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Items: 1 to 20 of 105

1.

Porphyromonas gingivalis interactions with complement receptor 3 (CR3): innate immunity or immune evasion?

Hajishengallis G, Harokopakis E.

Front Biosci. 2007 May 1;12:4547-57. Review.

PMID:
17485396
2.

Subversion of innate immunity by periodontopathic bacteria via exploitation of complement receptor-3.

Hajishengallis G, Wang M, Liang S, Shakhatreh MA, James D, Nishiyama S, Yoshimura F, Demuth DR.

Adv Exp Med Biol. 2008;632:203-19. Review.

3.

Intracellular signaling and cytokine induction upon interactions of Porphyromonas gingivalis fimbriae with pattern-recognition receptors.

Hajishengallis G, Sojar H, Genco RJ, DeNardin E.

Immunol Invest. 2004 May;33(2):157-72.

PMID:
15195695
4.
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6.

Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.

Wang M, Shakhatreh MA, James D, Liang S, Nishiyama S, Yoshimura F, Demuth DR, Hajishengallis G.

J Immunol. 2007 Aug 15;179(4):2349-58.

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Porphyromonas gingivalis fimbriae proactively modulate beta2 integrin adhesive activity and promote binding to and internalization by macrophages.

Hajishengallis G, Wang M, Harokopakis E, Triantafilou M, Triantafilou K.

Infect Immun. 2006 Oct;74(10):5658-66.

9.

Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus.

Hajishengallis G, Tapping RI, Harokopakis E, Nishiyama S, Ratti P, Schifferle RE, Lyle EA, Triantafilou M, Triantafilou K, Yoshimura F.

Cell Microbiol. 2006 Oct;8(10):1557-70.

PMID:
16984411
10.

Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte.

Hajishengallis G, Krauss JL, Jotwani R, Lambris JD.

Mol Oral Microbiol. 2017 Apr;32(2):154-165. doi: 10.1111/omi.12161. Epub 2016 May 18.

11.

Peptide mapping of bacterial fimbrial epitopes interacting with pattern recognition receptors.

Hajishengallis G, Ratti P, Harokopakis E.

J Biol Chem. 2005 Nov 25;280(47):38902-13. Epub 2005 Aug 29.

12.

Mechanism and implications of CXCR4-mediated integrin activation by Porphyromonas gingivalis.

Hajishengallis G, McIntosh ML, Nishiyama SI, Yoshimura F.

Mol Oral Microbiol. 2013 Aug;28(4):239-49. doi: 10.1111/omi.12021. Epub 2013 Jan 21.

15.

Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae.

Hajishengallis G, Wang M, Liang S.

J Immunol. 2009 Jun 1;182(11):6690-6. doi: 10.4049/jimmunol.0900524.

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17.

Porphyromonas gingivalis and the autophagic pathway: an innate immune interaction?

Rodrigues PH, BĂ©langer M, Dunn W Jr, Progulske-Fox A.

Front Biosci. 2008 Jan 1;13:178-87. Review.

PMID:
17981536
18.

Porphyromonas gingivalis fimbriae induce unique dendritic cell subsets via Toll-like receptor 2.

Kanaya S, Nemoto E, Ogawa T, Shimauchi H.

J Periodontal Res. 2009 Aug;44(4):543-9. doi: 10.1111/j.1600-0765.2008.01149.x. Epub 2009 Oct 7.

PMID:
18973528
19.

Bacterial fimbriae stimulate proinflammatory activation in the endothelium through distinct TLRs.

Davey M, Liu X, Ukai T, Jain V, Gudino C, Gibson FC 3rd, Golenbock D, Visintin A, Genco CA.

J Immunol. 2008 Feb 15;180(4):2187-95.

20.

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