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Items: 1 to 20 of 88

1.
2.

Hippocampal poly(ADP-Ribose) polymerase 1 and caspase 3 activation in neonatal bornavirus infection.

Williams BL, Hornig M, Yaddanapudi K, Lipkin WI.

J Virol. 2008 Feb;82(4):1748-58. Epub 2007 Dec 5.

3.
4.

Metallothioneins and zinc dysregulation contribute to neurodevelopmental damage in a model of perinatal viral infection.

Williams BL, Yaddanapudi K, Kirk CM, Soman A, Hornig M, Lipkin WI.

Brain Pathol. 2006 Jan;16(1):1-14.

PMID:
16612977
5.

Spatiotemporal analysis of purkinje cell degeneration relative to parasagittal expression domains in a model of neonatal viral infection.

Williams BL, Yaddanapudi K, Hornig M, Lipkin WI.

J Virol. 2007 Mar;81(6):2675-87. Epub 2006 Dec 20.

6.
7.

Neonatal Borna disease virus infection (BDV)-induced damage to the cerebellum is associated with sensorimotor deficits in developing Lewis rats.

Pletnikov MV, Rubin SA, Carbone KM, Moran TH, Schwartz GJ.

Brain Res Dev Brain Res. 2001 Jan 31;126(1):1-12.

PMID:
11172881
8.

Endoplasmic reticulum stress and neurodegeneration in experimental cerebral malaria.

Anand SS, Babu PP.

Neurosignals. 2013;21(1-2):99-111. doi: 10.1159/000336970. Epub 2012 May 15.

10.

Cytokine expression in the rat central nervous system following perinatal Borna disease virus infection.

Sauder C, de la Torre JC.

J Neuroimmunol. 1999 Apr 1;96(1):29-45.

PMID:
10227422
11.

Borna disease virus.

Jordan I, Lipkin WI.

Rev Med Virol. 2001 Jan-Feb;11(1):37-57. Review.

PMID:
11241801
12.

Astrocytes play a key role in activation of microglia by persistent Borna disease virus infection.

Ovanesov MV, Ayhan Y, Wolbert C, Moldovan K, Sauder C, Pletnikov MV.

J Neuroinflammation. 2008 Nov 11;5:50. doi: 10.1186/1742-2094-5-50.

13.

Cytokines downregulate the sarcoendoplasmic reticulum pump Ca2+ ATPase 2b and deplete endoplasmic reticulum Ca2+, leading to induction of endoplasmic reticulum stress in pancreatic beta-cells.

Cardozo AK, Ortis F, Storling J, Feng YM, Rasschaert J, Tonnesen M, Van Eylen F, Mandrup-Poulsen T, Herchuelz A, Eizirik DL.

Diabetes. 2005 Feb;54(2):452-61.

14.

Induction of unfolded protein response during neuronal induction of rat bone marrow stromal cells and mouse embryonic stem cells.

Cho YM, Jang YS, Jang YM, Chung SM, Kim HS, Lee JH, Jeong SW, Kim IK, Kim JJ, Kim KS, Kwon OJ.

Exp Mol Med. 2009 Jun 30;41(6):440-52. doi: 10.3858/emm.2009.41.6.049.

15.

Early activation of endoplasmic reticulum stress is associated with arginine-induced acute pancreatitis.

Kubisch CH, Sans MD, Arumugam T, Ernst SA, Williams JA, Logsdon CD.

Am J Physiol Gastrointest Liver Physiol. 2006 Aug;291(2):G238-45. Epub 2006 Mar 30.

16.

Effects of neonatal rat Borna disease virus (BDV) infection on the postnatal development of the brain monoaminergic systems.

Pletnikov MV, Rubin SA, Schwartz GJ, Carbone KM, Moran TH.

Brain Res Dev Brain Res. 2000 Feb 7;119(2):179-85.

PMID:
10675767
17.

Layer specific changes of astroglial gap junctions in the rat cerebellar cortex by persistent Borna Disease Virus infection.

Köster-Patzlaff C, Hosseini SM, Reuss B.

Brain Res. 2008 Jul 11;1219:143-58. doi: 10.1016/j.brainres.2008.04.062. Epub 2008 Apr 30.

PMID:
18538309
18.

Endoplasmic reticulum stress-mediated apoptosis in pancreatic beta-cells.

Oyadomari S, Araki E, Mori M.

Apoptosis. 2002 Aug;7(4):335-45. Review.

PMID:
12101393
19.

Oleate rescues INS-1E β-cells from palmitate-induced apoptosis by preventing activation of the unfolded protein response.

Sommerweiss D, Gorski T, Richter S, Garten A, Kiess W.

Biochem Biophys Res Commun. 2013 Nov 29;441(4):770-6. doi: 10.1016/j.bbrc.2013.10.130. Epub 2013 Nov 1.

20.

[The regulation of unfolded protein response by OASIS, a transmembrane bZIP transcription factor, in astrocytes].

Imaizumi K, Tohyama M.

Nihon Yakurigaku Zasshi. 2004 Dec;124(6):383-90. Review. Japanese.

PMID:
15572842

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