Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 85

1.

Herpes simplex virus virion host shutoff attenuates establishment of the antiviral state.

Pasieka TJ, Lu B, Crosby SD, Wylie KM, Morrison LA, Alexander DE, Menachery VD, Leib DA.

J Virol. 2008 Jun;82(11):5527-35. doi: 10.1128/JVI.02047-07.

3.

Selective ablation of virion host shutoff protein RNase activity attenuates herpes simplex virus 2 in mice.

Korom M, Wylie KM, Morrison LA.

J Virol. 2008 Apr;82(7):3642-53. doi: 10.1128/JVI.02409-07.

5.

Interferons regulate the phenotype of wild-type and mutant herpes simplex viruses in vivo.

Leib DA, Harrison TE, Laslo KM, Machalek MA, Moorman NJ, Virgin HW.

J Exp Med. 1999 Feb 15;189(4):663-72.

6.

The virion host shutoff protein of herpes simplex virus 1 blocks the replication-independent activation of NF-κB in dendritic cells in the absence of type I interferon signaling.

Cotter CR, Kim WK, Nguyen ML, Yount JS, López CB, Blaho JA, Moran TM.

J Virol. 2011 Dec;85(23):12662-72. doi: 10.1128/JVI.05557-11.

7.
9.

The herpes simplex virus virion host shutoff function.

Kwong AD, Frenkel N.

J Virol. 1989 Nov;63(11):4834-9.

11.

Herpesvirus papio 2 encodes a virion host shutoff function.

Bigger JE, Martin DW.

Virology. 2002 Dec 5;304(1):33-43.

12.

Herpes simplex virus virion host shutoff (vhs) activity alters periocular disease in mice.

Smith TJ, Ackland-Berglund CE, Leib DA.

J Virol. 2000 Apr;74(8):3598-604.

13.
14.

Effects of herpes simplex virus on mRNA stability.

Strom T, Frenkel N.

J Virol. 1987 Jul;61(7):2198-207.

15.

Pathogenesis of herpes simplex virus type 2 virion host shutoff (vhs) mutants.

Smith TJ, Morrison LA, Leib DA.

J Virol. 2002 Mar;76(5):2054-61.

16.

Type I IFN response to Papiine herpesvirus 2 (Herpesvirus papio 2; HVP2) determines neuropathogenicity in mice.

Rogers KM, Deatheridge M, Breshears MA, Chapman S, Black D, Ritchey JW, Payton M, Eberle R.

Virology. 2009 Apr 10;386(2):280-9. doi: 10.1016/j.virol.2009.01.001.

18.

Supplemental Content

Support Center