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Items: 1 to 20 of 98

1.

Design of a novel quantitative PCR (QPCR)-based protocol for genotyping mice carrying the neuroprotective Wallerian degeneration slow (Wlds) gene.

Wishart TM, Macdonald SH, Chen PE, Shipston MJ, Coleman MP, Gillingwater TH, Ribchester RR.

Mol Neurodegener. 2007 Oct 30;2:21.

2.

The WldS protein protects against axonal degeneration: a model of gene therapy for peripheral neuropathy.

Wang MS, Fang G, Culver DG, Davis AA, Rich MM, Glass JD.

Ann Neurol. 2001 Dec;50(6):773-9.

PMID:
11761475
3.

Modified cell cycle status in a mouse model of altered neuronal vulnerability (slow Wallerian degeneration; Wlds).

Wishart TM, Pemberton HN, James SR, McCabe CJ, Gillingwater TH.

Genome Biol. 2008;9(6):R101. doi: 10.1186/gb-2008-9-6-r101. Epub 2008 Jun 20.

4.

An 85-kb tandem triplication in the slow Wallerian degeneration (Wlds) mouse.

Coleman MP, Conforti L, Buckmaster EA, Tarlton A, Ewing RM, Brown MC, Lyon MF, Perry VH.

Proc Natl Acad Sci U S A. 1998 Aug 18;95(17):9985-90.

5.

WldS mice are resistant to paclitaxel (taxol) neuropathy.

Wang MS, Davis AA, Culver DG, Glass JD.

Ann Neurol. 2002 Oct;52(4):442-7.

PMID:
12325073
6.

Local axonal protection by WldS as revealed by conditional regulation of protein stability.

Wang JT, Medress ZA, Vargas ME, Barres BA.

Proc Natl Acad Sci U S A. 2015 Aug 18;112(33):10093-100. doi: 10.1073/pnas.1508337112. Epub 2015 Jul 24.

7.

The progressive nature of Wallerian degeneration in wild-type and slow Wallerian degeneration (WldS) nerves.

Beirowski B, Adalbert R, Wagner D, Grumme DS, Addicks K, Ribchester RR, Coleman MP.

BMC Neurosci. 2005 Feb 1;6:6.

8.

Differential proteomics analysis of synaptic proteins identifies potential cellular targets and protein mediators of synaptic neuroprotection conferred by the slow Wallerian degeneration (Wlds) gene.

Wishart TM, Paterson JM, Short DM, Meredith S, Robertson KA, Sutherland C, Cousin MA, Dutia MB, Gillingwater TH.

Mol Cell Proteomics. 2007 Aug;6(8):1318-30. Epub 2007 Apr 29.

9.

Synaptic protection in the brain of WldS mice occurs independently of age but is sensitive to gene-dose.

Wright AK, Wishart TM, Ingham CA, Gillingwater TH.

PLoS One. 2010 Nov 29;5(11):e15108. doi: 10.1371/journal.pone.0015108.

11.

The Wlds transgene reduces axon loss in a Charcot-Marie-Tooth disease 1A rat model and nicotinamide delays post-traumatic axonal degeneration.

Meyer zu Horste G, Miesbach TA, Muller JI, Fledrich R, Stassart RM, Kieseier BC, Coleman MP, Sereda MW.

Neurobiol Dis. 2011 Apr;42(1):1-8. doi: 10.1016/j.nbd.2010.12.006. Epub 2010 Dec 16.

PMID:
21168501
12.

Expression of the neuroprotective slow Wallerian degeneration (WldS) gene in non-neuronal tissues.

Wishart TM, Brownstein DG, Thomson D, Tabakova AM, Boothe KM, Tsao JW, Gillingwater TH.

BMC Neurosci. 2009 Dec 16;10:148. doi: 10.1186/1471-2202-10-148.

13.

Wlds-mediated protection of dopaminergic fibers in an animal model of Parkinson disease.

Sajadi A, Schneider BL, Aebischer P.

Curr Biol. 2004 Feb 17;14(4):326-30.

14.

WldS but not Nmnat1 protects dopaminergic neurites from MPP+ neurotoxicity.

Antenor-Dorsey JA, O'Malley KL.

Mol Neurodegener. 2012 Feb 8;7:5. doi: 10.1186/1750-1326-7-5.

15.

Demyelination can proceed independently of axonal degradation during Wallerian degeneration in wlds mice.

Takada H, Yuasa S, Araki T.

Eur J Neurosci. 2011 Aug;34(4):531-7. doi: 10.1111/j.1460-9568.2011.07783.x. Epub 2011 Jul 12.

PMID:
21749497
16.

Stable inheritance of an 85-kb triplication in C57BL/WldS mice.

Mi W, Glass JD, Coleman MP.

Mutat Res. 2003 May 15;526(1-2):33-7.

PMID:
12714180
17.

Elimination of motor nerve terminals in neonatal mice expressing a gene for slow wallerian degeneration (C57Bl/Wlds).

Parson SH, Mackintosh CL, Ribchester RR.

Eur J Neurosci. 1997 Aug;9(8):1586-92.

PMID:
9283813
18.

Protection of vincristine-induced neuropathy by WldS expression and the independence of the activity of Nmnat1.

Watanabe M, Tsukiyama T, Hatakeyama S.

Neurosci Lett. 2007 Jan 16;411(3):228-32. Epub 2006 Nov 17.

PMID:
17113230

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