Format
Sort by

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 437

1.

Development and progression of alopecia in the vitamin D receptor null mouse.

Bikle DD, Elalieh H, Chang S, Xie Z, Sundberg JP.

J Cell Physiol. 2006 May;207(2):340-53.

PMID:
16419036
2.

Vitamin D receptor ablation alters skin architecture and homeostasis of dendritic epidermal T cells.

Meindl S, Rot A, Hoetzenecker W, Kato S, Cross HS, Elbe-Bürger A.

Br J Dermatol. 2005 Feb;152(2):231-41.

PMID:
15727633
3.

Disruption of the hedgehog signaling pathway contributes to the hair follicle cycling deficiency in Vdr knockout mice.

Teichert A, Elalieh H, Bikle D.

J Cell Physiol. 2010 Nov;225(2):482-9. doi: 10.1002/jcp.22227.

4.

Physical and functional interaction between the vitamin D receptor and hairless corepressor, two proteins required for hair cycling.

Hsieh JC, Sisk JM, Jurutka PW, Haussler CA, Slater SA, Haussler MR, Thompson CC.

J Biol Chem. 2003 Oct 3;278(40):38665-74. Epub 2003 Jul 7.

5.

RXR-alpha ablation in skin keratinocytes results in alopecia and epidermal alterations.

Li M, Chiba H, Warot X, Messaddeq N, Gérard C, Chambon P, Metzger D.

Development. 2001 Mar;128(5):675-88.

6.

Ligand-independent actions of the vitamin D receptor maintain hair follicle homeostasis.

Skorija K, Cox M, Sisk JM, Dowd DR, MacDonald PN, Thompson CC, Demay MB.

Mol Endocrinol. 2005 Apr;19(4):855-62. Epub 2004 Dec 9.

PMID:
15591533
7.

Keratin 17 modulates hair follicle cycling in a TNFalpha-dependent fashion.

Tong X, Coulombe PA.

Genes Dev. 2006 May 15;20(10):1353-64.

8.
9.

N-WASP is a novel regulator of hair-follicle cycling that controls antiproliferative TGF{beta} pathways.

Lefever T, Pedersen E, Basse A, Paus R, Quondamatteo F, Stanley AC, Langbein L, Wu X, Wehland J, Lommel S, Brakebusch C.

J Cell Sci. 2010 Jan 1;123(Pt 1):128-40. doi: 10.1242/jcs.053835.

10.
11.
12.

Hair follicle expression of 1,25-dihydroxyvitamin D3 receptors during the murine hair cycle.

Reichrath J, Schilli M, Kerber A, Bahmer FA, Czarnetzki BM, Paus R.

Br J Dermatol. 1994 Oct;131(4):477-82.

PMID:
7947199
13.

Activation of Notch1 in the hair follicle leads to cell-fate switch and Mohawk alopecia.

Uyttendaele H, Panteleyev AA, de Berker D, Tobin DT, Christiano AM.

Differentiation. 2004 Oct;72(8):396-409.

PMID:
15606499
14.

Genetically null mice reveal a central role for epidermal growth factor receptor in the differentiation of the hair follicle and normal hair development.

Hansen LA, Alexander N, Hogan ME, Sundberg JP, Dlugosz A, Threadgill DW, Magnuson T, Yuspa SH.

Am J Pathol. 1997 Jun;150(6):1959-75.

16.

Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle.

Wojcik SM, Imakado S, Seki T, Longley MA, Petherbridge L, Bundman DS, Bickenbach JR, Rothnagel JA, Roop DR.

Differentiation. 1999 Oct;65(2):97-112.

PMID:
10550543
17.

Patterns of hairless (hr) gene expression in mouse hair follicle morphogenesis and cycling.

Panteleyev AA, Paus R, Christiano AM.

Am J Pathol. 2000 Oct;157(4):1071-9.

18.

Coactivator MED1 ablation in keratinocytes results in hair-cycling defects and epidermal alterations.

Oda Y, Hu L, Bul V, Elalieh H, Reddy JK, Bikle DD.

J Invest Dermatol. 2012 Apr;132(4):1075-83. doi: 10.1038/jid.2011.430. Epub 2011 Dec 22.

19.

Does D matter? The role of vitamin D in hair disorders and hair follicle cycling.

Amor KT, Rashid RM, Mirmirani P.

Dermatol Online J. 2010 Feb 15;16(2):3. Review.

20.

The vitamin D receptor is a Wnt effector that controls hair follicle differentiation and specifies tumor type in adult epidermis.

Pálmer HG, Anjos-Afonso F, Carmeliet G, Takeda H, Watt FM.

PLoS One. 2008 Jan 23;3(1):e1483. doi: 10.1371/journal.pone.0001483.

Items per page

Supplemental Content

Write to the Help Desk