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CD11b+ myeloid cells are the key mediators of Th2 cell homing into the airway in allergic inflammation.

Medoff BD, Seung E, Hong S, Thomas SY, Sandall BP, Duffield JS, Kuperman DA, Erle DJ, Luster AD.

J Immunol. 2009 Jan 1;182(1):623-35.


STAT6 expression in multiple cell types mediates the cooperative development of allergic airway disease.

Chapoval SP, Dasgupta P, Smith EP, DeTolla LJ, Lipsky MM, Kelly-Welch AE, Keegan AD.

J Immunol. 2011 Feb 15;186(4):2571-83. doi: 10.4049/jimmunol.1002567. Epub 2011 Jan 17.


Pulmonary IL-17E (IL-25) production and IL-17RB+ myeloid cell-derived Th2 cytokine production are dependent upon stem cell factor-induced responses during chronic allergic pulmonary disease.

Dolgachev V, Petersen BC, Budelsky AL, Berlin AA, Lukacs NW.

J Immunol. 2009 Nov 1;183(9):5705-15. doi: 10.4049/jimmunol.0901666. Epub 2009 Oct 14.


Prostaglandin I2 Suppresses Proinflammatory Chemokine Expression, CD4 T Cell Activation, and STAT6-Independent Allergic Lung Inflammation.

Zhou W, Zhang J, Goleniewska K, Dulek DE, Toki S, Newcomb DC, Cephus JY, Collins RD, Wu P, Boothby MR, Peebles RS Jr.

J Immunol. 2016 Sep 1;197(5):1577-86. doi: 10.4049/jimmunol.1501063. Epub 2016 Jul 25.


Cutting edge: Th2 cell trafficking into the allergic lung is dependent on chemoattractant receptor signaling.

Mathew A, Medoff BD, Carafone AD, Luster AD.

J Immunol. 2002 Jul 15;169(2):651-5.


A2B adenosine receptor expression by myeloid cells is proinflammatory in murine allergic-airway inflammation.

Belikoff BG, Vaickus LJ, Sitkovsky M, Remick DG.

J Immunol. 2012 Oct 1;189(7):3707-13. Epub 2012 Sep 5.


Cutting edge: STAT6 signaling in eosinophils is necessary for development of allergic airway inflammation.

Stokes K, LaMarche NM, Islam N, Wood A, Huang W, August A.

J Immunol. 2015 Mar 15;194(6):2477-81. doi: 10.4049/jimmunol.1402096. Epub 2015 Feb 13.


STAT6 controls the number of regulatory T cells in vivo, thereby regulating allergic lung inflammation.

Dorsey NJ, Chapoval SP, Smith EP, Skupsky J, Scott DW, Keegan AD.

J Immunol. 2013 Aug 15;191(4):1517-28. doi: 10.4049/jimmunol.1300486. Epub 2013 Jul 3.


TLR4/MyD88-induced CD11b+Gr-1 int F4/80+ non-migratory myeloid cells suppress Th2 effector function in the lung.

Arora M, Poe SL, Oriss TB, Krishnamoorthy N, Yarlagadda M, Wenzel SE, Billiar TR, Ray A, Ray P.

Mucosal Immunol. 2010 Nov;3(6):578-93. doi: 10.1038/mi.2010.41. Epub 2010 Jul 21. Erratum in: Mucosal Immunol. 2011 Jan;4(1):124.


Schnurri-2 regulates Th2-dependent airway inflammation and airway hyperresponsiveness.

Iwamura C, Kimura MY, Shinoda K, Endo Y, Hasegawa A, Yamashita M, Nakayama T.

Int Immunol. 2007 Jun;19(6):755-62. Epub 2007 May 9.


Pulmonary chemokine expression is coordinately regulated by STAT1, STAT6, and IFN-gamma.

Fulkerson PC, Zimmermann N, Hassman LM, Finkelman FD, Rothenberg ME.

J Immunol. 2004 Dec 15;173(12):7565-74.


CD11b+ and Sca-1+ Cells Exert the Main Beneficial Effects of Systemically Administered Bone Marrow-Derived Mononuclear Cells in a Murine Model of Mixed Th2/Th17 Allergic Airway Inflammation.

Cruz FF, Borg ZD, Goodwin M, Coffey AL, Wagner DE, Rocco PR, Weiss DJ.

Stem Cells Transl Med. 2016 Apr;5(4):488-99. doi: 10.5966/sctm.2015-0141. Epub 2016 Mar 1.


Endogenous attenuation of allergic lung inflammation by syndecan-1.

Xu J, Park PW, Kheradmand F, Corry DB.

J Immunol. 2005 May 1;174(9):5758-65.


Chemerin suppresses murine allergic asthma by inhibiting CCL2 production and subsequent airway recruitment of inflammatory dendritic cells.

Zhao L, Yang W, Yang X, Lin Y, Lv J, Dou X, Luo Q, Dong J, Chen Z, Chu Y, He R.

Allergy. 2014 Jun;69(6):763-74. doi: 10.1111/all.12408. Epub 2014 Apr 23.


Allergic pulmonary inflammation in mice is dependent on eosinophil-induced recruitment of effector T cells.

Jacobsen EA, Ochkur SI, Pero RS, Taranova AG, Protheroe CA, Colbert DC, Lee NA, Lee JJ.

J Exp Med. 2008 Mar 17;205(3):699-710. doi: 10.1084/jem.20071840. Epub 2008 Mar 3.


Protease-activated receptor 2 activation of myeloid dendritic cells regulates allergic airway inflammation.

Lewkowich IP, Day SB, Ledford JR, Zhou P, Dienger K, Wills-Karp M, Page K.

Respir Res. 2011 Sep 21;12:122. doi: 10.1186/1465-9921-12-122.


Blockade of CCR4 in a humanized model of asthma reveals a critical role for DC-derived CCL17 and CCL22 in attracting Th2 cells and inducing airway inflammation.

Perros F, Hoogsteden HC, Coyle AJ, Lambrecht BN, Hammad H.

Allergy. 2009 Jul;64(7):995-1002. doi: 10.1111/j.1398-9995.2009.02095.x.


A critical role for mouse CXC chemokine(s) in pulmonary neutrophilia during Th type 1-dependent airway inflammation.

Takaoka A, Tanaka Y, Tsuji T, Jinushi T, Hoshino A, Asakura Y, Mita Y, Watanabe K, Nakaike S, Togashi Y, Koda T, Matsushima K, Nishimura T.

J Immunol. 2001 Aug 15;167(4):2349-53.

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