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Items: 1 to 20 of 181

1.

Cell division in Escherichia coli cultures monitored at single cell resolution.

Roostalu J, Jõers A, Luidalepp H, Kaldalu N, Tenson T.

BMC Microbiol. 2008 Apr 23;8:68. doi: 10.1186/1471-2180-8-68.

2.

Persisters: a distinct physiological state of E. coli.

Shah D, Zhang Z, Khodursky A, Kaldalu N, Kurg K, Lewis K.

BMC Microbiol. 2006 Jun 12;6:53.

3.

Age of inoculum strongly influences persister frequency and can mask effects of mutations implicated in altered persistence.

Luidalepp H, Jõers A, Kaldalu N, Tenson T.

J Bacteriol. 2011 Jul;193(14):3598-605. doi: 10.1128/JB.00085-11.

4.

The frequency of persisters in Escherichia coli reflects the kinetics of awakening from dormancy.

Jõers A, Kaldalu N, Tenson T.

J Bacteriol. 2010 Jul;192(13):3379-84. doi: 10.1128/JB.00056-10.

5.

The formation of persister cells in stationary-phase cultures of Escherichia coli is associated with the aggregation of endogenous proteins.

Leszczynska D, Matuszewska E, Kuczynska-Wisnik D, Furmanek-Blaszk B, Laskowska E.

PLoS One. 2013;8(1):e54737. doi: 10.1371/journal.pone.0054737.

6.

Dual fluorescence system for flow cytometric analysis of Escherichia coli transcriptional response in multi-species context.

Miao H, Ratnasingam S, Pu CS, Desai MM, Sze CC.

J Microbiol Methods. 2009 Feb;76(2):109-19. doi: 10.1016/j.mimet.2008.09.015.

PMID:
18926860
7.

Rapid flow cytometric assessment of mecillinam and ampicillin bacterial susceptibility.

Walberg M, Gaustad P, Steen HB.

J Antimicrob Chemother. 1996 Jun;37(6):1063-75.

PMID:
8836810
9.

Specialized persister cells and the mechanism of multidrug tolerance in Escherichia coli.

Keren I, Shah D, Spoering A, Kaldalu N, Lewis K.

J Bacteriol. 2004 Dec;186(24):8172-80.

10.

Role of oxidative stress in persister tolerance.

Wu Y, Vulić M, Keren I, Lewis K.

Antimicrob Agents Chemother. 2012 Sep;56(9):4922-6. doi: 10.1128/AAC.00921-12.

11.

Bacterial persistence as a phenotypic switch.

Balaban NQ, Merrin J, Chait R, Kowalik L, Leibler S.

Science. 2004 Sep 10;305(5690):1622-5.

12.
13.

Phenotypic heterogeneity enables uropathogenic Escherichia coli to evade killing by antibiotics and serum complement.

Putrinš M, Kogermann K, Lukk E, Lippus M, Varik V, Tenson T.

Infect Immun. 2015 Mar;83(3):1056-67. doi: 10.1128/IAI.02725-14.

14.

Dormancy is not necessary or sufficient for bacterial persistence.

Orman MA, Brynildsen MP.

Antimicrob Agents Chemother. 2013 Jul;57(7):3230-9. doi: 10.1128/AAC.00243-13.

15.

Persister Heterogeneity Arising from a Single Metabolic Stress.

Amato SM, Brynildsen MP.

Curr Biol. 2015 Aug 17;25(16):2090-8. doi: 10.1016/j.cub.2015.06.034.

16.

Characteristics and dynamics of bacterial populations during postantibiotic effect determined by flow cytometry.

Gottfredsson M, Erlendsdóttir H, Sigfússon A, Gudmundsson S.

Antimicrob Agents Chemother. 1998 May;42(5):1005-11.

17.

Energy production genes sucB and ubiF are involved in persister survival and tolerance to multiple antibiotics and stresses in Escherichia coli.

Ma C, Sim S, Shi W, Du L, Xing D, Zhang Y.

FEMS Microbiol Lett. 2010 Feb;303(1):33-40. doi: 10.1111/j.1574-6968.2009.01857.x.

18.
19.

Persister cells mediate tolerance to metal oxyanions in Escherichia coli.

Harrison JJ, Ceri H, Roper NJ, Badry EA, Sproule KM, Turner RJ.

Microbiology. 2005 Oct;151(Pt 10):3181-95.

PMID:
16207903
20.

Stationary phase protein overproduction is a fundamental capability of Escherichia coli.

Ou J, Wang L, Ding X, Du J, Zhang Y, Chen H, Xu A.

Biochem Biophys Res Commun. 2004 Jan 30;314(1):174-80.

PMID:
14715262
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