2012 Dec;86(23):12741-59. doi: 10.1128/JVI.01655-12. Epub 2012 Sep 12.


Epigenetic analysis of KSHV latent and lytic genomes.

Toth Z, Maglinte DT, Lee SH, Lee HR, Wong LY, Brulois KF, Lee S, Buckley JD, Laird PW, Marquez VE, Jung JU.

PLoS Pathog. 2010 Jul 22;6(7):e1001013. doi: 10.1371/journal.ppat.1001013.


CCCTC-Binding Factor Acts as a Heterochromatin Barrier on Herpes Simplex Viral Latent Chromatin and Contributes to Poised Latent Infection.

Lee JS, Raja P, Pan D, Pesola JM, Coen DM, Knipe DM.

MBio. 2018 Feb 6;9(1). pii: e02372-17. doi: 10.1128/mBio.02372-17.


CTCF prevents the epigenetic drift of EBV latency promoter Qp.

Tempera I, Wiedmer A, Dheekollu J, Lieberman PM.

PLoS Pathog. 2010 Aug 12;6(8):e1001048. doi: 10.1371/journal.ppat.1001048.


A Herpesviral Lytic Protein Regulates the Structure of Latent Viral Chromatin.

Raja P, Lee JS, Pan D, Pesola JM, Coen DM, Knipe DM.

MBio. 2016 May 17;7(3). pii: e00633-16. doi: 10.1128/mBio.00633-16.


Epigenetic regulation of Kaposi's sarcoma-associated herpesvirus latency by virus-encoded microRNAs that target Rta and the cellular Rbl2-DNMT pathway.

Lu F, Stedman W, Yousef M, Renne R, Lieberman PM.

J Virol. 2010 Mar;84(6):2697-706. doi: 10.1128/JVI.01997-09. Epub 2010 Jan 13.


Thyroid hormone controls the gene expression of HSV-1 LAT and ICP0 in neuronal cells.

Bedadala GR, Pinnoji RC, Palem JR, Hsia SC.

Cell Res. 2010 May;20(5):587-98. doi: 10.1038/cr.2010.50. Epub 2010 Apr 13.


Chromatin dynamics during herpes simplex virus-1 lytic infection.

Placek BJ, Berger SL.

Biochim Biophys Acta. 2010 Mar-Apr;1799(3-4):223-7. doi: 10.1016/j.bbagrm.2010.01.012. Epub 2010 Feb 6. Review.


Supplemental Content

Support Center