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Items: 1 to 20 of 159

1.

Homologous recombination in DNA repair and DNA damage tolerance.

Li X, Heyer WD.

Cell Res. 2008 Jan;18(1):99-113. doi: 10.1038/cr.2008.1. Review.

2.

High levels of RAD51 perturb DNA replication elongation and cause unscheduled origin firing due to impaired CHK1 activation.

Parplys AC, Seelbach JI, Becker S, Behr M, Wrona A, Jend C, Mansour WY, Joosse SA, Stuerzbecher HW, Pospiech H, Petersen C, Dikomey E, Borgmann K.

Cell Cycle. 2015;14(19):3190-202. doi: 10.1080/15384101.2015.1055996.

3.

Mechanism of RAD51-dependent DNA interstrand cross-link repair.

Long DT, Räschle M, Joukov V, Walter JC.

Science. 2011 Jul 1;333(6038):84-7. doi: 10.1126/science.1204258.

4.

Rad51 replication fork recruitment is required for DNA damage tolerance.

González-Prieto R, Muñoz-Cabello AM, Cabello-Lobato MJ, Prado F.

EMBO J. 2013 May 2;32(9):1307-21. doi: 10.1038/emboj.2013.73. Epub 2013 Apr 5.

5.

Human HEL308 localizes to damaged replication forks and unwinds lagging strand structures.

Tafel AA, Wu L, McHugh PJ.

J Biol Chem. 2011 May 6;286(18):15832-40. doi: 10.1074/jbc.M111.228189. Epub 2011 Mar 11.

7.
8.

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity.

Michl J, Zimmer J, Tarsounas M.

EMBO J. 2016 May 2;35(9):909-23. doi: 10.15252/embj.201693860. Epub 2016 Apr 1. Review.

9.

Therapeutic exploitation of tumor cell defects in homologous recombination.

Powell SN, Kachnic LA.

Anticancer Agents Med Chem. 2008 May;8(4):448-60. Review.

PMID:
18473729
10.

Spontaneous homologous recombination is induced by collapsed replication forks that are caused by endogenous DNA single-strand breaks.

Saleh-Gohari N, Bryant HE, Schultz N, Parker KM, Cassel TN, Helleday T.

Mol Cell Biol. 2005 Aug;25(16):7158-69.

11.

C. elegans FANCD2 responds to replication stress and functions in interstrand cross-link repair.

Collis SJ, Barber LJ, Ward JD, Martin JS, Boulton SJ.

DNA Repair (Amst). 2006 Nov 8;5(11):1398-406. Epub 2006 Aug 17.

PMID:
16914393
12.

Homologous recombination in extrachromosomal plasmid substrates is not suppressed by p53.

Willers H, McCarthy EE, Hubbe P, Dahm-Daphi J, Powell SN.

Carcinogenesis. 2001 Nov;22(11):1757-63.

PMID:
11698336
13.

DNA repair in mammalian cells: DNA double-strand break repair: how to fix a broken relationship.

Pardo B, Gómez-González B, Aguilera A.

Cell Mol Life Sci. 2009 Mar;66(6):1039-56. doi: 10.1007/s00018-009-8740-3. Review.

PMID:
19153654
14.

Brca2, Rad51 and Mre11: performing balancing acts on replication forks.

Costanzo V.

DNA Repair (Amst). 2011 Oct 10;10(10):1060-5. doi: 10.1016/j.dnarep.2011.07.009. Epub 2011 Sep 6. Review.

PMID:
21900052
15.
16.

Replication blocking lesions present a unique substrate for homologous recombination.

Ward JD, Barber LJ, Petalcorin MI, Yanowitz J, Boulton SJ.

EMBO J. 2007 Jul 25;26(14):3384-96. Epub 2007 Jul 5.

17.

Deletion of BRCA2 exon 27 causes defects in response to both stalled and collapsed replication forks.

Kim TM, Son MY, Dodds S, Hu L, Hasty P.

Mutat Res. 2014 Aug-Sep;766-767:66-72. doi: 10.1016/j.mrfmmm.2014.06.003. Epub 2014 Jun 22.

PMID:
25847274
18.

Deletion of BRCA2 exon 27 causes defects in response to both stalled and collapsed replication forks.

Kim TM, Son MY, Dodds S, Hu L, Hasty P.

Mutat Res. 2014 Aug-Sep;766-767:66-72. doi: 10.1016/j.mrfmmm.2014.06.003. Epub 2014 Jun 22.

19.

Two replication fork maintenance pathways fuse inverted repeats to rearrange chromosomes.

Hu L, Kim TM, Son MY, Kim SA, Holland CL, Tateishi S, Kim DH, Yew PR, Montagna C, Dumitrache LC, Hasty P.

Nature. 2013 Sep 26;501(7468):569-72. doi: 10.1038/nature12500. Epub 2013 Sep 8.

20.

FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange.

Sommers JA, Rawtani N, Gupta R, Bugreev DV, Mazin AV, Cantor SB, Brosh RM Jr.

J Biol Chem. 2009 Mar 20;284(12):7505-17. doi: 10.1074/jbc.M809019200. Epub 2009 Jan 16.

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