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Items: 1 to 20 of 118

1.

The influence of the NOD Nss1/Idd5 loci on sialadenitis and gene expression in salivary glands of congenic mice.

Hjelmervik TO, Lindqvist AK, Petersen K, Johannesson M, Stavrum AK, Johansson A, Jonsson R, Holmdahl R, Bolstad AI.

Arthritis Res Ther. 2007;9(5):R99.

2.

The genetic control of sialadenitis versus arthritis in a NOD.QxB10.Q F2 cross.

Johansson AC, Nakken B, Sundler M, Lindqvist AK, Johannesson M, Alarcón-Riquelme M, Bolstad AI, Humphreys-Beher MG, Jonsson R, Skarstein K, Holmdahl R.

Eur J Immunol. 2002 Jan;32(1):243-50.

3.

Genetic analysis of autoimmune sialadenitis in nonobese diabetic mice: a major susceptibility region on chromosome 1.

Boulard O, Fluteau G, Eloy L, Damotte D, Bedossa P, Garchon HJ.

J Immunol. 2002 Apr 15;168(8):4192-201.

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An NZW-derived interval on chromosome 7 moderates sialadenitis, but not insulitis in congenic nonobese diabetic mice.

Burt RA, Watkins L, Tan IK, Wang N, Quirk F, Mackin L, Morgan P, Zhang JG, Berzins SP, Morahan G, Brodnicki TC.

J Immunol. 2010 Jan 15;184(2):859-68. doi: 10.4049/jimmunol.0903149. Epub 2009 Dec 9.

7.

Genetic interactions among Idd3, Idd5.1, Idd5.2, and Idd5.3 protective loci in the nonobese diabetic mouse model of type 1 diabetes.

Lin X, Hamilton-Williams EE, Rainbow DB, Hunter KM, Dai YD, Cheung J, Peterson LB, Wicker LS, Sherman LA.

J Immunol. 2013 Apr 1;190(7):3109-20. doi: 10.4049/jimmunol.1203422. Epub 2013 Feb 20.

8.

Increased autoimmune diabetes in pIgR-deficient NOD mice is due to a "Hitchhiking" interval that refines the genetic effect of Idd5.4.

Simpfendorfer KR, Strugnell RA, Brodnicki TC, Wijburg OL.

PLoS One. 2015 Apr 2;10(4):e0121979. doi: 10.1371/journal.pone.0121979. eCollection 2015.

9.

Fine mapping, gene content, comparative sequencing, and expression analyses support Ctla4 and Nramp1 as candidates for Idd5.1 and Idd5.2 in the nonobese diabetic mouse.

Wicker LS, Chamberlain G, Hunter K, Rainbow D, Howlett S, Tiffen P, Clark J, Gonzalez-Munoz A, Cumiskey AM, Rosa RL, Howson JM, Smink LJ, Kingsnorth A, Lyons PA, Gregory S, Rogers J, Todd JA, Peterson LB.

J Immunol. 2004 Jul 1;173(1):164-73.

10.

Alleles from chromosomes 1 and 3 of NOD mice combine to influence Sjögren's syndrome-like autoimmune exocrinopathy.

Brayer J, Lowry J, Cha S, Robinson CP, Yamachika S, Peck AB, Humphreys-Beher MG.

J Rheumatol. 2000 Aug;27(8):1896-904.

PMID:
10955330
11.

The diabetes susceptibility locus Idd5.1 on mouse chromosome 1 regulates ICOS expression and modulates murine experimental autoimmune encephalomyelitis.

Greve B, Vijayakrishnan L, Kubal A, Sobel RA, Peterson LB, Wicker LS, Kuchroo VK.

J Immunol. 2004 Jul 1;173(1):157-63.

12.

Influence on spontaneous tissue inflammation by the major histocompatibility complex region in the nonobese diabetic mouse.

Lindqvist AK, Nakken B, Sundler M, Kjellén P, Jonsson R, Holmdahl R, Skarstein K.

Scand J Immunol. 2005 Feb;61(2):119-27.

13.

Candidate chromosome 1 disease susceptibility genes for Sjogren's syndrome xerostomia are narrowed by novel NOD.B10 congenic mice.

Mongini PK, Kramer JM, Ishikawa TO, Herschman H, Esposito D.

Clin Immunol. 2014 Jul;153(1):79-90. doi: 10.1016/j.clim.2014.03.012. Epub 2014 Mar 29.

14.

Further mapping of the Idd5.1 locus for autoimmune diabetes in NOD mice.

Lamhamedi-Cherradi SE, Boulard O, Gonzalez C, Kassis N, Damotte D, Eloy L, Fluteau G, Lévi-Strauss M, Garchon HJ.

Diabetes. 2001 Dec;50(12):2874-8.

15.

NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans.

Hill NJ, Lyons PA, Armitage N, Todd JA, Wicker LS, Peterson LB.

Diabetes. 2000 Oct;49(10):1744-7.

PMID:
11016460
16.

Genes within the Idd5 and Idd9/11 diabetes susceptibility loci affect the pathogenic activity of B cells in nonobese diabetic mice.

Silveira PA, Chapman HD, Stolp J, Johnson E, Cox SL, Hunter K, Wicker LS, Serreze DV.

J Immunol. 2006 Nov 15;177(10):7033-41.

17.

Combining mouse congenic strains and microarray gene expression analyses to study a complex trait: the NOD model of type 1 diabetes.

Eaves IA, Wicker LS, Ghandour G, Lyons PA, Peterson LB, Todd JA, Glynne RJ.

Genome Res. 2002 Feb;12(2):232-43.

18.

Genetic evidence that the differential expression of the ligand-independent isoform of CTLA-4 is the molecular basis of the Idd5.1 type 1 diabetes region in nonobese diabetic mice.

Araki M, Chung D, Liu S, Rainbow DB, Chamberlain G, Garner V, Hunter KM, Vijayakrishnan L, Peterson LB, Oukka M, Sharpe AH, Sobel R, Kuchroo VK, Wicker LS.

J Immunol. 2009 Oct 15;183(8):5146-57. doi: 10.4049/jimmunol.0802610. Epub 2009 Sep 25.

19.

Differential gene expression in the salivary gland during development and onset of xerostomia in Sjögren's syndrome-like disease of the C57BL/6.NOD-Aec1Aec2 mouse.

Nguyen CQ, Sharma A, Lee BH, She JX, McIndoe RA, Peck AB.

Arthritis Res Ther. 2009;11(2):R56. doi: 10.1186/ar2676. Epub 2009 Apr 20.

20.

P2Y2 nucleotide receptor up-regulation in submandibular gland cells from the NOD.B10 mouse model of Sjögren's syndrome.

Schrader AM, Camden JM, Weisman GA.

Arch Oral Biol. 2005 Jun;50(6):533-40. Epub 2004 Dec 15.

PMID:
15848146

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