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Items: 1 to 20 of 455

1.

Smad2 and Smad3 have opposing roles in breast cancer bone metastasis by differentially affecting tumor angiogenesis.

Petersen M, Pardali E, van der Horst G, Cheung H, van den Hoogen C, van der Pluijm G, Ten Dijke P.

Oncogene. 2010 Mar 4;29(9):1351-61. doi: 10.1038/onc.2009.426. Epub 2009 Dec 14.

PMID:
20010874
2.

The tumor suppressor Smad4 is required for transforming growth factor beta-induced epithelial to mesenchymal transition and bone metastasis of breast cancer cells.

Deckers M, van Dinther M, Buijs J, Que I, Löwik C, van der Pluijm G, ten Dijke P.

Cancer Res. 2006 Feb 15;66(4):2202-9.

3.

A tale of two proteins: differential roles and regulation of Smad2 and Smad3 in TGF-beta signaling.

Brown KA, Pietenpol JA, Moses HL.

J Cell Biochem. 2007 May 1;101(1):9-33. Review.

PMID:
17340614
4.

Smad2 and Smad3 phosphorylated at both linker and COOH-terminal regions transmit malignant TGF-beta signal in later stages of human colorectal cancer.

Matsuzaki K, Kitano C, Murata M, Sekimoto G, Yoshida K, Uemura Y, Seki T, Taketani S, Fujisawa J, Okazaki K.

Cancer Res. 2009 Jul 1;69(13):5321-30. doi: 10.1158/0008-5472.CAN-08-4203. Epub 2009 Jun 16.

5.

Functional characterization of transforming growth factor beta signaling in Smad2- and Smad3-deficient fibroblasts.

Piek E, Ju WJ, Heyer J, Escalante-Alcalde D, Stewart CL, Weinstein M, Deng C, Kucherlapati R, Bottinger EP, Roberts AB.

J Biol Chem. 2001 Jun 8;276(23):19945-53. Epub 2001 Mar 21.

6.

Differential regulation of TGF-beta signaling through Smad2, Smad3 and Smad4.

Kretschmer A, Moepert K, Dames S, Sternberger M, Kaufmann J, Klippel A.

Oncogene. 2003 Oct 2;22(43):6748-63.

PMID:
14555988
7.

TGF-beta-induced nuclear localization of Smad2 and Smad3 in Smad4 null cancer cell lines.

Fink SP, Mikkola D, Willson JK, Markowitz S.

Oncogene. 2003 Mar 6;22(9):1317-23.

PMID:
12618756
8.

Diffuse-type gastric carcinoma: progression, angiogenesis, and transforming growth factor beta signaling.

Komuro A, Yashiro M, Iwata C, Morishita Y, Johansson E, Matsumoto Y, Watanabe A, Aburatani H, Miyoshi H, Kiyono K, Shirai YT, Suzuki HI, Hirakawa K, Kano MR, Miyazono K.

J Natl Cancer Inst. 2009 Apr 15;101(8):592-604. doi: 10.1093/jnci/djp058. Epub 2009 Apr 7.

9.

Smad3 is a key nonredundant mediator of transforming growth factor beta signaling in Nme mouse mammary epithelial cells.

Dzwonek J, Preobrazhenska O, Cazzola S, Conidi A, Schellens A, van Dinther M, Stubbs A, Klippel A, Huylebroeck D, ten Dijke P, Verschueren K.

Mol Cancer Res. 2009 Aug;7(8):1342-53. doi: 10.1158/1541-7786.MCR-08-0558. Epub 2009 Aug 11.

10.

Alterations of Smad signaling in human breast carcinoma are associated with poor outcome: a tissue microarray study.

Xie W, Mertens JC, Reiss DJ, Rimm DL, Camp RL, Haffty BG, Reiss M.

Cancer Res. 2002 Jan 15;62(2):497-505.

11.

Smad-binding defective mutant of transforming growth factor beta type I receptor enhances tumorigenesis but suppresses metastasis of breast cancer cell lines.

Tian F, Byfield SD, Parks WT, Stuelten CH, Nemani D, Zhang YE, Roberts AB.

Cancer Res. 2004 Jul 1;64(13):4523-30.

12.
13.

Smad2 and Smad3 are redundantly essential for the TGF-beta-mediated regulation of regulatory T plasticity and Th1 development.

Takimoto T, Wakabayashi Y, Sekiya T, Inoue N, Morita R, Ichiyama K, Takahashi R, Asakawa M, Muto G, Mori T, Hasegawa E, Saika S, Hara T, Nomura M, Yoshimura A.

J Immunol. 2010 Jul 15;185(2):842-55. doi: 10.4049/jimmunol.0904100. Epub 2010 Jun 14. Erratum in: J Immunol. 2011 Jan 1;186(1):632. Shizuya, Saika [corrected to Saika, Shizuya].

14.

Transforming growth factor-beta suppressed Id-1 Expression in a smad3-dependent manner in LoVo cells.

Song H, Guo B, Zhang J, Song C.

Anat Rec (Hoboken). 2010 Jan;293(1):42-7. doi: 10.1002/ar.21012.

15.
16.

Clusterin, a novel modulator of TGF-beta signaling, is involved in Smad2/3 stability.

Lee KB, Jeon JH, Choi I, Kwon OY, Yu K, You KH.

Biochem Biophys Res Commun. 2008 Feb 22;366(4):905-9. Epub 2007 Dec 17.

PMID:
18082619
17.

Chromatin immunoprecipitation on microarray analysis of Smad2/3 binding sites reveals roles of ETS1 and TFAP2A in transforming growth factor beta signaling.

Koinuma D, Tsutsumi S, Kamimura N, Taniguchi H, Miyazawa K, Sunamura M, Imamura T, Miyazono K, Aburatani H.

Mol Cell Biol. 2009 Jan;29(1):172-86. doi: 10.1128/MCB.01038-08. Epub 2008 Oct 27.

19.

The endogenous ratio of Smad2 and Smad3 influences the cytostatic function of Smad3.

Kim SG, Kim HA, Jong HS, Park JH, Kim NK, Hong SH, Kim TY, Bang YJ.

Mol Biol Cell. 2005 Oct;16(10):4672-83. Epub 2005 Aug 10.

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