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Simultaneous cell-to-cell transmission of human immunodeficiency virus to multiple targets through polysynapses.

Rudnicka D, Feldmann J, Porrot F, Wietgrefe S, Guadagnini S, Prévost MC, Estaquier J, Haase AT, Sol-Foulon N, Schwartz O.

J Virol. 2009 Jun;83(12):6234-46. doi: 10.1128/JVI.00282-09. Epub 2009 Apr 15.


Inefficient human immunodeficiency virus replication in mobile lymphocytes.

Sourisseau M, Sol-Foulon N, Porrot F, Blanchet F, Schwartz O.

J Virol. 2007 Jan;81(2):1000-12. Epub 2006 Nov 1.


A role for CD81 on the late steps of HIV-1 replication in a chronically infected T cell line.

Grigorov B, Attuil-Audenis V, Perugi F, Nedelec M, Watson S, Pique C, Darlix JL, Conjeaud H, Muriaux D.

Retrovirology. 2009 Mar 11;6:28. doi: 10.1186/1742-4690-6-28.


LFA-1 Engagement Triggers T Cell Polarization at the HIV-1 Virological Synapse.

Starling S, Jolly C.

J Virol. 2016 Oct 14;90(21):9841-9854. Print 2016 Nov 1.


Human hepatoma cells transmit surface bound HIV-1 to CD4+ T cells through an ICAM-1/LFA-1-dependent mechanism.

Fromentin R, Tardif MR, Tremblay MJ.

Virology. 2010 Mar 15;398(2):168-75. doi: 10.1016/j.virol.2009.12.008. Epub 2009 Dec 24.


Human immunodeficiency virus (HIV) infection in CD4+ T lymphocytes genetically deficient in LFA-1: LFA-1 is required for HIV-mediated cell fusion but not for viral transmission.

Pantaleo G, Butini L, Graziosi C, Poli G, Schnittman SM, Greenhouse JJ, Gallin JI, Fauci AS.

J Exp Med. 1991 Feb 1;173(2):511-4.


HIV transfer between CD4 T cells does not require LFA-1 binding to ICAM-1 and is governed by the interaction of HIV envelope glycoprotein with CD4.

Puigdomènech I, Massanella M, Izquierdo-Useros N, Ruiz-Hernandez R, Curriu M, Bofill M, Martinez-Picado J, Juan M, Clotet B, Blanco J.

Retrovirology. 2008 Mar 31;5:32. doi: 10.1186/1742-4690-5-32.


[Characterization of HIV-1 Gag-specific proliferative T cell responses in Chinese HIV-1 infected patients].

Wang Q, Shang H, Wang YN, Zhang ZN, Jiang YJ.

Zhonghua Yi Xue Za Zhi. 2006 Nov 28;86(44):3099-103. Chinese.


Roles of the interactions between Env and Gag proteins in the HIV-1 replication cycle.

Murakami T.

Microbiol Immunol. 2008 May;52(5):287-95. doi: 10.1111/j.1348-0421.2008.00008.x. Review.


Impaired processing and presentation of cytotoxic-T-lymphocyte (CTL) epitopes are major escape mechanisms from CTL immune pressure in human immunodeficiency virus type 1 infection.

Yokomaku Y, Miura H, Tomiyama H, Kawana-Tachikawa A, Takiguchi M, Kojima A, Nagai Y, Iwamoto A, Matsuda Z, Ariyoshi K.

J Virol. 2004 Feb;78(3):1324-32.


The infectious synapse formed between mature dendritic cells and CD4(+) T cells is independent of the presence of the HIV-1 envelope glycoprotein.

Rodriguez-Plata MT, Puigdomènech I, Izquierdo-Useros N, Puertas MC, Carrillo J, Erkizia I, Clotet B, Blanco J, Martinez-Picado J.

Retrovirology. 2013 Apr 16;10:42. doi: 10.1186/1742-4690-10-42.


The differential ability of HLA B*5701+ long-term nonprogressors and progressors to restrict human immunodeficiency virus replication is not caused by loss of recognition of autologous viral gag sequences.

Migueles SA, Laborico AC, Imamichi H, Shupert WL, Royce C, McLaughlin M, Ehler L, Metcalf J, Liu S, Hallahan CW, Connors M.

J Virol. 2003 Jun;77(12):6889-98.


Cytopathic killing of peripheral blood CD4(+) T lymphocytes by human immunodeficiency virus type 1 appears necrotic rather than apoptotic and does not require env.

Lenardo MJ, Angleman SB, Bounkeua V, Dimas J, Duvall MG, Graubard MB, Hornung F, Selkirk MC, Speirs CK, Trageser C, Orenstein JO, Bolton DL.

J Virol. 2002 May;76(10):5082-93.

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