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Items: 1 to 20 of 177

1.

Early atherosclerosis in humans: role of diffuse intimal thickening and extracellular matrix proteoglycans.

Nakashima Y, Wight TN, Sueishi K.

Cardiovasc Res. 2008 Jul 1;79(1):14-23. doi: 10.1093/cvr/cvn099. Review.

PMID:
18430750
2.

Early human atherosclerosis: accumulation of lipid and proteoglycans in intimal thickenings followed by macrophage infiltration.

Nakashima Y, Fujii H, Sumiyoshi S, Wight TN, Sueishi K.

Arterioscler Thromb Vasc Biol. 2007 May;27(5):1159-65.

3.

Normal and oxidized low density lipoproteins accumulate deep in physiologically thickened intima of human coronary arteries.

Fukuchi M, Watanabe J, Kumagai K, Baba S, Shinozaki T, Miura M, Kagaya Y, Shirato K.

Lab Invest. 2002 Oct;82(10):1437-47.

PMID:
12379778
4.

Transplant vascular disease: role of lipids and proteoglycans.

Rahmani M, McDonald PC, Wong BW, McManus BM.

Can J Cardiol. 2004 Aug;20 Suppl B:58B-65B. Review.

PMID:
15309207
5.

A definition of advanced types of atherosclerotic lesions and a histological classification of atherosclerosis. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association.

Stary HC, Chandler AB, Dinsmore RE, Fuster V, Glagov S, Insull W Jr, Rosenfeld ME, Schwartz CJ, Wagner WD, Wissler RW.

Circulation. 1995 Sep 1;92(5):1355-74. Review.

6.

Natural progression of atherosclerosis from pathologic intimal thickening to late fibroatheroma in human coronary arteries: A pathology study.

Otsuka F, Kramer MC, Woudstra P, Yahagi K, Ladich E, Finn AV, de Winter RJ, Kolodgie FD, Wight TN, Davis HR, Joner M, Virmani R.

Atherosclerosis. 2015 Aug;241(2):772-82. doi: 10.1016/j.atherosclerosis.2015.05.011.

7.

Hyperelongated biglycan: the surreptitious initiator of atherosclerosis.

Little PJ, Osman N, O'Brien KD.

Curr Opin Lipidol. 2008 Oct;19(5):448-54. doi: 10.1097/MOL.0b013e32830dd7c4. Review.

PMID:
18769225
8.
12.

The extracellular matrix on atherogenesis and diabetes-associated vascular disease.

Camejo G, Olsson U, Hurt-Camejo E, Baharamian N, Bondjers G.

Atheroscler Suppl. 2002 May;3(1):3-9. Review.

PMID:
12044579
13.

[Role of inflammation in atherogenesis].

Rodríguez G, Mago N, Rosa F.

Invest Clin. 2009 Mar;50(1):109-29. Review. Spanish.

PMID:
19418732
14.

Monocyte recruitment and foam cell formation in atherosclerosis.

Bobryshev YV.

Micron. 2006;37(3):208-22. Review.

PMID:
16360317
15.

Macrophage-mediated proteolytic remodeling of the extracellular matrix in atherosclerosis results in neoepitopes: a potential new class of biochemical markers.

Skjøt-Arkil H, Barascuk N, Register T, Karsdal MA.

Assay Drug Dev Technol. 2010 Oct;8(5):542-52. doi: 10.1089/adt.2009.0258. Review.

PMID:
20662734
16.

Fatty acid-induced atherogenic changes in extracellular matrix proteoglycans.

Rodriguéz-Lee M, Bondjers G, Camejo G.

Curr Opin Lipidol. 2007 Oct;18(5):546-53. Review.

PMID:
17885426
17.

Endostatin binds biglycan and LDL and interferes with LDL retention to the subendothelial matrix during atherosclerosis.

Zeng X, Chen J, Miller YI, Javaherian K, Moulton KS.

J Lipid Res. 2005 Sep;46(9):1849-59.

18.

Progression of atherosclerosis: the cell biology.

Chait A.

Eur Heart J. 1987 Aug;8 Suppl E:15-22. Review.

PMID:
3315672
20.

Association between complement factor H and proteoglycans in early human coronary atherosclerotic lesions: implications for local regulation of complement activation.

Oksjoki R, Jarva H, Kovanen PT, Laine P, Meri S, Pentikäinen MO.

Arterioscler Thromb Vasc Biol. 2003 Apr 1;23(4):630-6.

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