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Items: 1 to 20 of 174

1.

Ventricular unloading is associated with increased 20s proteasome protein expression in the myocardium.

Wohlschlaeger J, Sixt SU, Stoeppler T, Schmitz KJ, Levkau B, Tsagakis K, Vahlhaus C, Schmid C, Peters J, Schmid KW, Milting H, Baba HA.

J Heart Lung Transplant. 2010 Jan;29(1):125-32. doi: 10.1016/j.healun.2009.07.022. Epub 2009 Oct 17.

PMID:
19837610
2.

Reversible regulation of the retinoblastoma protein/E2F-1 pathway during "reverse cardiac remodelling" after ventricular unloading.

Wohlschlaeger J, Schmitz KJ, Takeda A, Takeda N, Vahlhaus C, Stypmann J, Schmid C, Baba HA.

J Heart Lung Transplant. 2010 Jan;29(1):117-24. doi: 10.1016/j.healun.2009.09.017.

PMID:
20123249
3.

Cardiomyocyte survivin protein expression is associated with cell size and DNA content in the failing human heart and is reversibly regulated after ventricular unloading.

Wohlschlaeger J, Meier B, Schmitz KJ, Takeda A, Takeda N, Vahlhaus C, Levkau B, Stypmann J, Schmid C, Schmid KW, Baba HA.

J Heart Lung Transplant. 2010 Nov;29(11):1286-92. doi: 10.1016/j.healun.2010.06.015. Epub 2010 Sep 20.

PMID:
20851637
4.

Decreased myocardial chromogranin a expression and colocalization with brain natriuretic peptide during reverse cardiac remodeling after ventricular unloading.

Wohlschlaeger J, von Winterfeld M, Milting H, El Banayosy A, Schmitz KJ, Takeda A, Takeda N, Azhari P, Schmid C, August C, Schmid KW, Baba HA.

J Heart Lung Transplant. 2008 Apr;27(4):442-9. doi: 10.1016/j.healun.2008.01.017.

PMID:
18374882
5.

Roles of cyclooxygenase-2 and phosphorylated Akt (Thr308) in cardiac hypertrophy regression mediated by left-ventricular unloading.

Wohlschlaeger J, Schmitz KJ, Palatty J, Takeda A, Takeda N, Vahlhaus C, Levkau B, Stypmann J, Schmid C, Schmid KW, Baba HA.

J Thorac Cardiovasc Surg. 2007 Jan;133(1):37-43.

6.

Hemodynamic support by left ventricular assist devices reduces cardiomyocyte DNA content in the failing human heart.

Wohlschlaeger J, Levkau B, Brockhoff G, Schmitz KJ, von Winterfeld M, Takeda A, Takeda N, Stypmann J, Vahlhaus C, Schmid C, Pomjanski N, Böcking A, Baba HA.

Circulation. 2010 Mar 2;121(8):989-96. doi: 10.1161/CIRCULATIONAHA.108.808071. Epub 2010 Feb 16.

7.

Left ventricular assist device support reverses altered cardiac expression and function of natriuretic peptides and receptors in end-stage heart failure.

Kuhn M, Voss M, Mitko D, Stypmann J, Schmid C, Kawaguchi N, Grabellus F, Baba HA.

Cardiovasc Res. 2004 Nov 1;64(2):308-14.

PMID:
15485690
8.

Sarcomeric genes involved in reverse remodeling of the heart during left ventricular assist device support.

Rodrigue-Way A, Burkhoff D, Geesaman BJ, Golden S, Xu J, Pollman MJ, Donoghue M, Jeyaseelan R, Houser S, Breitbart RE, Marks A, Acton S.

J Heart Lung Transplant. 2005 Jan;24(1):73-80.

PMID:
15653383
9.

Biventricular assist device-induced right ventricular reverse structural and functional remodeling.

Klotz S, Naka Y, Oz MC, Burkhoff D.

J Heart Lung Transplant. 2005 Sep;24(9):1195-201.

