Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 106

1.

Hypoxia exposure induces the emergence of fibroblasts lacking replication repressor signals of PKCzeta in the pulmonary artery adventitia.

Das M, Burns N, Wilson SJ, Zawada WM, Stenmark KR.

Cardiovasc Res. 2008 Jun 1;78(3):440-8. doi: 10.1093/cvr/cvn014. Epub 2008 Jan 24.

PMID:
18218684
2.

Hypoxia induces unique proliferative response in adventitial fibroblasts by activating PDGFβ receptor-JNK1 signalling.

Panzhinskiy E, Zawada WM, Stenmark KR, Das M.

Cardiovasc Res. 2012 Aug 1;95(3):356-65. doi: 10.1093/cvr/cvs194. Epub 2012 Jun 26.

3.

Protein kinase Czeta attenuates hypoxia-induced proliferation of fibroblasts by regulating MAP kinase phosphatase-1 expression.

Short MD, Fox SM, Lam CF, Stenmark KR, Das M.

Mol Biol Cell. 2006 Apr;17(4):1995-2008. Epub 2006 Feb 8.

4.

Chronic hypoxia induces exaggerated growth responses in pulmonary artery adventitial fibroblasts: potential contribution of specific protein kinase c isozymes.

Das M, Dempsey EC, Bouchey D, Reyland ME, Stenmark KR.

Am J Respir Cell Mol Biol. 2000 Jan;22(1):15-25.

PMID:
10615061
5.

Pulmonary hyperplasia and the two sides of PKCzeta.

Graf K.

Cardiovasc Res. 2008 Jun 1;78(3):409-10. doi: 10.1093/cvr/cvn089. Epub 2008 Apr 8. No abstract available.

PMID:
18397964
6.

Hypoxia-induced proliferative response of vascular adventitial fibroblasts is dependent on g protein-mediated activation of mitogen-activated protein kinases.

Das M, Bouchey DM, Moore MJ, Hopkins DC, Nemenoff RA, Stenmark KR.

J Biol Chem. 2001 May 11;276(19):15631-40. Epub 2001 Feb 22.

7.

Low-dose fluvastatin reverses the hypoxic pulmonary adventitial fibroblast phenotype in experimental pulmonary hypertension.

Carlin CM, Celnik DF, Pak O, Wadsworth R, Peacock AJ, Welsh DJ.

Am J Respir Cell Mol Biol. 2012 Aug;47(2):140-8. doi: 10.1165/rcmb.2011-0411OC. Epub 2012 Mar 1.

PMID:
22383583
8.

Pulmonary artery smooth muscle cells from chronically hypoxic neonatal calves retain fetal-like and acquire new growth properties.

Xu Y, Stenmark KR, Das M, Walchak SJ, Ruff LJ, Dempsey EC.

Am J Physiol. 1997 Jul;273(1 Pt 1):L234-45.

PMID:
9252561
9.
10.

Selective expansion of fibroblast subpopulations from pulmonary artery adventitia in response to hypoxia.

Das M, Dempsey EC, Reeves JT, Stenmark KR.

Am J Physiol Lung Cell Mol Physiol. 2002 May;282(5):L976-86.

12.

Induction of Connective Tissue Growth Factor Expression by Hypoxia in Human Lung Fibroblasts via the MEKK1/MEK1/ERK1/GLI-1/GLI-2 and AP-1 Pathways.

Cheng Y, Lin CH, Chen JY, Li CH, Liu YT, Chen BC.

PLoS One. 2016 Aug 3;11(8):e0160593. doi: 10.1371/journal.pone.0160593. eCollection 2016. Erratum in: PLoS One. 2017 Nov 21;12 (11):e0188608.

13.

Vascular endothelial growth factor-induced osteopontin expression mediates vascular inflammation and neointima formation via Flt-1 in adventitial fibroblasts.

Li XD, Chen J, Ruan CC, Zhu DL, Gao PJ.

Arterioscler Thromb Vasc Biol. 2012 Sep;32(9):2250-8. doi: 10.1161/ATVBAHA.112.255216. Epub 2012 Jul 19.

14.

Hypoxic stimulation of the stress-activated protein kinases in pulmonary artery fibroblasts.

Scott PH, Paul A, Belham CM, Peacock AJ, Wadsworth RM, Gould GW, Welsh D, Plevin R.

Am J Respir Crit Care Med. 1998 Sep;158(3):958-62.

PMID:
9731031
15.

Oncogenic tyrosine kinase NPM/ALK induces activation of the MEK/ERK signaling pathway independently of c-Raf.

Marzec M, Kasprzycka M, Liu X, Raghunath PN, Wlodarski P, Wasik MA.

Oncogene. 2007 Feb 8;26(6):813-21. Epub 2006 Aug 7.

PMID:
16909118
16.
17.

Hypoxia induces differentiation of pulmonary artery adventitial fibroblasts into myofibroblasts.

Short M, Nemenoff RA, Zawada WM, Stenmark KR, Das M.

Am J Physiol Cell Physiol. 2004 Feb;286(2):C416-25. Epub 2003 Oct 15.

18.

ERK1/2 regulates ANG II-dependent cell proliferation via cytoplasmic activation of RSK2 and nuclear activation of elk1.

Godeny MD, Sayeski PP.

Am J Physiol Cell Physiol. 2006 Dec;291(6):C1308-17. Epub 2006 May 24.

19.

Peptides modified by myristoylation activate eNOS in endothelial cells through Akt phosphorylation.

Krotova K, Hu H, Xia SL, Belayev L, Patel JM, Block ER, Zharikov S.

Br J Pharmacol. 2006 Jul;148(5):732-40. Epub 2006 May 22.

20.

Supplemental Content

Support Center