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Items: 1 to 20 of 241

1.

Apoptotic cells, through transforming growth factor-beta, coordinately induce anti-inflammatory and suppress pro-inflammatory eicosanoid and NO synthesis in murine macrophages.

Freire-de-Lima CG, Xiao YQ, Gardai SJ, Bratton DL, Schiemann WP, Henson PM.

J Biol Chem. 2006 Dec 15;281(50):38376-84. Epub 2006 Oct 20.

4.

Transforming growth factor beta 1 alters rat peritoneal macrophage mediator production and improves survival during endotoxic shock.

Pender BS, Chen H, Ashton S, Wise WC, Zingarelli B, Cusumano V, Cook JA.

Eur Cytokine Netw. 1996 Apr-Jun;7(2):137-42.

PMID:
8688491
5.

Particle digestibility is required for induction of the phosphatidylserine recognition mechanism used by murine macrophages to phagocytose apoptotic cells.

Fadok VA, Laszlo DJ, Noble PW, Weinstein L, Riches DW, Henson PM.

J Immunol. 1993 Oct 15;151(8):4274-85.

PMID:
8409401
6.

Control of nitric oxide production by endogenous TGF-beta1 and systemic nitric oxide in retinal pigment epithelial cells and peritoneal macrophages.

Vodovotz Y, Letterio JJ, Geiser AG, Chesler L, Roberts AB, Sparrow J.

J Leukoc Biol. 1996 Aug;60(2):261-70.

PMID:
8773588
7.

Intracisternal administration of transforming growth factor-beta evokes fever through the induction of cyclooxygenase-2 in brain endothelial cells.

Matsumura S, Shibakusa T, Fujikawa T, Yamada H, Matsumura K, Inoue K, Fushiki T.

Am J Physiol Regul Integr Comp Physiol. 2008 Jan;294(1):R266-75. Epub 2007 Oct 24.

8.

Dual role for TGF-beta1 in apoptosis.

Sánchez-Capelo A.

Cytokine Growth Factor Rev. 2005 Feb;16(1):15-34. Epub 2005 Jan 25. Review.

PMID:
15733830
9.
10.

Oxidants selectively reverse TGF-beta suppression of proinflammatory mediator production.

Xiao YQ, Freire-de-Lima CG, Janssen WJ, Morimoto K, Lyu D, Bratton DL, Henson PM.

J Immunol. 2006 Jan 15;176(2):1209-17.

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Caffeic acid phenethyl ester protects mice from lethal endotoxin shock and inhibits lipopolysaccharide-induced cyclooxygenase-2 and inducible nitric oxide synthase expression in RAW 264.7 macrophages via the p38/ERK and NF-kappaB pathways.

Jung WK, Choi I, Lee DY, Yea SS, Choi YH, Kim MM, Park SG, Seo SK, Lee SW, Lee CM, Park YM, Choi IW.

Int J Biochem Cell Biol. 2008;40(11):2572-82. doi: 10.1016/j.biocel.2008.05.005. Epub 2008 May 15.

PMID:
18571461
13.

M-1/M-2 macrophages and the Th1/Th2 paradigm.

Mills CD, Kincaid K, Alt JM, Heilman MJ, Hill AM.

J Immunol. 2000 Jun 15;164(12):6166-73.

15.

Lipoxins, aspirin-triggered epi-lipoxins, lipoxin stable analogues, and the resolution of inflammation: stimulation of macrophage phagocytosis of apoptotic neutrophils in vivo.

Mitchell S, Thomas G, Harvey K, Cottell D, Reville K, Berlasconi G, Petasis NA, Erwig L, Rees AJ, Savill J, Brady HR, Godson C.

J Am Soc Nephrol. 2002 Oct;13(10):2497-507.

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Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages.

Freire-de-Lima CG, Nascimento DO, Soares MB, Bozza PT, Castro-Faria-Neto HC, de Mello FG, DosReis GA, Lopes MF.

Nature. 2000 Jan 13;403(6766):199-203. Erratum in: Nature 2000 Apr 20;404(6780):904.

PMID:
10646605
20.

Transforming growth factor-beta primes macrophages to express inflammatory gene products in response to particulate stimuli by an autocrine/paracrine mechanism.

Noble PW, Henson PM, Lucas C, Mora-Worms M, Carré PC, Riches DW.

J Immunol. 1993 Jul 15;151(2):979-89.

PMID:
8335923

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