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Items: 1 to 20 of 79

1.

Small-molecule aggregation inhibitors reduce excess amyloid in a trisomy 16 mouse cortical cell line.

Paula Lima AC, Arriagada C, Toro R, Cárdenas AM, Caviedes R, Ferreira ST, Caviedes P.

Biol Res. 2008;41(2):129-36. doi: /S0716-97602008000200001.

2.

Endosomal abnormalities related to amyloid precursor protein in cholesterol treated cerebral cortex neuronal cells derived from trisomy 16 mice, an animal model of Down syndrome.

Arriagada C, Astorga C, Atwater I, Rojas E, Mears D, Caviedes R, Caviedes P.

Neurosci Lett. 2007 Aug 16;423(2):172-7.

PMID:
17706358
3.

Effect of the knockdown of amyloid precursor protein on intracellular calcium increases in a neuronal cell line derived from the cerebral cortex of a trisomy 16 mouse.

Rojas G, Cárdenas AM, Fernández-Olivares P, Shimahara T, Segura-Aguilar J, Caviedes R, Caviedes P.

Exp Neurol. 2008 Jan;209(1):234-42.

PMID:
17976585
4.

Impaired cholinergic function in cell lines derived from the cerebral cortex of normal and trisomy 16 mice.

Allen DD, Martín J, Arriagada C, Cárdenas AM, Rapoport SI, Caviedes R, Caviedes P.

Eur J Neurosci. 2000 Sep;12(9):3259-64.

PMID:
10998109
5.

Apoptosis is directly related to intracellular amyloid accumulation in a cell line derived from the cerebral cortex of a trisomy 16 mouse, an animal model of Down syndrome.

Arriagada C, Bustamante M, Atwater I, Rojas E, Caviedes R, Caviedes P.

Neurosci Lett. 2010 Feb 5;470(1):81-5. doi: 10.1016/j.neulet.2009.12.062.

PMID:
20043975
6.

Altered voltage dependent calcium currents in a neuronal cell line derived from the cerebral cortex of a trisomy 16 fetal mouse, an animal model of Down syndrome.

Acuña MA, Pérez-Nuñez R, Noriega J, Cárdenas AM, Bacigalupo J, Delgado R, Arriagada C, Segura-Aguilar J, Caviedes R, Caviedes P.

Neurotox Res. 2012 Jul;22(1):59-68. doi: 10.1007/s12640-011-9304-5.

PMID:
22203612
7.

Amyloid precursor protein processing and retinal pathology in mouse models of Alzheimer's disease.

Dutescu RM, Li QX, Crowston J, Masters CL, Baird PN, Culvenor JG.

Graefes Arch Clin Exp Ophthalmol. 2009 Sep;247(9):1213-21. doi: 10.1007/s00417-009-1060-3.

PMID:
19271231
8.

Knockdown of amyloid precursor protein normalizes cholinergic function in a cell line derived from the cerebral cortex of a trisomy 16 mouse: An animal model of down syndrome.

Opazo P, Saud K, de Saint Pierre M, Cárdenas AM, Allen DD, Segura-Aguilar J, Caviedes R, Caviedes P.

J Neurosci Res. 2006 Nov 1;84(6):1303-10.

PMID:
16941497
9.

Calcium signals in cell lines derived from the cerebral cortex of normal and trisomy 16 mice.

Cárdenas AM, Rodríguez MP, Cortés MP, Alvarez RM, Wei W, Rapoport SI, Shimahara T, Caviedes R, Caviedes P.

Neuroreport. 1999 Feb 5;10(2):363-9.

PMID:
10203336
10.

Rac1 inhibition negatively regulates transcriptional activity of the amyloid precursor protein gene.

Wang PL, Niidome T, Akaike A, Kihara T, Sugimoto H.

J Neurosci Res. 2009 Jul;87(9):2105-14. doi: 10.1002/jnr.22039.

PMID:
19267423
11.

Neurotrophins and dementia--keeping in touch.

Castrén E, Tanila H.

Neuron. 2006 Jul 6;51(1):1-3. Review.

12.

GEPT extract reduces Abeta deposition by regulating the balance between production and degradation of Abeta in APPV717I transgenic mice.

Tian J, Shi J, Zhang L, Yin J, Hu Q, Xu Y, Sheng S, Wang P, Ren Y, Wang R, Wang Y.

Curr Alzheimer Res. 2009 Apr;6(2):118-31.

PMID:
19355846
13.

Vascular endothelial growth factor (VEGF) affects processing of amyloid precursor protein and beta-amyloidogenesis in brain slice cultures derived from transgenic Tg2576 mouse brain.

Bürger S, Noack M, Kirazov LP, Kirazov EP, Naydenov CL, Kouznetsova E, Yafai Y, Schliebs R.

Int J Dev Neurosci. 2009 Oct;27(6):517-23. doi: 10.1016/j.ijdevneu.2009.06.011.

PMID:
19589380
14.

A dorsal root ganglia cell line derived from trisomy 16 fetal mice, a model for Down syndrome.

Allen DD, Cárdenas AM, Arriagada C, Bennett LB, García CJ, Caviedes R, Rapoport SI, Caviedes P.

Neuroreport. 2002 Mar 25;13(4):491-6.

PMID:
11930168
15.

Increase in presenilin 1 (PS1) levels in senescence-accelerated mice (SAMP8) may indirectly impair memory by affecting amyloid precursor protein (APP) processing.

Kumar VB, Franko M, Banks WA, Kasinadhuni P, Farr SA, Vyas K, Choudhuri V, Morley JE.

J Exp Biol. 2009 Feb;212(Pt 4):494-8. doi: 10.1242/jeb.022780.

16.

The pathology of APP transgenic mice: a model of Alzheimer's disease or simply overexpression of APP?

Howlett DR, Richardson JC.

Histol Histopathol. 2009 Jan;24(1):83-100. Review.

PMID:
19012248
17.

Glutathione levels and nerve cell loss in hippocampal cultures from trisomy 16 mouse--a model of Down syndrome.

Stabel-Burow J, Kleu A, Schuchmann S, Heinemann U.

Brain Res. 1997 Aug 15;765(2):313-8.

PMID:
9313904
18.

Expression of human amyloid precursor protein in rat cortical neurons inhibits calcium oscillations.

Santos SF, Pierrot N, Morel N, Gailly P, Sindic C, Octave JN.

J Neurosci. 2009 Apr 15;29(15):4708-18. doi: 10.1523/JNEUROSCI.4917-08.2009.

19.

Thyroid hormones reverse the UV-induced repression of APP in neuroblastoma cells.

Cuesta A, Zambrano A, López E, Pascual A.

FEBS Lett. 2009 Jul 21;583(14):2401-6. doi: 10.1016/j.febslet.2009.06.040.

20.

Noggin and BMP4 co-modulate adult hippocampal neurogenesis in the APP(swe)/PS1(DeltaE9) transgenic mouse model of Alzheimer's disease.

Tang J, Song M, Wang Y, Fan X, Xu H, Bai Y.

Biochem Biophys Res Commun. 2009 Jul 31;385(3):341-5. doi: 10.1016/j.bbrc.2009.05.067.

PMID:
19463786
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