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Items: 1 to 20 of 256

1.

Plaques of Alzheimer's disease originate from cysts of Borrelia burgdorferi, the Lyme disease spirochete.

MacDonald AB.

Med Hypotheses. 2006;67(3):592-600. Epub 2006 May 3.

PMID:
16675154
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Progranulin expression correlates with dense-core amyloid plaque burden in Alzheimer disease mouse models.

Pereson S, Wils H, Kleinberger G, McGowan E, Vandewoestyne M, Van Broeck B, Joris G, Cuijt I, Deforce D, Hutton M, Van Broeckhoven C, Kumar-Singh S.

J Pathol. 2009 Oct;219(2):173-81. doi: 10.1002/path.2580.

PMID:
19557827
5.

Binding of two potential imaging agents targeting amyloid plaques in postmortem brain tissues of patients with Alzheimer's disease.

Kung MP, Hou C, Zhuang ZP, Skovronsky D, Kung HF.

Brain Res. 2004 Oct 29;1025(1-2):98-105. Erratum in: Brain Res. 2005 Jan 21;1031(2):302.

PMID:
15464749
6.

Magnetic resonance imaging of Alzheimer's pathology in the brains of living transgenic mice: a new tool in Alzheimer's disease research.

Jack CR Jr, Marjanska M, Wengenack TM, Reyes DA, Curran GL, Lin J, Preboske GM, Poduslo JF, Garwood M.

Neuroscientist. 2007 Feb;13(1):38-48. Review.

PMID:
17229974
7.

Evidence that neurones accumulating amyloid can undergo lysis to form amyloid plaques in Alzheimer's disease.

D'Andrea MR, Nagele RG, Wang HY, Peterson PA, Lee DH.

Histopathology. 2001 Feb;38(2):120-34.

PMID:
11207825
8.

Novel 'inflammatory plaque' pathology in presenilin-1 Alzheimer's disease.

Shepherd CE, Gregory GC, Vickers JC, Halliday GM.

Neuropathol Appl Neurobiol. 2005 Oct;31(5):503-11.

PMID:
16150121
9.

Borrelia burgdorferi persists in the brain in chronic lyme neuroborreliosis and may be associated with Alzheimer disease.

Miklossy J, Khalili K, Gern L, Ericson RL, Darekar P, Bolle L, Hurlimann J, Paster BJ.

J Alzheimers Dis. 2004 Dec;6(6):639-49; discussion 673-81.

PMID:
15665404
10.

Morphologically distinct types of amyloid plaques point the way to a better understanding of Alzheimer's disease pathogenesis.

D'Andrea MR, Nagele RG.

Biotech Histochem. 2010 Apr;85(2):133-47. doi: 10.3109/10520290903389445.

PMID:
20121465
11.

Is aging part of Alzheimer's disease, or is Alzheimer's disease part of aging?

Swerdlow RH.

Neurobiol Aging. 2007 Oct;28(10):1465-80. Epub 2006 Jul 31. Review.

PMID:
16876913
12.

Analysis of single Alzheimer solid plaque cores by laser capture microscopy and nanoelectrospray/tandem mass spectrometry.

Söderberg L, Bogdanovic N, Axelsson B, Winblad B, Näslund J, Tjernberg LO.

Biochemistry. 2006 Aug 15;45(32):9849-56.

PMID:
16893185
13.

[Reconstruction of the three-dimensional structure of senile plaques in Alzheimer disease].

Ye W, Zhou JN, Hu XY, Liu JW, He JH, Tang XW.

Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2003 May;35(5):449-53. Chinese.

PMID:
12766806
14.

[Etiology and pathogenesis of Alzheimer dementia].

Kratzsch T, Peters J, Frölich L.

Wien Med Wochenschr. 2002;152(3-4):72-6. German.

PMID:
11925775
15.

What initiates the formation of senile plaques? The origin of Alzheimer-like dementias in capillary haemorrhages.

Stone J.

Med Hypotheses. 2008 Sep;71(3):347-59. doi: 10.1016/j.mehy.2008.04.007. Epub 2008 Jun 3.

PMID:
18524497
16.

Confocal observation of senile plaques in Alzheimer's disease: senile plaque morphology and relationship between senile plaques and astrocytes.

Kato S, Gondo T, Hoshii Y, Takahashi M, Yamada M, Ishihara T.

Pathol Int. 1998 May;48(5):332-40.

PMID:
9704339
17.

Alzheimer-like plaque formation by human macrophages is reduced by fibrillation inhibitors and lovastatin.

Gellermann GP, Ullrich K, Tannert A, Unger C, Habicht G, Sauter SR, Hortschansky P, Horn U, Möllmann U, Decker M, Lehmann J, Fändrich M.

J Mol Biol. 2006 Jul 7;360(2):251-7. Epub 2006 May 24.

PMID:
16765377
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