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Items: 1 to 20 of 109

1.

Induction of airway hyperreactivity by IL-25 is dependent on a subset of invariant NKT cells expressing IL-17RB.

Stock P, Lombardi V, Kohlrautz V, Akbari O.

J Immunol. 2009 Apr 15;182(8):5116-22. doi: 10.4049/jimmunol.0804213.

2.

A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity.

Terashima A, Watarai H, Inoue S, Sekine E, Nakagawa R, Hase K, Iwamura C, Nakajima H, Nakayama T, Taniguchi M.

J Exp Med. 2008 Nov 24;205(12):2727-33. doi: 10.1084/jem.20080698. Epub 2008 Nov 17.

3.

ICOS/ICOSL interaction is required for CD4+ invariant NKT cell function and homeostatic survival.

Akbari O, Stock P, Meyer EH, Freeman GJ, Sharpe AH, Umetsu DT, DeKruyff RH.

J Immunol. 2008 Apr 15;180(8):5448-56.

4.

Invariant NKT cell defects in vitamin D receptor knockout mice prevents experimental lung inflammation.

Yu S, Zhao J, Cantorna MT.

J Immunol. 2011 Nov 1;187(9):4907-12. doi: 10.4049/jimmunol.1101519. Epub 2011 Sep 23.

5.

PD-L1 and PD-L2 modulate airway inflammation and iNKT-cell-dependent airway hyperreactivity in opposing directions.

Akbari O, Stock P, Singh AK, Lombardi V, Lee WL, Freeman GJ, Sharpe AH, Umetsu DT, Dekruyff RH.

Mucosal Immunol. 2010 Jan;3(1):81-91. doi: 10.1038/mi.2009.112. Epub 2009 Sep 9.

6.

Development and function of invariant natural killer T cells producing T(h)2- and T(h)17-cytokines.

Watarai H, Sekine-Kondo E, Shigeura T, Motomura Y, Yasuda T, Satoh R, Yoshida H, Kubo M, Kawamoto H, Koseki H, Taniguchi M.

PLoS Biol. 2012 Feb;10(2):e1001255. doi: 10.1371/journal.pbio.1001255. Epub 2012 Feb 7.

7.

Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity.

Kim HY, Chang YJ, Subramanian S, Lee HH, Albacker LA, Matangkasombut P, Savage PB, McKenzie AN, Smith DE, Rottman JB, DeKruyff RH, Umetsu DT.

J Allergy Clin Immunol. 2012 Jan;129(1):216-27.e1-6. doi: 10.1016/j.jaci.2011.10.036. Epub 2011 Nov 25.

8.

Plasticity of invariant NKT cell regulation of allergic airway disease is dependent on IFN-gamma production.

Matsuda H, Takeda K, Koya T, Okamoto M, Shiraishi Y, Miyahara N, Dakhama A, Matsuda JL, Gapin L, Gelfand EW.

J Immunol. 2010 Jul 1;185(1):253-62. doi: 10.4049/jimmunol.0902301. Epub 2010 Jun 4.

9.

The development of airway hyperreactivity in T-bet-deficient mice requires CD1d-restricted NKT cells.

Kim HY, Pichavant M, Matangkasombut P, Koh YI, Savage PB, DeKruyff RH, Umetsu DT.

J Immunol. 2009 Mar 1;182(5):3252-61. doi: 10.4049/jimmunol.0803339.

10.

Production of both IL-27 and IFN-gamma after the treatment with a ligand for invariant NK T cells is responsible for the suppression of Th2 response and allergic inflammation in a mouse experimental asthma model.

Fujita H, Teng A, Nozawa R, Takamoto-Matsui Y, Katagiri-Matsumura H, Ikezawa Z, Ishii Y.

J Immunol. 2009 Jul 1;183(1):254-60. doi: 10.4049/jimmunol.0800520.

11.

Activation of nonclassical CD1d-restricted NK T cells induces airway hyperreactivity in beta 2-microglobulin-deficient mice.

