Similar articles for PMID: 16987815

Year	Number of Results
1996	1
1997	2
1999	2
2000	5
2001	3
2002	12
2003	8
2004	9
2005	10
2006	9
2007	10
2008	7
2009	15
2010	13
2011	13
2012	14
2013	11
2014	10
2015	8
2016	4
2019	2
2020	6
2021	11
2022	11
2023	3
2024	0

1

A membrane-targeted BID BCL-2 homology 3 peptide is sufficient for high potency activation of BAX in vitro.

Oh KJ, Barbuto S, Pitter K, Morash J, Walensky LD, Korsmeyer SJ. Oh KJ, et al. J Biol Chem. 2006 Dec 1;281(48):36999-7008. doi: 10.1074/jbc.M602341200. Epub 2006 Sep 20. J Biol Chem. 2006. PMID: 16987815 Free article.

2

A stapled BID BH3 helix directly binds and activates BAX.

Walensky LD, Pitter K, Morash J, Oh KJ, Barbuto S, Fisher J, Smith E, Verdine GL, Korsmeyer SJ. Walensky LD, et al. Mol Cell. 2006 Oct 20;24(2):199-210. doi: 10.1016/j.molcel.2006.08.020. Mol Cell. 2006. PMID: 17052454 Free article.

3

tBid elicits a conformational alteration in membrane-bound Bcl-2 such that it inhibits Bax pore formation.

Peng J, Tan C, Roberts GJ, Nikolaeva O, Zhang Z, Lapolla SM, Primorac S, Andrews DW, Lin J. Peng J, et al. J Biol Chem. 2006 Nov 24;281(47):35802-11. doi: 10.1074/jbc.M608303200. Epub 2006 Sep 27. J Biol Chem. 2006. PMID: 17005564 Free PMC article.

4

Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.

Werner AB, de Vries E, Tait SW, Bontjer I, Borst J. Werner AB, et al. J Biol Chem. 2002 Jun 21;277(25):22781-8. doi: 10.1074/jbc.M201469200. Epub 2002 Apr 19. J Biol Chem. 2002. PMID: 11929871 Free article.

5

BH3-only protein bid participates in the Bcl-2 network in healthy liver cells.

Hikita H, Takehara T, Kodama T, Shimizu S, Hosui A, Miyagi T, Tatsumi T, Ishida H, Ohkawa K, Li W, Kanto T, Hiramatsu N, Hennighausen L, Yin XM, Hayashi N. Hikita H, et al. Hepatology. 2009 Dec;50(6):1972-80. doi: 10.1002/hep.23207. Hepatology. 2009. PMID: 19839062 Free PMC article.

6

Distinct lipid effects on tBid and Bim activation of membrane permeabilization by pro-apoptotic Bax.

Shamas-Din A, Bindner S, Chi X, Leber B, Andrews DW, Fradin C. Shamas-Din A, et al. Biochem J. 2015 May 1;467(3):495-505. doi: 10.1042/BJ20141291. Biochem J. 2015. PMID: 25714678

7

BIM and tBID are not mechanistically equivalent when assisting BAX to permeabilize bilayer membranes.

Terrones O, Etxebarria A, Landajuela A, Landeta O, Antonsson B, Basañez G. Terrones O, et al. J Biol Chem. 2008 Mar 21;283(12):7790-803. doi: 10.1074/jbc.M708814200. Epub 2008 Jan 14. J Biol Chem. 2008. PMID: 18195012 Free article.

8

The Bcl-2-associated death promoter (BAD) lowers the threshold at which the Bcl-2-interacting domain death agonist (BID) triggers mitochondria disintegration.

Howells CC, Baumann WT, Samuels DC, Finkielstein CV. Howells CC, et al. J Theor Biol. 2011 Feb 21;271(1):114-23. doi: 10.1016/j.jtbi.2010.11.040. Epub 2010 Dec 3. J Theor Biol. 2011. PMID: 21130780

9

BH3 domains other than Bim and Bid can directly activate Bax/Bak.

Du H, Wolf J, Schafer B, Moldoveanu T, Chipuk JE, Kuwana T. Du H, et al. J Biol Chem. 2011 Jan 7;286(1):491-501. doi: 10.1074/jbc.M110.167148. Epub 2010 Nov 1. J Biol Chem. 2011. PMID: 21041309 Free PMC article.

10

Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax.

Hockings C, Anwari K, Ninnis RL, Brouwer J, O'Hely M, Evangelista M, Hinds MG, Czabotar PE, Lee EF, Fairlie WD, Dewson G, Kluck RM. Hockings C, et al. Cell Death Dis. 2015 Apr 23;6(4):e1735. doi: 10.1038/cddis.2015.105. Cell Death Dis. 2015. PMID: 25906158 Free PMC article.

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