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Items: 1 to 20 of 140

1.

Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants.

Schmidt KH, Kolodner RD.

Proc Natl Acad Sci U S A. 2006 Nov 28;103(48):18196-201. Epub 2006 Nov 17.

2.
3.

The Saccharomyces cerevisiae Rad6 postreplication repair and Siz1/Srs2 homologous recombination-inhibiting pathways process DNA damage that arises in asf1 mutants.

Kats ES, Enserink JM, Martinez S, Kolodner RD.

Mol Cell Biol. 2009 Oct;29(19):5226-37. doi: 10.1128/MCB.00894-09. Epub 2009 Jul 27.

4.

Srs2 DNA helicase is involved in checkpoint response and its regulation requires a functional Mec1-dependent pathway and Cdk1 activity.

Liberi G, Chiolo I, Pellicioli A, Lopes M, Plevani P, Muzi-Falconi M, Foiani M.

EMBO J. 2000 Sep 15;19(18):5027-38.

5.

A Rad53 independent function of Rad9 becomes crucial for genome maintenance in the absence of the Recq helicase Sgs1.

Nielsen I, Bentsen IB, Andersen AH, Gasser SM, Bjergbaek L.

PLoS One. 2013 Nov 20;8(11):e81015. doi: 10.1371/journal.pone.0081015. eCollection 2013.

7.

Differential involvement of the related DNA helicases Pif1p and Rrm3p in mtDNA point mutagenesis and stability.

O'Rourke TW, Doudican NA, Zhang H, Eaton JS, Doetsch PW, Shadel GS.

Gene. 2005 Jul 18;354:86-92.

PMID:
15907372
8.
9.

Rad53-Mediated Regulation of Rrm3 and Pif1 DNA Helicases Contributes to Prevention of Aberrant Fork Transitions under Replication Stress.

Rossi SE, Ajazi A, Carotenuto W, Foiani M, Giannattasio M.

Cell Rep. 2015 Oct 6;13(1):80-92. doi: 10.1016/j.celrep.2015.08.073. Epub 2015 Sep 24.

10.

Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death.

Schmidt KH, Viebranz EB, Harris LB, Mirzaei-Souderjani H, Syed S, Medicus R.

Eukaryot Cell. 2010 Feb;9(2):315-24. doi: 10.1128/EC.00260-09. Epub 2009 Dec 11.

11.

Replisome instability, fork collapse, and gross chromosomal rearrangements arise synergistically from Mec1 kinase and RecQ helicase mutations.

Cobb JA, Schleker T, Rojas V, Bjergbaek L, Tercero JA, Gasser SM.

Genes Dev. 2005 Dec 15;19(24):3055-69.

12.

Remodeling of the Rad51 DNA strand-exchange protein by the Srs2 helicase.

Sasanuma H, Furihata Y, Shinohara M, Shinohara A.

Genetics. 2013 Aug;194(4):859-72. doi: 10.1534/genetics.113.150615. Epub 2013 Jun 14.

14.

Tid1/Rdh54 translocase is phosphorylated through a Mec1- and Rad53-dependent manner in the presence of DSB lesions in budding yeast.

Ferrari M, Nachimuthu BT, Donnianni RA, Klein H, Pellicioli A.

DNA Repair (Amst). 2013 May 1;12(5):347-55. doi: 10.1016/j.dnarep.2013.02.004. Epub 2013 Mar 7.

15.

DNA damage checkpoint and recombinational repair differentially affect the replication stress tolerance of Smc6 mutants.

Chen YH, Szakal B, Castellucci F, Branzei D, Zhao X.

Mol Biol Cell. 2013 Aug;24(15):2431-41. doi: 10.1091/mbc.E12-11-0836. Epub 2013 Jun 19.

18.

Recovery from checkpoint-mediated arrest after repair of a double-strand break requires Srs2 helicase.

Vaze MB, Pellicioli A, Lee SE, Ira G, Liberi G, Arbel-Eden A, Foiani M, Haber JE.

Mol Cell. 2002 Aug;10(2):373-85.

19.

Rrm3 protects the Saccharomyces cerevisiae genome from instability at nascent sites of retrotransposition.

Stamenova R, Maxwell PH, Kenny AE, Curcio MJ.

Genetics. 2009 Jul;182(3):711-23. doi: 10.1534/genetics.109.104208. Epub 2009 May 4.

20.

The processing of double-strand breaks and binding of single-strand-binding proteins RPA and Rad51 modulate the formation of ATR-kinase foci in yeast.

Dubrana K, van Attikum H, Hediger F, Gasser SM.

J Cell Sci. 2007 Dec 1;120(Pt 23):4209-20. Epub 2007 Nov 14.

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