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Items: 1 to 20 of 119

1.

Fas-mediated apoptosis regulates the composition of peripheral alphabeta T cell repertoire by constitutively purging out double negative T cells.

Mohamood AS, Bargatze D, Xiao Z, Jie C, Yagita H, Ruben D, Watson J, Chakravarti S, Schneck JP, Hamad AR.

PLoS One. 2008;3(10):e3465. doi: 10.1371/journal.pone.0003465. Epub 2008 Oct 21.

3.

B220+ double-negative T cells suppress polyclonal T cell activation by a Fas-independent mechanism that involves inhibition of IL-2 production.

Hamad AR, Mohamood AS, Trujillo CJ, Huang CT, Yuan E, Schneck JP.

J Immunol. 2003 Sep 1;171(5):2421-6.

5.

Origin of CD4-CD8-B220+ T cells in MRL-lpr/lpr mice. Clues from a T cell receptor beta transgenic mouse.

Zhou T, Bluethmann H, Eldridge J, Berry K, Mountz JD.

J Immunol. 1993 Apr 15;150(8 Pt 1):3651-67.

PMID:
7682246
6.

Evidence for early onset, polyclonal activation of T cell subsets in mice homozygous for lpr.

Giese T, Davidson WF.

J Immunol. 1992 Nov 1;149(9):3097-106.

PMID:
1383337
7.

Double negative (DN) αβ T cells: misperception and overdue recognition.

Martina MN, Noel S, Saxena A, Rabb H, Hamad AR.

Immunol Cell Biol. 2015 Mar;93(3):305-10. doi: 10.1038/icb.2014.99. Epub 2014 Nov 25. Review.

9.

Regulation of apoptosis in mature alphabeta+CD4-CD8- antigen-specific suppressor T cell clones.

Khan Q, Penninger JM, Yang L, Marra LE, Kozieradzki I, Zhang L.

J Immunol. 1999 May 15;162(10):5860-7.

11.

FcRγ controls the fas-dependent regulatory function of lymphoproliferative double negative T cells.

Juvet SC, Thomson CW, Kim EY, Han M, Zhang L.

PLoS One. 2013 Jun 6;8(6):e65253. doi: 10.1371/journal.pone.0065253. Print 2013.

12.

Loss of P2X7 receptor plasma membrane expression and function in pathogenic B220+ double-negative T lymphocytes of autoimmune MRL/lpr mice.

Le Gall SM, Legrand J, Benbijja M, Safya H, Benihoud K, Kanellopoulos JM, Bobé P.

PLoS One. 2012;7(12):e52161. doi: 10.1371/journal.pone.0052161. Epub 2012 Dec 21.

13.
14.

A model for the origin of TCR-alphabeta+ CD4-CD8- B220+ cells based on high affinity TCR signals.

Mixter PF, Russell JQ, Morrissette GJ, Charland C, Aleman-Hoey D, Budd RC.

J Immunol. 1999 May 15;162(10):5747-56.

15.

Changes in sensitivity of peripheral lymphocytes of autoimmune gld mice to FasL-mediated apoptosis reveal a mechanism for the preferential deletion of CD4-CD8-B220+ T cells.

Xiao S, Zhang X, Mann KK, Jodo S, Li L, Jarjour WN, Marshak-Rothstein A, Sherr DH, Ju ST.

Int Immunol. 2004 May;16(5):759-66. Epub 2004 Apr 13.

PMID:
15096479
16.
17.

Cytokine secretion by C3H-lpr and -gld T cells. Hypersecretion of IFN-gamma and tumor necrosis factor-alpha by stimulated CD4+ T cells.

Davidson WF, Calkins C, Hügins A, Giese T, Holmes KL.

J Immunol. 1991 Jun 15;146(12):4138-48.

PMID:
1674953
18.

MRL/lpr CD4- CD8- and CD8+ T cells, respectively, mediate Fas-dependent and perforin cytotoxic pathways.

Benihoud K, Bonardelle D, Bobé P, Kiger N.

Eur J Immunol. 1997 Feb;27(2):415-20.

PMID:
9045912
19.

Intestinal intraepithelial lymphocyte T cells are resistant to lpr gene-induced T cell abnormalities.

Aicher WK, Fujihashi K, Taguchi T, McGhee JR, Yamamoto M, Eldridge JH, Gay S, Kiyono H.

Eur J Immunol. 1992 Jan;22(1):137-45.

PMID:
1346108
20.

Sp1 is the major fasl gene activator in abnormal CD4(-)CD8(-)B220(+) T cells of lpr and gld mice.

Xiao S, Marshak-Rothstein A, Ju ST.

Eur J Immunol. 2001 Nov;31(11):3339-48.

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