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ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus.

Yu X, Wang Z, Mertz JE.

PLoS Pathog. 2007 Dec;3(12):e194.


Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.

Ellis AL, Wang Z, Yu X, Mertz JE.

J Virol. 2010 Jun;84(12):6139-52. doi: 10.1128/JVI.02706-09. Epub 2010 Apr 7.


Shutoff of BZLF1 gene expression is necessary for immortalization of primary B cells by Epstein-Barr virus.

Yu X, McCarthy PJ, Wang Z, Gorlen DA, Mertz JE.

J Virol. 2012 Aug;86(15):8086-96. doi: 10.1128/JVI.00234-12. Epub 2012 May 23.


ZEB1 and c-Jun levels contribute to the establishment of highly lytic Epstein-Barr virus infection in gastric AGS cells.

Feng WH, Kraus RJ, Dickerson SJ, Lim HJ, Jones RJ, Yu X, Mertz JE, Kenney SC.

J Virol. 2007 Sep;81(18):10113-22. Epub 2007 Jul 11.


Zinc finger E-box binding factor 1 plays a central role in regulating Epstein-Barr virus (EBV) latent-lytic switch and acts as a therapeutic target in EBV-associated gastric cancer.

Zhao J, Jin H, Cheung KF, Tong JH, Zhang S, Go MY, Tian L, Kang W, Leung PP, Zeng Z, Li X, To KF, Sung JJ, Yu J.

Cancer. 2012 Feb 15;118(4):924-36. doi: 10.1002/cncr.26184. Epub 2011 Jun 29.


Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Mirocha SJ, Stephany HM, Puchalski JR, Mertz JE.

J Virol. 2001 Jan;75(2):867-77.


Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.

Ellis-Connell AL, Iempridee T, Xu I, Mertz JE.

J Virol. 2010 Oct;84(19):10329-43. doi: 10.1128/JVI.00923-10. Epub 2010 Jul 28.


The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latency.

Yu X, McCarthy PJ, Lim HJ, Iempridee T, Kraus RJ, Gorlen DA, Mertz JE.

J Virol. 2011 May;85(10):5081-90. doi: 10.1128/JVI.02615-10. Epub 2011 Mar 9.


ZEB negatively regulates the lytic-switch BZLF1 gene promoter of Epstein-Barr virus.

Kraus RJ, Perrigoue JG, Mertz JE.

J Virol. 2003 Jan;77(1):199-207.


Protein kinase C-independent activation of the Epstein-Barr virus lytic cycle.

Gradoville L, Kwa D, El-Guindy A, Miller G.

J Virol. 2002 Jun;76(11):5612-26.


Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.

Kraus RJ, Yu X, Cordes BA, Sathiamoorthi S, Iempridee T, Nawandar DM, Ma S, Romero-Masters JC, McChesney KG, Lin Z, Makielski KR, Lee DL, Lambert PF, Johannsen EC, Kenney SC, Mertz JE.

PLoS Pathog. 2017 Jun 15;13(6):e1006404. doi: 10.1371/journal.ppat.1006404. eCollection 2017 Jun.


Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells.

Nawandar DM, Ohashi M, Djavadian R, Barlow E, Makielski K, Ali A, Lee D, Lambert PF, Johannsen E, Kenney SC.

J Virol. 2017 Mar 29;91(8). pii: e02438-16. doi: 10.1128/JVI.02438-16. Print 2017 Apr 15.


Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.

Reusch JA, Nawandar DM, Wright KL, Kenney SC, Mertz JE.

J Virol. 2015 Feb;89(3):1731-43. doi: 10.1128/JVI.02781-14. Epub 2014 Nov 19.


Switching of EBV cycles between latent and lytic states.

Murata T, Tsurumi T.

Rev Med Virol. 2014 May;24(3):142-53. doi: 10.1002/rmv.1780. Epub 2013 Dec 11. Review.


Interferon-γ-inducible protein 16 (IFI16) is required for the maintenance of Epstein-Barr virus latency.

Pisano G, Roy A, Ahmed Ansari M, Kumar B, Chikoti L, Chandran B.

Virol J. 2017 Nov 13;14(1):221. doi: 10.1186/s12985-017-0891-5.


The Epstein-Barr virus (EBV) nuclear antigen 1 BamHI F promoter is activated on entry of EBV-transformed B cells into the lytic cycle.

Lear AL, Rowe M, Kurilla MG, Lee S, Henderson S, Kieff E, Rickinson AB.

J Virol. 1992 Dec;66(12):7461-8.


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