PMID:
16143233
10.

The ubiquitin-proteasome system in cardiac physiology and pathology.

Powell SR.

Am J Physiol Heart Circ Physiol. 2006 Jul;291(1):H1-H19. Epub 2006 Feb 24. Review.

11.

The ubiquitin-proteasome system: focus on the heart.

Zolk O, Schenke C, Sarikas A.

Cardiovasc Res. 2006 Jun 1;70(3):410-21. Epub 2006 Feb 23. Review.

PMID:
16497285
12.

Mechanical unloading increases caveolin expression in the failing human heart.

Uray IP, Connelly JH, Frazier OH, Taegtmeyer H, Davies PJ.

Cardiovasc Res. 2003 Jul 1;59(1):57-66.

PMID:
12829176
13.

Depression of proteasome activities during the progression of cardiac dysfunction in pressure-overloaded heart of mice.

Tsukamoto O, Minamino T, Okada K, Shintani Y, Takashima S, Kato H, Liao Y, Okazaki H, Asai M, Hirata A, Fujita M, Asano Y, Yamazaki S, Asanuma H, Hori M, Kitakaze M.

Biochem Biophys Res Commun. 2006 Feb 24;340(4):1125-33. Epub 2005 Dec 28.

PMID:
16403436
14.

Mechanical unloading of the heart activates the calpain system.

Razeghi P, Volpini KC, Wang ME, Youker KA, Stepkowski S, Taegtmeyer H.

J Mol Cell Cardiol. 2007 Feb;42(2):449-52. Epub 2006 Oct 4.

PMID:
17027024
15.

The structural examination of myocardial samples from patients with end-stage heart failure supported by ventricular assist devices using electron microscopy and amino acid analysis reveals low degree of reverse remodeling.

Milting H, Jacob M, Kassner A, Heimann P, Mannherz HG, Becker G, Meyer HE, Böthig D, Arusoglu L, Morshuis M, Körfer R, El Banayosy A.

J Heart Lung Transplant. 2004 Apr;23(4):396-404.

PMID:
15063398
16.

Reversal of metallothionein expression is different throughout the human myocardium after prolonged left-ventricular mechanical support.

Baba HA, Grabellus F, August C, Plenz G, Takeda A, Tjan TD, Schmid C, Deng MC.

J Heart Lung Transplant. 2000 Jul;19(7):668-74.

PMID:
10930816
17.

Alterations in Ca2+ cycling proteins and G alpha q signaling after left ventricular assist device support in failing human hearts.

Takeishi Y, Jalili T, Hoit BD, Kirkpatrick DL, Wagoner LE, Abraham WT, Walsh RA.

Cardiovasc Res. 2000 Mar;45(4):883-8.

PMID:
10728414
18.

Brain natriuretic peptide is produced both by cardiomyocytes and cells infiltrating the heart in patients with severe heart failure supported by a left ventricular assist device.

Bruggink AH, de Jonge N, van Oosterhout MF, Van Wichen DF, de Koning E, Lahpor JR, Kemperman H, Gmelig-Meyling FH, de Weger RA.

J Heart Lung Transplant. 2006 Feb;25(2):174-80.

PMID:
16446217
19.

Myocardial gene expression of regulators of myocyte apoptosis and myocyte calcium homeostasis during hemodynamic unloading by ventricular assist devices in patients with end-stage heart failure.

Bartling B, Milting H, Schumann H, Darmer D, Arusoglu L, Koerner MM, El-Banayosy A, Koerfer R, Holtz J, Zerkowski HR.

Circulation. 1999 Nov 9;100(19 Suppl):II216-23.

20.

Markers of autophagy are downregulated in failing human heart after mechanical unloading.

Kassiotis C, Ballal K, Wellnitz K, Vela D, Gong M, Salazar R, Frazier OH, Taegtmeyer H.

Circulation. 2009 Sep 15;120(11 Suppl):S191-7. doi: 10.1161/CIRCULATIONAHA.108.842252.

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