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Items: 1 to 20 of 612

2.

Cell activation by Porphyromonas gingivalis lipid A molecule through Toll-like receptor 4- and myeloid differentiation factor 88-dependent signaling pathway.

Ogawa T, Asai Y, Hashimoto M, Takeuchi O, Kurita T, Yoshikai Y, Miyake K, Akira S.

Int Immunol. 2002 Nov;14(11):1325-32.

PMID:
12407023
3.

Contribution of interferon-beta to the murine macrophage response to the toll-like receptor 4 agonist, lipopolysaccharide.

Thomas KE, Galligan CL, Newman RD, Fish EN, Vogel SN.

J Biol Chem. 2006 Oct 13;281(41):31119-30. Epub 2006 Aug 15.

4.

TLR2-dependent inflammatory response to Porphyromonas gingivalis is MyD88 independent, whereas MyD88 is required to clear infection.

Burns E, Eliyahu T, Uematsu S, Akira S, Nussbaum G.

J Immunol. 2010 Feb 1;184(3):1455-62. doi: 10.4049/jimmunol.0900378. Epub 2009 Dec 30.

5.

Astrocyte TLR4 activation induces a proinflammatory environment through the interplay between MyD88-dependent NFκB signaling, MAPK, and Jak1/Stat1 pathways.

Gorina R, Font-Nieves M, Márquez-Kisinousky L, Santalucia T, Planas AM.

Glia. 2011 Feb;59(2):242-55. doi: 10.1002/glia.21094.

PMID:
21125645
6.

The induction of macrophage gene expression by LPS predominantly utilizes Myd88-independent signaling cascades.

Björkbacka H, Fitzgerald KA, Huet F, Li X, Gregory JA, Lee MA, Ordija CM, Dowley NE, Golenbock DT, Freeman MW.

Physiol Genomics. 2004 Nov 17;19(3):319-30. Epub 2004 Sep 14.

7.
8.

Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.

Zhou Q, Desta T, Fenton M, Graves DT, Amar S.

Infect Immun. 2005 Feb;73(2):935-43. Erratum in: Infect Immun. 2006 Mar;74(3):1995.

10.

Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus.

Hajishengallis G, Tapping RI, Harokopakis E, Nishiyama S, Ratti P, Schifferle RE, Lyle EA, Triantafilou M, Triantafilou K, Yoshimura F.

Cell Microbiol. 2006 Oct;8(10):1557-70.

PMID:
16984411
11.

Differential effects of CpG DNA on IFN-beta induction and STAT1 activation in murine macrophages versus dendritic cells: alternatively activated STAT1 negatively regulates TLR signaling in macrophages.

Schroder K, Spille M, Pilz A, Lattin J, Bode KA, Irvine KM, Burrows AD, Ravasi T, Weighardt H, Stacey KJ, Decker T, Hume DA, Dalpke AH, Sweet MJ.

J Immunol. 2007 Sep 15;179(6):3495-503.

13.

Signaling pathways in osteoblast proinflammatory responses to infection by Porphyromonas gingivalis.

Ohno T, Okahashi N, Morisaki I, Amano A.

Oral Microbiol Immunol. 2008 Apr;23(2):96-104. doi: 10.1111/j.1399-302X.2007.00393.x.

PMID:
18279176
14.

Lipopolysaccharide-binding protein critically regulates lipopolysaccharide-induced IFN-beta signaling pathway in human monocytes.

Kato A, Ogasawara T, Homma T, Saito H, Matsumoto K.

J Immunol. 2004 May 15;172(10):6185-94.

15.

Porphyromonas gingivalis selectively up-regulates the HIV-1 coreceptor CCR5 in oral keratinocytes.

Giacaman RA, Nobbs AH, Ross KF, Herzberg MC.

J Immunol. 2007 Aug 15;179(4):2542-50.

16.

Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.

Wang M, Shakhatreh MA, James D, Liang S, Nishiyama S, Yoshimura F, Demuth DR, Hajishengallis G.

J Immunol. 2007 Aug 15;179(4):2349-58.

17.

Peptide mapping of bacterial fimbrial epitopes interacting with pattern recognition receptors.

Hajishengallis G, Ratti P, Harokopakis E.

J Biol Chem. 2005 Nov 25;280(47):38902-13. Epub 2005 Aug 29.

20.

Transcriptional profiling of immune genes in bovine monocyte-derived macrophages exposed to bacterial antigens.

Taraktsoglou M, Szalabska U, Magee DA, Browne JA, Sweeney T, Gormley E, MacHugh DE.

Vet Immunol Immunopathol. 2011 Mar 15;140(1-2):130-9. doi: 10.1016/j.vetimm.2010.12.002. Epub 2010 Dec 10.

PMID:
21242003

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