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Items: 1 to 20 of 98

1.

D-cycloserine improves functional outcome after traumatic brain injury with wide therapeutic window.

Adeleye A, Shohami E, Nachman D, Alexandrovich A, Trembovler V, Yaka R, Shoshan Y, Dhawan J, Biegon A.

Eur J Pharmacol. 2010 Mar 10;629(1-3):25-30. doi: 10.1016/j.ejphar.2009.11.066.

2.

D-cycloserine improves functional recovery and reinstates long-term potentiation (LTP) in a mouse model of closed head injury.

Yaka R, Biegon A, Grigoriadis N, Simeonidou C, Grigoriadis S, Alexandrovich AG, Matzner H, Schumann J, Trembovler V, Tsenter J, Shohami E.

FASEB J. 2007 Jul;21(9):2033-41.

3.

Novel approach to the role of NMDA receptors in traumatic brain injury.

Shohami E, Biegon A.

CNS Neurol Disord Drug Targets. 2014;13(4):567-73. Review.

PMID:
24168367
4.
5.

Intrathecally administered D-cycloserine produces nociceptive behavior through the activation of N-methyl-D-aspartate receptor ion-channel complex acting on the glycine recognition site.

Tan-No K, Esashi A, Nakagawasai O, Niijima F, Furuta S, Sato T, Satoh S, Yasuhara H, Tadano T.

J Pharmacol Sci. 2007 May;104(1):39-45.

6.
8.

Dynamic changes in N-methyl-D-aspartate receptors after closed head injury in mice: Implications for treatment of neurological and cognitive deficits.

Biegon A, Fry PA, Paden CM, Alexandrovich A, Tsenter J, Shohami E.

Proc Natl Acad Sci U S A. 2004 Apr 6;101(14):5117-22.

9.

Absence of significant interactive effects of high-dose D-cycloserine and ethanol in healthy human subjects: preliminary insights into ethanol actions at the glycine B site of NMDA glutamate receptors.

Trevisan L, Petrakis IL, Pittman B, Gueorguieva R, D'Souza DC, Perry E, Limoncelli D, Krystal JH.

Alcohol Clin Exp Res. 2008 Jan;32(1):36-42.

PMID:
18028532
10.
12.

Glycine and D: -serine, but not D: -cycloserine, attenuate prepulse inhibition deficits induced by NMDA receptor antagonist MK-801.

Kanahara N, Shimizu E, Ohgake S, Fujita Y, Kohno M, Hashimoto T, Matsuzawa D, Shirayama Y, Hashimoto K, Iyo M.

Psychopharmacology (Berl). 2008 Jun;198(3):363-74. doi: 10.1007/s00213-008-1151-6.

PMID:
18437299
13.

Reconsolidation and extinction of conditioned fear: inhibition and potentiation.

Lee JL, Milton AL, Everitt BJ.

J Neurosci. 2006 Sep 27;26(39):10051-6.

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NMDA receptor antagonist MK-801 reduces neuronal damage and preserves learning and memory in a rat model of traumatic brain injury.

Han RZ, Hu JJ, Weng YC, Li DF, Huang Y.

Neurosci Bull. 2009 Dec;25(6):367-75. doi: 10.1007/s12264-009-0608-x.

PMID:
19927173
16.

Stress and a glycinergic intervention interact in the modulation of MK-801-elicited mouse popping behavior.

Deutsch SI, Rosse RB, Schwartz BL, Powell DG, Mastropaolo J.

Pharmacol Biochem Behav. 1999 Feb;62(2):395-8.

PMID:
9972709
17.

Modulatory effects of an NMDAR partial agonist in MK-801-induced memory impairment.

Vishnoi S, Raisuddin S, Parvez S.

Neuroscience. 2015 Dec 17;311:22-33. doi: 10.1016/j.neuroscience.2015.10.008.

PMID:
26454025
18.

D-cycloserine injected into the dorsolateral periaqueductal gray induces anxiolytic-like effects in rats.

Gomes FV, Kakihata AM, Semedo AC, Hott SC, Uliana DL, Guimarães FS, Resstel LB.

Behav Brain Res. 2014 Sep 1;271:374-9. doi: 10.1016/j.bbr.2014.06.009.

PMID:
24931794
19.

Selective blockade of the mGluR1 receptor reduces traumatic neuronal injury in vitro and improvesoOutcome after brain trauma.

Faden AI, O'Leary DM, Fan L, Bao W, Mullins PG, Movsesyan VA.

Exp Neurol. 2001 Feb;167(2):435-44.

PMID:
11161632
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