Koh YI, Kim HY, Meyer EH, Pichavant M, Akbari O, Yasumi T, Savage PB, DeKruyff RH, Umetsu DT.

J Immunol. 2008 Oct 1;181(7):4560-4569.

12.

Allergic airway hyperresponsiveness-enhancing gammadelta T cells develop in normal untreated mice and fail to produce IL-4/13, unlike Th2 and NKT cells.

Jin N, Roark CL, Miyahara N, Taube C, Aydintug MK, Wands JM, Huang Y, Hahn YS, Gelfand EW, O'Brien RL, Born WK.

J Immunol. 2009 Feb 15;182(4):2002-10. doi: 10.4049/jimmunol.0803280.

13.

Glycolipid activation of invariant T cell receptor+ NK T cells is sufficient to induce airway hyperreactivity independent of conventional CD4+ T cells.

Meyer EH, Goya S, Akbari O, Berry GJ, Savage PB, Kronenberg M, Nakayama T, DeKruyff RH, Umetsu DT.

Proc Natl Acad Sci U S A. 2006 Feb 21;103(8):2782-7. Epub 2006 Feb 14.

14.

Invariant NKT cells act as an adjuvant to enhance Th2 inflammatory response in an OVA-induced mouse model of asthma.

Nie H, Yang Q, Zhang G, Wang A, He Q, Liu M, Li P, Yang J, Huang Y, Ding X, Yu H, Hu S.

PLoS One. 2015 Apr 1;10(4):e0119901. doi: 10.1371/journal.pone.0119901. eCollection 2015.

15.

CD1d restricted natural killer T cells are not required for allergic skin inflammation.

Elkhal A, Pichavant M, He R, Scott J, Meyer E, Goya S, Geha RS, Umetsu DT.

J Allergy Clin Immunol. 2006 Dec;118(6):1363-8. Epub 2006 Sep 25.

PMID:
17157667
16.

Functional invariant NKT cells in pig lungs regulate the airway hyperreactivity: a potential animal model.

Renukaradhya GJ, Manickam C, Khatri M, Rauf A, Li X, Tsuji M, Rajashekara G, Dwivedi V.

J Clin Immunol. 2011 Apr;31(2):228-39. doi: 10.1007/s10875-010-9476-4. Epub 2010 Nov 2.

17.

A CD1d-dependent antagonist inhibits the activation of invariant NKT cells and prevents development of allergen-induced airway hyperreactivity.

Lombardi V, Stock P, Singh AK, Kerzerho J, Yang W, Sullivan BA, Li X, Shiratsuchi T, Hnatiuk NE, Howell AR, Yu KO, Porcelli SA, Tsuji M, Kronenberg M, Wilson SB, Akbari O.

J Immunol. 2010 Feb 15;184(4):2107-15. doi: 10.4049/jimmunol.0901208. Epub 2010 Jan 18.

18.

Poly(I:C) promotes the production of IL-17A by murine CD1d-driven invariant NKT cells in airway inflammation.

Vultaggio A, Nencini F, Pratesi S, Petroni G, Romagnani S, Maggi E.

Allergy. 2012 Oct;67(10):1223-32. doi: 10.1111/j.1398-9995.2012.02876.x.. Epub 2012 Aug 7.

19.

Distinct and overlapping effector functions of expanded human CD4+, CD8α+ and CD4-CD8α- invariant natural killer T cells.

O'Reilly V, Zeng SG, Bricard G, Atzberger A, Hogan AE, Jackson J, Feighery C, Porcelli SA, Doherty DG.

PLoS One. 2011;6(12):e28648. doi: 10.1371/journal.pone.0028648. Epub 2011 Dec 12.

20.

α-Galactosylceramide-induced airway eosinophilia is mediated through the activation of NKT cells.

Chuang YH, Wang TC, Jen HY, Yu AL, Chiang BL.

J Immunol. 2011 Apr 15;186(8):4687-92. doi: 10.4049/jimmunol.1003659. Epub 2011 Mar 7.

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