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Items: 1 to 20 of 341

2.

Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.

Zhou Q, Desta T, Fenton M, Graves DT, Amar S.

Infect Immun. 2005 Feb;73(2):935-43. Erratum in: Infect Immun. 2006 Mar;74(3):1995.

4.

Proteomic mapping of stimulus-specific signaling pathways involved in THP-1 cells exposed to Porphyromonas gingivalis or its purified components.

Saba JA, McComb ME, Potts DL, Costello CE, Amar S.

J Proteome Res. 2007 Jun;6(6):2211-21. Epub 2007 May 4.

6.

Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide.

Martin M, Schifferle RE, Cuesta N, Vogel SN, Katz J, Michalek SM.

J Immunol. 2003 Jul 15;171(2):717-25.

7.

Differential activation of human gingival epithelial cells and monocytes by Porphyromonas gingivalis fimbriae.

Eskan MA, Hajishengallis G, Kinane DF.

Infect Immun. 2007 Feb;75(2):892-8. Epub 2006 Nov 21.

8.

Effects of Porphyromonas gingivalis and Escherichia coli lipopolysaccharides on mononuclear phagocytes.

Roberts FA, Richardson GJ, Michalek SM.

Infect Immun. 1997 Aug;65(8):3248-54.

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Lipopolysaccharide (LPS) of Porphyromonas gingivalis induces IL-1beta, TNF-alpha and IL-6 production by THP-1 cells in a way different from that of Escherichia coli LPS.

Diya Zhang, Lili Chen, Shenglai Li, Zhiyuan Gu, Jie Yan.

Innate Immun. 2008 Apr;14(2):99-107. doi: 10.1177/1753425907088244.

PMID:
18713726
14.

Exposure to Porphyromonas gingivalis LPS during macrophage polarisation leads to diminished inflammatory cytokine production.

Belfield LA, Bennett JH, Abate W, Jackson SK.

Arch Oral Biol. 2017 Sep;81:41-47. doi: 10.1016/j.archoralbio.2017.04.021. Epub 2017 Apr 20.

PMID:
28472719
15.

Differential expression of immunoregulatory genes in monocytes in response to Porphyromonas gingivalis and Escherichia coli lipopolysaccharide.

Barksby HE, Nile CJ, Jaedicke KM, Taylor JJ, Preshaw PM.

Clin Exp Immunol. 2009 Jun;156(3):479-87. doi: 10.1111/j.1365-2249.2009.03920.x.

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Porphyromonas gingivalis lipopolysaccharide alters atherosclerotic-related gene expression in oxidized low-density-lipoprotein-induced macrophages and foam cells.

Lei L, Li H, Yan F, Li Y, Xiao Y.

J Periodontal Res. 2011 Aug;46(4):427-37. doi: 10.1111/j.1600-0765.2011.01356.x. Epub 2011 Mar 21.

PMID:
21418223
18.

Porphyromonas gingivalis lipopolysaccharide weakly activates M1 and M2 polarized mouse macrophages but induces inflammatory cytokines.

Holden JA, Attard TJ, Laughton KM, Mansell A, O'Brien-Simpson NM, Reynolds EC.

Infect Immun. 2014 Oct;82(10):4190-203. doi: 10.1128/IAI.02325-14. Epub 2014 Jul 21.

19.

Analysis of immunostimulatory activity of Porphyromonas gingivalis fimbriae conferred by Toll-like receptor 2.

Aoki Y, Tabeta K, Murakami Y, Yoshimura F, Yamazaki K.

Biochem Biophys Res Commun. 2010 Jul 16;398(1):86-91. doi: 10.1016/j.bbrc.2010.06.040. Epub 2010 Jun 15.

PMID:
20558141
20.

Blocking proinflammatory cytokine release modulates peripheral blood mononuclear cell response to Porphyromonas gingivalis.

Berker E, Kantarci A, Hasturk H, Van Dyke TE.

J Periodontol. 2013 Sep;84(9):1337-45. doi: 10.1902/jop.2012.120422. Epub 2012 Nov 23.

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