Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 147

1.

Injury enhances resistance to Escherichia coli infection by boosting innate immune system function.

Maung AA, Fujimi S, MacConmara MP, Tajima G, McKenna AM, Delisle AJ, Stallwood C, Onderdonk AB, Mannick JA, Lederer JA.

J Immunol. 2008 Feb 15;180(4):2450-8.

2.

Mast cells and resistance to peritoneal sepsis after burn injury.

Shelley O, Murphy T, Lederer JA, Mannick JA, Rodrick ML.

Shock. 2003 Jun;19(6):513-8.

PMID:
12785005
3.

Burn injury initiates a shift in superantigen-induced T cell responses and host survival.

Zang Y, Dolan SM, Ni Choileain N, Kriynovich SJ, Murphy TJ, Sayles P, Mannick JA, Lederer JA.

J Immunol. 2004 Apr 15;172(8):4883-92.

4.

A novel antibacterial gene transfer treatment for multidrug-resistant Acinetobacter baumannii-induced burn sepsis.

Shankar R, He LK, Szilagyi A, Muthu K, Gamelli RL, Filutowicz M, Wendt JL, Suzuki H, Dominguez M.

J Burn Care Res. 2007 Jan-Feb;28(1):6-12.

PMID:
17211194
5.

Temporal correlation of impaired immune response after thermal injury with susceptibility to infection in a murine model.

Moss NM, Gough DB, Jordan AL, Grbic JT, Wood JJ, Rodrick ML, Mannick JA.

Surgery. 1988 Nov;104(5):882-7.

PMID:
3263707
6.
7.

Toll-like receptor 2 and 4 ligation results in complex altered cytokine profiles early and late after burn injury.

Cairns BA, Barnes CM, Mlot S, Meyer AA, Maile R.

J Trauma. 2008 Apr;64(4):1069-77; discussion 1077-8. doi: 10.1097/TA.0b013e318166b7d9.

PMID:
18404077
8.

Production of granulocyte colony-stimulating factor in the nonspecific acute phase response enhances host resistance to bacterial infection.

Noursadeghi M, Bickerstaff MC, Herbert J, Moyes D, Cohen J, Pepys MB.

J Immunol. 2002 Jul 15;169(2):913-9.

9.
10.

Influence of systemic inflammatory response syndrome on host resistance against bacterial infections.

Takahashi H, Tsuda Y, Takeuchi D, Kobayashi M, Herndon DN, Suzuki F.

Crit Care Med. 2004 Sep;32(9):1879-85.

PMID:
15343016
11.

Enterococcus faecalis translocation in mice with severe burn injury: a pathogenic role of CCL2 and alternatively activated macrophages (M2aMphi and M2cMphi).

Shigematsu K, Asai A, Kobayashi M, Herndon DN, Suzuki F.

J Leukoc Biol. 2009 Oct;86(4):999-1005. doi: 10.1189/jlb.0409235. Epub 2009 Jul 21.

PMID:
19622799
12.

Essential involvement of CX3CR1-mediated signals in the bactericidal host defense during septic peritonitis.

Ishida Y, Hayashi T, Goto T, Kimura A, Akimoto S, Mukaida N, Kondo T.

J Immunol. 2008 Sep 15;181(6):4208-18.

13.

Effect of acute ethanol exposure on the dermal inflammatory response after burn injury.

Faunce DE, Garner JL, Llanas JN, Patel PJ, Gregory MS, Duffner LA, Gamelli RL, Kovacs EJ.

Alcohol Clin Exp Res. 2003 Jul;27(7):1199-206.

PMID:
12878929
14.

Burn-Evoked Reactive Oxygen Species Immediately After Injury are Crucial to Restore the Neutrophil Function Against Postburn Infection in Mice.

Miyazaki H, Kinoshita M, Ono S, Seki S, Saitoh D.

Shock. 2015 Sep;44(3):252-7. doi: 10.1097/SHK.0000000000000404.

PMID:
26009813
15.

Beneficial versus detrimental effects of neutrophils are determined by the nature of the insult.

Perl M, Chung CS, Perl U, Biffl WL, Cioffi WG, Ayala A.

J Am Coll Surg. 2007 May;204(5):840-52; discussion 852-3.

PMID:
17481496
16.

Early infection during burn-induced inflammatory response results in increased mortality and p38-mediated neutrophil dysfunction.

Adediran SG, Dauplaise DJ, Kasten KR, Tschöp J, Dattilo J, Goetzman HS, England LG, Cave CM, Robinson CT, Caldwell CC.

Am J Physiol Regul Integr Comp Physiol. 2010 Sep;299(3):R918-25. doi: 10.1152/ajpregu.00132.2010. Epub 2010 Jun 30.

17.

Receptor for advanced glycation end products facilitates host defense during Escherichia coli-induced abdominal sepsis in mice.

van Zoelen MA, Schmidt AM, Florquin S, Meijers JC, de Beer R, de Vos AF, Nawroth PP, Bierhaus A, van der Poll T.

J Infect Dis. 2009 Sep 1;200(5):765-73. doi: 10.1086/604730. Erratum in: J Infect Dis. 2009 Nov 15;200(10):1629.

PMID:
19627249
18.

Dendritic cells modulate lung response to Pseudomonas aeruginosa in a murine model of sepsis-induced immune dysfunction.

Pène F, Zuber B, Courtine E, Rousseau C, Ouaaz F, Toubiana J, Tazi A, Mira JP, Chiche JD.

J Immunol. 2008 Dec 15;181(12):8513-20.

19.

Gr-1(+)CD11b(+) cells as an accelerator of sepsis stemming from Pseudomonas aeruginosa wound infection in thermally injured mice.

Kobayashi M, Yoshida T, Takeuchi D, Jones VC, Shigematsu K, Herndon DN, Suzuki F.

J Leukoc Biol. 2008 Jun;83(6):1354-62. doi: 10.1189/jlb.0807541. Epub 2008 Mar 27.

PMID:
18372338
20.

Restoration of natural IgM production from liver B cells by exogenous IL-18 improves the survival of burn-injured mice infected with Pseudomonas aeruginosa.

Kinoshita M, Shinomiya N, Ono S, Tsujimoto H, Kawabata T, Matsumoto A, Hiraide H, Seki S.

J Immunol. 2006 Oct 1;177(7):4627-35.

Supplemental Content

Support Center