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Items: 1 to 20 of 105

1.

Breakpoints in immunoregulation required for Th1 cells to induce diabetes.

Neighbors M, Hartley SB, Xu X, Castro AG, Bouley DM, O'Garra A.

Eur J Immunol. 2006 Sep;36(9):2315-23.

3.

B7-2 (CD86) controls the priming of autoreactive CD4 T cell response against pancreatic islets.

Yadav D, Judkowski V, Flodstrom-Tullberg M, Sterling L, Redmond WL, Sherman L, Sarvetnick N.

J Immunol. 2004 Sep 15;173(6):3631-9.

4.

Stability of Th1 and Th2 populations.

Perez VL, Lederer JA, Lichtman AH, Abbas AK.

Int Immunol. 1995 May;7(5):869-75.

PMID:
7547713
5.

Cooperative roles of CTLA-4 and regulatory T cells in tolerance to an islet cell antigen.

Eggena MP, Walker LS, Nagabhushanam V, Barron L, Chodos A, Abbas AK.

J Exp Med. 2004 Jun 21;199(12):1725-30.

6.

Cellular and molecular events in the localization of diabetogenic T cells to islets of Langerhans.

Calderon B, Carrero JA, Miller MJ, Unanue ER.

Proc Natl Acad Sci U S A. 2011 Jan 25;108(4):1561-6. doi: 10.1073/pnas.1018973108. Epub 2011 Jan 10.

7.

Cellular requirements for diabetes induction in DO11.10xRIPmOVA mice.

Wesley JD, Sather BD, Perdue NR, Ziegler SF, Campbell DJ.

J Immunol. 2010 Oct 15;185(8):4760-8. doi: 10.4049/jimmunol.1000820. Epub 2010 Sep 20.

8.

Strength of TCR signal determines the costimulatory requirements for Th1 and Th2 CD4+ T cell differentiation.

Tao X, Constant S, Jorritsma P, Bottomly K.

J Immunol. 1997 Dec 15;159(12):5956-63.

PMID:
9550393
9.
10.

T-bet-deficient NOD mice are protected from diabetes due to defects in both T cell and innate immune system function.

Esensten JH, Lee MR, Glimcher LH, Bluestone JA.

J Immunol. 2009 Jul 1;183(1):75-82. doi: 10.4049/jimmunol.0804154. Epub 2009 Jun 17.

11.

Autoreactive human T-cell receptor initiates insulitis and impaired glucose tolerance in HLA DR4 transgenic mice.

Gebe JA, Unrath KA, Yue BB, Miyake T, Falk BA, Nepom GT.

J Autoimmun. 2008 Jun;30(4):197-206. Epub 2007 Oct 18.

12.

A range of CD4 T cell tolerance: partial inactivation to organ-specific antigen allows nondestructive thyroiditis or insulitis.

Akkaraju S, Ho WY, Leong D, Canaan K, Davis MM, Goodnow CC.

Immunity. 1997 Aug;7(2):255-71.

13.

Anti-islet autoantibodies trigger autoimmune diabetes in the presence of an increased frequency of islet-reactive CD4 T cells.

Silva DG, Daley SR, Hogan J, Lee SK, Teh CE, Hu DY, Lam KP, Goodnow CC, Vinuesa CG.

Diabetes. 2011 Aug;60(8):2102-11. doi: 10.2337/db10-1344.

14.

Adaptive islet-specific regulatory CD4 T cells control autoimmune diabetes and mediate the disappearance of pathogenic Th1 cells in vivo.

Weber SE, Harbertson J, Godebu E, Mros GA, Padrick RC, Carson BD, Ziegler SF, Bradley LM.

J Immunol. 2006 Apr 15;176(8):4730-9.

15.

Manipulation of Th1/Th2 balance in vivo by adoptive transfer of antigen-specific Th1 or Th2 cells.

Ohta A, Sato N, Yahata T, Ohmi Y, Santa K, Sato T, Tashiro H, Habu S, Nishimura T.

J Immunol Methods. 1997 Nov 10;209(1):85-92.

PMID:
9448037
16.
17.

Complexities of CD28/B7: CTLA-4 costimulatory pathways in autoimmunity and transplantation.

Salomon B, Bluestone JA.

Annu Rev Immunol. 2001;19:225-52. Review.

PMID:
11244036
19.

Functional tolerance is maintained despite proliferation of CD4 T cells after encounter with tissue-derived antigen.

Ausubel LJ, Chodos A, Bekarian N, Abbas AK, Walker LS.

Dev Immunol. 2002 Sep;9(3):173-6.

20.

Defining Th1 and Th2 immune responses in a reciprocal cytokine environment in vivo.

Chitnis T, Salama AD, Grusby MJ, Sayegh MH, Khoury SJ.

J Immunol. 2004 Apr 1;172(7):4260-5.

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