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Items: 1 to 20 of 625

1.

Mice with GFAP-targeted loss of neurofibromin demonstrate increased axonal MET expression with aging.

Su W, Xing R, Guha A, Gutmann DH, Sherman LS.

Glia. 2007 May;55(7):723-33.

PMID:
17348023
2.

Neurofibromin expression and astrogliosis in neurofibromatosis (type 1) brains.

Nordlund ML, Rizvi TA, Brannan CI, Ratner N.

J Neuropathol Exp Neurol. 1995 Jul;54(4):588-600.

PMID:
7602332
3.

Aberrant growth and differentiation of oligodendrocyte progenitors in neurofibromatosis type 1 mutants.

Bennett MR, Rizvi TA, Karyala S, McKinnon RD, Ratner N.

J Neurosci. 2003 Aug 6;23(18):7207-17.

5.

Glioma formation in neurofibromatosis 1 reflects preferential activation of K-RAS in astrocytes.

Dasgupta B, Li W, Perry A, Gutmann DH.

Cancer Res. 2005 Jan 1;65(1):236-45.

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7.

Glial scar and axonal regeneration in the CNS: lessons from GFAP and vimentin transgenic mice.

Ribotta MG, Menet V, Privat A.

Acta Neurochir Suppl. 2004;89:87-92.

PMID:
15335106
8.

Bone marrow-derived cells expressing green fluorescent protein under the control of the glial fibrillary acidic protein promoter do not differentiate into astrocytes in vitro and in vivo.

Wehner T, Böntert M, Eyüpoglu I, Prass K, Prinz M, Klett FF, Heinze M, Bechmann I, Nitsch R, Kirchhoff F, Kettenmann H, Dirnagl U, Priller J.

J Neurosci. 2003 Jun 15;23(12):5004-11.

9.

Neurofibromatosis 1 (NF1) heterozygosity results in a cell-autonomous growth advantage for astrocytes.

Bajenaru ML, Donahoe J, Corral T, Reilly KM, Brophy S, Pellicer A, Gutmann DH.

Glia. 2001 Mar 15;33(4):314-23.

PMID:
11246230
10.

Neurite outgrowth on non-permissive substrates in vitro is enhanced by ectopic expression of the neural adhesion molecule L1 by mouse astrocytes.

Mohajeri MH, Bartsch U, van der Putten H, Sansig G, Mucke L, Schachner M.

Eur J Neurosci. 1996 Jun;8(6):1085-97.

PMID:
8752578
11.
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13.

Heterozygosity for the neurofibromatosis 1 (NF1) tumor suppressor results in abnormalities in cell attachment, spreading and motility in astrocytes.

Gutmann DH, Wu YL, Hedrick NM, Zhu Y, Guha A, Parada LF.

Hum Mol Genet. 2001 Dec 15;10(26):3009-16.

PMID:
11751683
14.

Inactivation of the glial fibrillary acidic protein gene, but not that of vimentin, improves neuronal survival and neurite growth by modifying adhesion molecule expression.

Menet V, Giménez y Ribotta M, Chauvet N, Drian MJ, Lannoy J, Colucci-Guyon E, Privat A.

J Neurosci. 2001 Aug 15;21(16):6147-58.

15.

Oligodendrogliomas result from the expression of an activated mutant epidermal growth factor receptor in a RAS transgenic mouse astrocytoma model.

Ding H, Shannon P, Lau N, Wu X, Roncari L, Baldwin RL, Takebayashi H, Nagy A, Gutmann DH, Guha A.

Cancer Res. 2003 Mar 1;63(5):1106-13.

16.

Astrocyte-targeted expression of interleukin-3 and interferon-alpha causes region-specific changes in metallothionein expression in the brain.

Giralt M, Carrasco J, Penkowa M, Morcillo MA, Santamaría J, Campbell IL, Hidalgo J.

Exp Neurol. 2001 Apr;168(2):334-46.

PMID:
11259121
17.

Neuro-glia interaction effects on GFAP gene: a novel role for transforming growth factor-beta1.

de Sampaio e Spohr TC, Martinez R, da Silva EF, Neto VM, Gomes FC.

Eur J Neurosci. 2002 Dec;16(11):2059-69.

PMID:
12473073
18.

Loss of glial fibrillary acidic protein results in decreased glutamate transport and inhibition of PKA-induced EAAT2 cell surface trafficking.

Hughes EG, Maguire JL, McMinn MT, Scholz RE, Sutherland ML.

Brain Res Mol Brain Res. 2004 May 19;124(2):114-23.

PMID:
15135219
19.

NF1 regulates a Ras-dependent vascular smooth muscle proliferative injury response.

Xu J, Ismat FA, Wang T, Yang J, Epstein JA.

Circulation. 2007 Nov 6;116(19):2148-56. Epub 2007 Oct 22.

20.

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