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Items: 1 to 20 of 67

1.

Letter by Strande regarding article "Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy".

Strande JL.

Circulation. 2008 Jun 17;117(24):e495; author reply e496. doi: 10.1161/CIRCULATIONAHA.107.758219. No abstract available.

2.

Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy.

Pawlinski R, Tencati M, Hampton CR, Shishido T, Bullard TA, Casey LM, Andrade-Gordon P, Kotzsch M, Spring D, Luther T, Abe J, Pohlman TH, Verrier ED, Blaxall BC, Mackman N.

Circulation. 2007 Nov 13;116(20):2298-306. Epub 2007 Oct 29.

3.

Progression from hypertrophic to dilated cardiomyopathy in mice that express a mutant myosin transgene.

Freeman K, Colon-Rivera C, Olsson MC, Moore RL, Weinberger HD, Grupp IL, Vikstrom KL, Iaccarino G, Koch WJ, Leinwand LA.

Am J Physiol Heart Circ Physiol. 2001 Jan;280(1):H151-9.

4.

Etiology-dependency of ionic remodeling in cardiomyopathic rabbits.

Verkerk AO, Baartscheer A, de Groot JR, Wilders R, Coronel R.

Int J Cardiol. 2011 Apr 14;148(2):154-60. doi: 10.1016/j.ijcard.2009.10.047. Epub 2009 Nov 17.

PMID:
19919884
5.

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress.

Huang ZP, Chen J, Seok HY, Zhang Z, Kataoka M, Hu X, Wang DZ.

Circ Res. 2013 Apr 26;112(9):1234-43. doi: 10.1161/CIRCRESAHA.112.300682. Epub 2013 Mar 22.

6.

Dilated cardiomyopathy in Erb-b4-deficient ventricular muscle.

García-Rivello H, Taranda J, Said M, Cabeza-Meckert P, Vila-Petroff M, Scaglione J, Ghio S, Chen J, Lai C, Laguens RP, Lloyd KC, Hertig CM.

Am J Physiol Heart Circ Physiol. 2005 Sep;289(3):H1153-60. Epub 2005 Apr 29.

7.

Substance P is associated with heart enlargement and apoptosis in murine dilated cardiomyopathy induced by Taenia crassiceps infection.

D'Souza M, Garza MA, Xie M, Weinstock J, Xiang Q, Robinson P.

J Parasitol. 2007 Oct;93(5):1121-7. doi: 10.1645/GE-596R1.1.

PMID:
18163347
8.

Transgenic overexpression of platelet-derived growth factor-C in the mouse heart induces cardiac fibrosis, hypertrophy, and dilated cardiomyopathy.

Pontén A, Li X, Thorén P, Aase K, Sjöblom T, Ostman A, Eriksson U.

Am J Pathol. 2003 Aug;163(2):673-82.

9.

MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failure.

Arber S, Hunter JJ, Ross J Jr, Hongo M, Sansig G, Borg J, Perriard JC, Chien KR, Caroni P.

Cell. 1997 Feb 7;88(3):393-403.

10.

The transcription factor GATA-6 regulates pathological cardiac hypertrophy.

van Berlo JH, Elrod JW, van den Hoogenhof MM, York AJ, Aronow BJ, Duncan SA, Molkentin JD.

Circ Res. 2010 Oct 15;107(8):1032-40. doi: 10.1161/CIRCRESAHA.110.220764. Epub 2010 Aug 12.

11.

Adenine nucleotide translocase 1 deficiency results in dilated cardiomyopathy with defects in myocardial mechanics, histopathological alterations, and activation of apoptosis.

Narula N, Zaragoza MV, Sengupta PP, Li P, Haider N, Verjans J, Waymire K, Vannan M, Wallace DC.

JACC Cardiovasc Imaging. 2011 Jan;4(1):1-10. doi: 10.1016/j.jcmg.2010.06.018.

12.

Activating transcription factor 3 deficiency promotes cardiac hypertrophy, dysfunction, and fibrosis induced by pressure overload.

Zhou H, Shen DF, Bian ZY, Zong J, Deng W, Zhang Y, Guo YY, Li H, Tang QZ.

PLoS One. 2011;6(10):e26744. doi: 10.1371/journal.pone.0026744. Epub 2011 Oct 28.

13.

[Evaluation of myocardial disorders in patients with dilated cardiomyopathy and left ventricular eccentric hypertrophy--by 201Tl myocardial SPECT].

Yamazaki J, Ohsawa H, Uchi T, Iida M, Wakakura M, Okamoto K, Hosoi H, Morishita T, Nakano H, Yabe Y.

Kokyu To Junkan. 1992 Mar;40(3):275-81. Japanese.

PMID:
1533725
14.

Pathological and physiological hypertrophies are regulated by distinct gene programs.

Beisvag V, Kemi OJ, Arbo I, Loennechen JP, Wisløff U, Langaas M, Sandvik AK, Ellingsen Ø.

Eur J Cardiovasc Prev Rehabil. 2009 Dec;16(6):690-7. doi: 10.1097/HJR.0b013e32833158a2.

PMID:
19809332
15.

The E-domain region of mechano-growth factor inhibits cellular apoptosis and preserves cardiac function during myocardial infarction.

Mavrommatis E, Shioura KM, Los T, Goldspink PH.

Mol Cell Biochem. 2013 Sep;381(1-2):69-83. doi: 10.1007/s11010-013-1689-4. Epub 2013 May 28.

16.

Cardiac oxidative stress and remodeling following infarction: role of NADPH oxidase.

Zhao W, Zhao D, Yan R, Sun Y.

Cardiovasc Pathol. 2009 May-Jun;18(3):156-66. doi: 10.1016/j.carpath.2007.12.013. Epub 2008 Mar 4.

17.

Transgenic over-expression of YY1 induces pathologic cardiac hypertrophy in a sex-specific manner.

Stauffer BL, Dockstader K, Russell G, Hijmans J, Walker L, Cecil M, Demos-Davies K, Medway A, McKinsey TA, Sucharov CC.

Biochem Biophys Res Commun. 2015 Jun 26;462(2):131-7. doi: 10.1016/j.bbrc.2015.04.106. Epub 2015 Apr 29.

18.

Aspects of left ventricular volume comparison between 3D echocardiography and MRI.

Voormolen MM, Danilouchkine MG.

J Am Soc Echocardiogr. 2007 Dec;20(12):1421-2. Epub 2007 Sep 29. No abstract available.

PMID:
17904805
19.

Molecular mechanisms of inherited cardiomyopathies.

Fatkin D, Graham RM.

Physiol Rev. 2002 Oct;82(4):945-80. Review.

20.

AMPK deficiency in cardiac muscle results in dilated cardiomyopathy in the absence of changes in energy metabolism.

Sung MM, Zordoky BN, Bujak AL, Lally JS, Fung D, Young ME, Horman S, Miller EJ, Light PE, Kemp BE, Steinberg GR, Dyck JR.

Cardiovasc Res. 2015 Jul 15;107(2):235-45. doi: 10.1093/cvr/cvv166. Epub 2015 May 28.

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