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Items: 1 to 20 of 156

1.

RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability.

Xu D, Guo R, Sobeck A, Bachrati CZ, Yang J, Enomoto T, Brown GW, Hoatlin ME, Hickson ID, Wang W.

Genes Dev. 2008 Oct 15;22(20):2843-55. doi: 10.1101/gad.1708608.

2.

BLAP18/RMI2, a novel OB-fold-containing protein, is an essential component of the Bloom helicase-double Holliday junction dissolvasome.

Singh TR, Ali AM, Busygina V, Raynard S, Fan Q, Du CH, Andreassen PR, Sung P, Meetei AR.

Genes Dev. 2008 Oct 15;22(20):2856-68. doi: 10.1101/gad.1725108.

3.

Crystal structures of RMI1 and RMI2, two OB-fold regulatory subunits of the BLM complex.

Wang F, Yang Y, Singh TR, Busygina V, Guo R, Wan K, Wang W, Sung P, Meetei AR, Lei M.

Structure. 2010 Sep 8;18(9):1159-70. doi: 10.1016/j.str.2010.06.008.

5.

Role of replication protein A in double holliday junction dissolution mediated by the BLM-Topo IIIα-RMI1-RMI2 protein complex.

Xue X, Raynard S, Busygina V, Singh AK, Sung P.

J Biol Chem. 2013 May 17;288(20):14221-7. doi: 10.1074/jbc.M113.465609. Epub 2013 Mar 30.

6.

Structure and cellular roles of the RMI core complex from the bloom syndrome dissolvasome.

Hoadley KA, Xu D, Xue Y, Satyshur KA, Wang W, Keck JL.

Structure. 2010 Sep 8;18(9):1149-58. doi: 10.1016/j.str.2010.06.009.

7.

BLAP75/RMI1 promotes the BLM-dependent dissolution of homologous recombination intermediates.

Wu L, Bachrati CZ, Ou J, Xu C, Yin J, Chang M, Wang W, Li L, Brown GW, Hickson ID.

Proc Natl Acad Sci U S A. 2006 Mar 14;103(11):4068-73. Epub 2006 Mar 6.

8.

Accumulation and Phosphorylation of RecQ-Mediated Genome Instability Protein 1 (RMI1) at Serine 284 and Serine 292 during Mitosis.

Xu C, Wang Y, Wang L, Wang Q, Du LQ, Fan S, Liu Q, Li L.

Int J Mol Sci. 2015 Nov 4;16(11):26395-405. doi: 10.3390/ijms161125965.

9.

Multifaceted role of the Topo IIIα-RMI1-RMI2 complex and DNA2 in the BLM-dependent pathway of DNA break end resection.

Daley JM, Chiba T, Xue X, Niu H, Sung P.

Nucleic Acids Res. 2014;42(17):11083-91. doi: 10.1093/nar/gku803. Epub 2014 Sep 8.

10.

TopBP1 interacts with BLM to maintain genome stability but is dispensable for preventing BLM degradation.

Blackford AN, Nieminuszczy J, Schwab RA, Galanty Y, Jackson SP, Niedzwiedz W.

Mol Cell. 2015 Mar 19;57(6):1133-1141. doi: 10.1016/j.molcel.2015.02.012.

11.

More complexity to the Bloom's syndrome complex.

Liu Y, West SC.

Genes Dev. 2008 Oct 15;22(20):2737-42. doi: 10.1101/gad.1732808.

12.

Loss of RMI2 Increases Genome Instability and Causes a Bloom-Like Syndrome.

Hudson DF, Amor DJ, Boys A, Butler K, Williams L, Zhang T, Kalitsis P.

PLoS Genet. 2016 Dec 15;12(12):e1006483. doi: 10.1371/journal.pgen.1006483. eCollection 2016 Dec.

13.

Defining the molecular interface that connects the Fanconi anemia protein FANCM to the Bloom syndrome dissolvasome.

Hoadley KA, Xue Y, Ling C, Takata M, Wang W, Keck JL.

Proc Natl Acad Sci U S A. 2012 Mar 20;109(12):4437-42. doi: 10.1073/pnas.1117279109. Epub 2012 Mar 5.

14.

BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity.

Yin J, Sobeck A, Xu C, Meetei AR, Hoatlin M, Li L, Wang W.

EMBO J. 2005 Apr 6;24(7):1465-76. Epub 2005 Mar 17.

15.

TopBP1 controls BLM protein level to maintain genome stability.

Wang J, Chen J, Gong Z.

Mol Cell. 2013 Dec 12;52(5):667-78. doi: 10.1016/j.molcel.2013.10.012. Epub 2013 Nov 14.

16.

Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replication.

Xu D, Muniandy P, Leo E, Yin J, Thangavel S, Shen X, Ii M, Agama K, Guo R, Fox D 3rd, Meetei AR, Wilson L, Nguyen H, Weng NP, Brill SJ, Li L, Vindigni A, Pommier Y, Seidman M, Wang W.

EMBO J. 2010 Sep 15;29(18):3140-55. doi: 10.1038/emboj.2010.186. Epub 2010 Aug 13.

17.

Dissolution of double Holliday junctions by the concerted action of BLM and topoisomerase IIIalpha.

Bachrati CZ, Hickson ID.

Methods Mol Biol. 2009;582:91-102. doi: 10.1007/978-1-60761-340-4_8.

PMID:
19763944
18.

Vertebrate WRNIP1 and BLM are required for efficient maintenance of genome stability.

Hayashi T, Seki M, Inoue E, Yoshimura A, Kusa Y, Tada S, Enomoto T.

Genes Genet Syst. 2008 Feb;83(1):95-100.

19.

Evidence for BLM and Topoisomerase IIIalpha interaction in genomic stability.

Hu P, Beresten SF, van Brabant AJ, Ye TZ, Pandolfi PP, Johnson FB, Guarente L, Ellis NA.

Hum Mol Genet. 2001 Jun 1;10(12):1287-98.

PMID:
11406610
20.

Human topoisomerase IIIalpha is a single-stranded DNA decatenase that is stimulated by BLM and RMI1.

Yang J, Bachrati CZ, Ou J, Hickson ID, Brown GW.

J Biol Chem. 2010 Jul 9;285(28):21426-36. doi: 10.1074/jbc.M110.123216. Epub 2010 May 5.

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