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Items: 1 to 20 of 495

2.

Ethanol exposure in early adolescence inhibits intrinsic neuronal plasticity via sigma-1 receptor activation in hippocampal CA1 neurons.

Sabeti J.

Alcohol Clin Exp Res. 2011 May;35(5):885-904. doi: 10.1111/j.1530-0277.2010.01419.x. Epub 2011 Feb 11.

5.

The transient depression of hippocampal CA1 LTP induced by chronic intermittent ethanol exposure is associated with an inhibition of the MAP kinase pathway.

Roberto M, Nelson TE, Ur CL, Brunelli M, Sanna PP, Gruol DL.

Eur J Neurosci. 2003 Apr;17(8):1646-54.

PMID:
12752382
7.
8.

Synaptic strength at the temporoammonic input to the hippocampal CA1 region in vivo is regulated by NMDA receptors, metabotropic glutamate receptors and voltage-gated calcium channels.

Aksoy-Aksel A, Manahan-Vaughan D.

Neuroscience. 2015 Nov 19;309:191-9. doi: 10.1016/j.neuroscience.2015.03.014. Epub 2015 Mar 17.

9.

A NMDA receptor glycine site partial agonist, GLYX-13, simultaneously enhances LTP and reduces LTD at Schaffer collateral-CA1 synapses in hippocampus.

Zhang XL, Sullivan JA, Moskal JR, Stanton PK.

Neuropharmacology. 2008 Dec;55(7):1238-50. doi: 10.1016/j.neuropharm.2008.08.018. Epub 2008 Aug 29.

10.

Plasticity of synaptic GluN receptors is required for the Src-dependent induction of long-term potentiation at CA3-CA1 synapses.

Li HB, Jackson MF, Yang K, Trepanier C, Salter MW, Orser BA, Macdonald JF.

Hippocampus. 2011 Oct;21(10):1053-61. doi: 10.1002/hipo.20818. Epub 2010 Jun 2.

PMID:
20865743
11.

Hippocampal long-term synaptic plasticity and signal amplification of NMDA receptors.

MacDonald JF, Jackson MF, Beazely MA.

Crit Rev Neurobiol. 2006;18(1-2):71-84. Review.

PMID:
17725510
12.

Repeated third trimester-equivalent ethanol exposure inhibits long-term potentiation in the hippocampal CA1 region of neonatal rats.

Puglia MP, Valenzuela CF.

Alcohol. 2010 May;44(3):283-90. doi: 10.1016/j.alcohol.2010.03.001. Epub 2010 May 20.

13.

Acute and chronic ethanol exposure differentially affect induction of hippocampal LTP.

Fujii S, Yamazaki Y, Sugihara T, Wakabayashi I.

Brain Res. 2008 May 23;1211:13-21. doi: 10.1016/j.brainres.2008.02.052. Epub 2008 Feb 29.

PMID:
18423576
14.

Reparatory effects of nicotine on NMDA receptor-mediated synaptic plasticity in the hippocampal CA1 region of chronically lead-exposed rats.

Wang HL, Chen XT, Luo L, Lou ZY, Wang S, Chen JT, Wang M, Sun LG, Ruan DY.

Eur J Neurosci. 2006 Mar;23(5):1111-9.

PMID:
16553775
15.

Selective alterations of NMDAR function and plasticity in D1 and D2 medium spiny neurons in the nucleus accumbens shell following chronic intermittent ethanol exposure.

Renteria R, Maier EY, Buske TR, Morrisett RA.

Neuropharmacology. 2017 Jan;112(Pt A):164-171. doi: 10.1016/j.neuropharm.2016.03.004. Epub 2016 Mar 2.

16.

Coexistence of muscarinic long-term depression with electrically induced long-term potentiation and depression at CA3-CA1 synapses.

McCutchen E, Scheiderer CL, Dobrunz LE, McMahon LL.

J Neurophysiol. 2006 Dec;96(6):3114-21. Epub 2006 Sep 27.

PMID:
17005622
17.

Bidirectional synaptic plasticity induced by conditioned stimulations with different number of pulse at hippocampal CA1 synapses: roles of N-methyl-D-aspartate and metabotropic glutamate receptors.

Hsu JC, Cheng SJ, Yang HW, Wang HJ, Chiu TH, Min MY, Lin YW.

Synapse. 2011 Aug;65(8):795-803. doi: 10.1002/syn.20906. Epub 2011 Mar 10.

PMID:
21218453
18.

Evidence for a role for GABA(A) and NMDA receptors in ethanol inhibition of long-term potentiation.

Schummers J, Browning MD.

Brain Res Mol Brain Res. 2001 Oct 19;94(1-2):9-14.

PMID:
11597760
19.

Long-term potentiation in the rat hippocampus is reversibly depressed by chronic intermittent ethanol exposure.

Roberto M, Nelson TE, Ur CL, Gruol DL.

J Neurophysiol. 2002 May;87(5):2385-97.

PMID:
11976376
20.

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